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Journal Abstract Search

272 related items for PubMed ID: 34697420

  • 1. Prenatal dexamethasone exposure programs the decreased testosterone synthesis in offspring rats by low level of endogenous glucocorticoids.
    Liu M, Liu Y, Pei LG, Zhang Q, Xiao H, Chen YW, Wang H.
    Acta Pharmacol Sin; 2022 Jun; 43(6):1461-1472. PubMed ID: 34697420
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  • 5. The GC-IGF1 axis-mediated testicular dysplasia caused by prenatal caffeine exposure.
    Pei LG, Zhang Q, Yuan C, Liu M, Zou YF, Lv F, Luo DJ, Zhong S, Wang H.
    J Endocrinol; 2019 Jul 01; 242(1):M17-M32. PubMed ID: 31141788
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  • 6. Corticosterone rather than ethanol epigenetic programmed testicular dysplasia caused by prenatal ethanol exposure in male offspring rats.
    Liu M, Zhang Q, Pei L, Zou Y, Chen G, Wang H.
    Epigenetics; 2019 Mar 01; 14(3):245-259. PubMed ID: 30821590
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  • 8. Dexamethasone exposure during pregnancy triggers metabolic syndrome in offspring via epigenetic alteration of IGF1.
    Xiao H, He B, Liu H, Chen Y, Xiao D, Wang H.
    Cell Commun Signal; 2024 Jan 23; 22(1):62. PubMed ID: 38263047
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  • 9. MiR-133a-3p/Sirt1 epigenetic programming mediates hypercholesterolemia susceptibility in female offspring induced by prenatal dexamethasone exposure.
    Li X, Hu W, Li L, Chen Z, Jiang T, Zhang D, Liu K, Wang H.
    Biochem Pharmacol; 2022 Dec 23; 206():115306. PubMed ID: 36326533
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  • 15. Decreased H3K9ac level of KLF4 mediates podocyte developmental toxicity induced by prenatal caffeine exposure in male offspring rats.
    Zhu Y, Chen H, Zhao X, Li B, He H, Cheng H, Wang H, Ao Y.
    Toxicol Lett; 2019 Oct 10; 314():63-74. PubMed ID: 31306741
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  • 16. Epigenetic repression of AT2 receptor is involved in β cell dysfunction and glucose intolerance of adult female offspring rats exposed to dexamethasone prenatally.
    Kou H, Gui S, Dai Y, Guo Y, Wang H.
    Toxicol Appl Pharmacol; 2020 Oct 01; 404():115187. PubMed ID: 32791177
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  • 17. Programming changes of hippocampal miR-134-5p/SOX2 signal mediate the susceptibility to depression in prenatal dexamethasone-exposed female offspring.
    Jiang T, Hu S, Dai S, Yi Y, Wang T, Li X, Luo M, Li K, Chen L, Wang H, Xu D.
    Cell Biol Toxicol; 2022 Feb 01; 38(1):69-86. PubMed ID: 33619658
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  • 18. 11β-Hydroxysteroid dehydrogenase 2: A key mediator of high susceptibility to osteoporosis in offspring after prenatal dexamethasone exposure.
    Wu Z, Wen Y, Xiao H, Zhu J, Li B, Shangguan Y, He H, Wang H, Chen L.
    Pharmacol Res; 2022 Jan 01; 175():105990. PubMed ID: 34808367
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