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Journal Abstract Search

165 related items for PubMed ID: 34524791

  • 21. Postmortem Neocortical 3H-PiB Binding and Levels of Unmodified and Pyroglutamate Aβ in Down Syndrome and Sporadic Alzheimer's Disease.
    Pivtoraiko VN, Racic T, Abrahamson EE, Villemagne VL, Handen BL, Lott IT, Head E, Ikonomovic MD.
    Front Aging Neurosci; 2021; 13():728739. PubMed ID: 34489686
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  • 22. Structural variation in amyloid-β fibrils from Alzheimer's disease clinical subtypes.
    Qiang W, Yau WM, Lu JX, Collinge J, Tycko R.
    Nature; 2017 Jan 12; 541(7636):217-221. PubMed ID: 28052060
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  • 23. Transthyretin Inhibits Primary and Secondary Nucleations of Amyloid-β Peptide Aggregation and Reduces the Toxicity of Its Oligomers.
    Ghadami SA, Chia S, Ruggeri FS, Meisl G, Bemporad F, Habchi J, Cascella R, Dobson CM, Vendruscolo M, Knowles TPJ, Chiti F.
    Biomacromolecules; 2020 Mar 09; 21(3):1112-1125. PubMed ID: 32011129
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  • 24. Conformation of Pyroglutamated Amyloid β (3-40) and (11-40) Fibrils - Extended or Hairpin?
    Scheidt HA, Korn A, Schwarze B, Krueger M, Huster D.
    J Phys Chem B; 2024 Feb 22; 128(7):1647-1655. PubMed ID: 38334278
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  • 26. Purification and Characterization of Recombinant N-Terminally Pyroglutamate-Modified Amyloid-β Variants and Structural Analysis by Solution NMR Spectroscopy.
    Dammers C, Gremer L, Neudecker P, Demuth HU, Schwarten M, Willbold D.
    PLoS One; 2015 Feb 22; 10(10):e0139710. PubMed ID: 26436664
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  • 27. Structural characteristics of oligomers formed by pyroglutamate-modified amyloid β peptides studied by solid-state NMR.
    Scheidt HA, Das A, Korn A, Krueger M, Maiti S, Huster D.
    Phys Chem Chem Phys; 2020 Jul 29; 22(29):16887-16895. PubMed ID: 32666970
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  • 28. Structural Polymorphism of Alzheimer's β-Amyloid Fibrils as Controlled by an E22 Switch: A Solid-State NMR Study.
    Elkins MR, Wang T, Nick M, Jo H, Lemmin T, Prusiner SB, DeGrado WF, Stöhr J, Hong M.
    J Am Chem Soc; 2016 Aug 10; 138(31):9840-52. PubMed ID: 27414264
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  • 29. Neuropathology and biochemistry of Aβ and its aggregates in Alzheimer's disease.
    Thal DR, Walter J, Saido TC, Fändrich M.
    Acta Neuropathol; 2015 Feb 10; 129(2):167-82. PubMed ID: 25534025
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  • 30. Human cerebral vascular amyloid contains both antiparallel and parallel in-register Aβ40 fibrils.
    Irizarry BA, Davis J, Zhu X, Boon BDC, Rozemuller AJM, Van Nostrand WE, Smith SO.
    J Biol Chem; 2021 Nov 10; 297(5):101259. PubMed ID: 34599967
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  • 32. CLVFFA-Functionalized Gold Nanoclusters Inhibit Aβ40 Fibrillation, Fibrils' Prolongation, and Mature Fibrils' Disaggregation.
    Hao S, Li X, Han A, Yang Y, Fang G, Liu J, Wang S.
    ACS Chem Neurosci; 2019 Nov 20; 10(11):4633-4642. PubMed ID: 31637909
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  • 33. Amyloid-β40 E22K fibril in familial Alzheimer's disease is more thermostable and susceptible to seeding.
    Lee MC, Liao YH, Chen SH, Chen YR.
    IUBMB Life; 2022 Aug 20; 74(8):739-747. PubMed ID: 34724333
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  • 34. Rapid Conversion of Amyloid-Beta 1-40 Oligomers to Mature Fibrils through a Self-Catalytic Bimolecular Process.
    Morel B, Carrasco-Jiménez MP, Jurado S, Conejero-Lara F.
    Int J Mol Sci; 2021 Jun 14; 22(12):. PubMed ID: 34198692
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  • 35. Intracellular amyloid-β accumulation in calcium-binding protein-deficient neurons leads to amyloid-β plaque formation in animal model of Alzheimer's disease.
    Moon M, Hong HS, Nam DW, Baik SH, Song H, Kook SY, Kim YS, Lee J, Mook-Jung I.
    J Alzheimers Dis; 2012 Jun 14; 29(3):615-28. PubMed ID: 22269161
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