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173 related items for PubMed ID: 34632595

  • 1. Insulin acts as an atypical KCNQ1/KCNE1-current activator and reverses long QT in insulin-resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3 -adrenergic receptor signaling pathway.
    Olgar Y, Durak A, Bitirim CV, Tuncay E, Turan B.
    J Cell Physiol; 2022 Feb; 237(2):1353-1371. PubMed ID: 34632595
    [Abstract] [Full Text] [Related]

  • 2. Insights into Cardiac IKs (KCNQ1/KCNE1) Channels Regulation.
    Wu X, Larsson HP.
    Int J Mol Sci; 2020 Dec 11; 21(24):. PubMed ID: 33322401
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  • 3. LQT1 mutations in KCNQ1 C-terminus assembly domain suppress IKs using different mechanisms.
    Aromolaran AS, Subramanyam P, Chang DD, Kobertz WR, Colecraft HM.
    Cardiovasc Res; 2014 Dec 01; 104(3):501-11. PubMed ID: 25344363
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  • 4. Dysfunctional potassium channel subunit interaction as a novel mechanism of long QT syndrome.
    Hoosien M, Ahearn ME, Myerburg RJ, Pham TV, Miller TE, Smets MJ, Baumbach-Reardon L, Young ML, Farooq A, Bishopric NH.
    Heart Rhythm; 2013 May 01; 10(5):728-37. PubMed ID: 23291057
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  • 6. Delayed KCNQ1/KCNE1 assembly on the cell surface helps IKs fulfil its function as a repolarization reserve in the heart.
    Wilson ZT, Jiang M, Geng J, Kaur S, Workman SW, Hao J, Bernas T, Tseng GN.
    J Physiol; 2021 Jul 01; 599(13):3337-3361. PubMed ID: 33963564
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  • 7. Long QT mutations at the interface between KCNQ1 helix C and KCNE1 disrupt I(KS) regulation by PKA and PIP₂.
    Dvir M, Strulovich R, Sachyani D, Ben-Tal Cohen I, Haitin Y, Dessauer C, Pongs O, Kass R, Hirsch JA, Attali B.
    J Cell Sci; 2014 Sep 15; 127(Pt 18):3943-55. PubMed ID: 25037568
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  • 8. Adult Ventricular Myocytes Segregate KCNQ1 and KCNE1 to Keep the IKs Amplitude in Check Until When Larger IKs Is Needed.
    Jiang M, Wang Y, Tseng GN.
    Circ Arrhythm Electrophysiol; 2017 Jun 15; 10(6):. PubMed ID: 28611207
    [Abstract] [Full Text] [Related]

  • 9. Synergistic modulation of KCNQ1/KCNE1 K(+) channels (IKs) by phosphatidylinositol 4,5-bisphosphate (PIP2) and [ATP]i.
    Kienitz MC, Vladimirova D.
    Cell Signal; 2015 Jul 15; 27(7):1457-68. PubMed ID: 25892084
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  • 14. Receptor Species-dependent Desensitization Controls KCNQ1/KCNE1 K+ Channels as Downstream Effectors of Gq Protein-coupled Receptors.
    Kienitz MC, Vladimirova D, Müller C, Pott L, Rinne A.
    J Biol Chem; 2016 Dec 16; 291(51):26410-26426. PubMed ID: 27834678
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  • 16. Dynamic subunit stoichiometry confers a progressive continuum of pharmacological sensitivity by KCNQ potassium channels.
    Yu H, Lin Z, Mattmann ME, Zou B, Terrenoire C, Zhang H, Wu M, McManus OB, Kass RS, Lindsley CW, Hopkins CR, Li M.
    Proc Natl Acad Sci U S A; 2013 May 21; 110(21):8732-7. PubMed ID: 23650380
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  • 18. Modulation of IKs channel-PIP2 interaction by PRMT1 plays a critical role in the control of cardiac repolarization.
    An X, Lee J, Kim GH, Kim HJ, Pyo HJ, Kwon I, Cho H.
    J Cell Physiol; 2022 Jul 21; 237(7):3069-3079. PubMed ID: 35580065
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  • 20. PKCβII specifically regulates KCNQ1/KCNE1 channel membrane localization.
    Braun C, Parks XX, Qudsi H, Lopes CM.
    J Mol Cell Cardiol; 2020 Jan 21; 138():283-290. PubMed ID: 31785237
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