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Journal Abstract Search

340 related items for PubMed ID: 34830074

  • 1. Metals in ALS TDP-43 Pathology.
    Koski L, Ronnevi C, Berntsson E, Wärmländer SKTS, Roos PM.
    Int J Mol Sci; 2021 Nov 11; 22(22):. PubMed ID: 34830074
    [Abstract] [Full Text] [Related]

  • 2. From nucleation to widespread propagation: A prion-like concept for ALS.
    Maniecka Z, Polymenidou M.
    Virus Res; 2015 Sep 02; 207():94-105. PubMed ID: 25656065
    [Abstract] [Full Text] [Related]

  • 3. RNA and Protein Interactors with TDP-43 in Human Spinal-Cord Lysates in Amyotrophic Lateral Sclerosis.
    Volkening K, Keller BA, Leystra-Lantz C, Strong MJ.
    J Proteome Res; 2018 Apr 06; 17(4):1712-1729. PubMed ID: 29513014
    [Abstract] [Full Text] [Related]

  • 4. Effects of intracellular calcium accumulation on proteins encoded by the major genes underlying amyotrophic lateral sclerosis.
    De Marco G, Lomartire A, Manera U, Canosa A, Grassano M, Casale F, Fuda G, Salamone P, Rinaudo MT, Colombatto S, Moglia C, Chiò A, Calvo A.
    Sci Rep; 2022 Jan 10; 12(1):395. PubMed ID: 35013445
    [Abstract] [Full Text] [Related]

  • 5. Connecting RNA-Modifying Similarities of TDP-43, FUS, and SOD1 with MicroRNA Dysregulation Amidst A Renewed Network Perspective of Amyotrophic Lateral Sclerosis Proteinopathy.
    Pham J, Keon M, Brennan S, Saksena N.
    Int J Mol Sci; 2020 May 14; 21(10):. PubMed ID: 32422969
    [Abstract] [Full Text] [Related]

  • 6. Molecular pathology and genetic advances in amyotrophic lateral sclerosis: an emerging molecular pathway and the significance of glial pathology.
    Ince PG, Highley JR, Kirby J, Wharton SB, Takahashi H, Strong MJ, Shaw PJ.
    Acta Neuropathol; 2011 Dec 14; 122(6):657-71. PubMed ID: 22105541
    [Abstract] [Full Text] [Related]

  • 7. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
    Soo KY, Halloran M, Sundaramoorthy V, Parakh S, Toth RP, Southam KA, McLean CA, Lock P, King A, Farg MA, Atkin JD.
    Acta Neuropathol; 2015 Nov 14; 130(5):679-97. PubMed ID: 26298469
    [Abstract] [Full Text] [Related]

  • 8. Co-aggregation of RNA binding proteins in ALS spinal motor neurons: evidence of a common pathogenic mechanism.
    Keller BA, Volkening K, Droppelmann CA, Ang LC, Rademakers R, Strong MJ.
    Acta Neuropathol; 2012 Nov 14; 124(5):733-47. PubMed ID: 22941224
    [Abstract] [Full Text] [Related]

  • 9. TDP-43 or FUS-induced misfolded human wild-type SOD1 can propagate intercellularly in a prion-like fashion.
    Pokrishevsky E, Grad LI, Cashman NR.
    Sci Rep; 2016 Mar 01; 6():22155. PubMed ID: 26926802
    [Abstract] [Full Text] [Related]

  • 10. From Mouse Models to Human Disease: An Approach for Amyotrophic Lateral Sclerosis.
    Alrafiah AR.
    In Vivo; 2018 Mar 01; 32(5):983-998. PubMed ID: 30150420
    [Abstract] [Full Text] [Related]

  • 11. The relevance of contact-independent cell-to-cell transfer of TDP-43 and SOD1 in amyotrophic lateral sclerosis.
    Hanspal MA, Dobson CM, Yerbury JJ, Kumita JR.
    Biochim Biophys Acta Mol Basis Dis; 2017 Nov 01; 1863(11):2762-2771. PubMed ID: 28711596
    [Abstract] [Full Text] [Related]

  • 12. Oxr1 improves pathogenic cellular features of ALS-associated FUS and TDP-43 mutations.
    Finelli MJ, Liu KX, Wu Y, Oliver PL, Davies KE.
    Hum Mol Genet; 2015 Jun 15; 24(12):3529-44. PubMed ID: 25792726
    [Abstract] [Full Text] [Related]

  • 13. Molecular Mechanisms Underlying TDP-43 Pathology in Cellular and Animal Models of ALS and FTLD.
    Wood A, Gurfinkel Y, Polain N, Lamont W, Lyn Rea S.
    Int J Mol Sci; 2021 Apr 29; 22(9):. PubMed ID: 33946763
    [Abstract] [Full Text] [Related]

  • 14. Role and therapeutic potential of liquid-liquid phase separation in amyotrophic lateral sclerosis.
    Pakravan D, Orlando G, Bercier V, Van Den Bosch L.
    J Mol Cell Biol; 2021 Apr 10; 13(1):15-28. PubMed ID: 32976566
    [Abstract] [Full Text] [Related]

  • 15. Genetics of amyotrophic lateral sclerosis: A review.
    Mathis S, Goizet C, Soulages A, Vallat JM, Masson GL.
    J Neurol Sci; 2019 Apr 15; 399():217-226. PubMed ID: 30870681
    [Abstract] [Full Text] [Related]

  • 16. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS, Shim YM, Lee DY, Kim JS, Kang M, Ahn SH, Shin JY, Geum D, Hong YH, Sung JJ.
    Mol Neurobiol; 2019 Mar 15; 56(3):2007-2021. PubMed ID: 29982983
    [Abstract] [Full Text] [Related]

  • 17. Conjoint pathologic cascades mediated by ALS/FTLD-U linked RNA-binding proteins TDP-43 and FUS.
    Ito D, Suzuki N.
    Neurology; 2011 Oct 25; 77(17):1636-43. PubMed ID: 21956718
    [Abstract] [Full Text] [Related]

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  • 19. "STRESSED OUT": The role of FUS and TDP-43 in amyotrophic lateral sclerosis.
    Aksoy YA, Deng W, Stoddart J, Chung R, Guillemin G, Cole NJ, Neely GG, Hesselson D.
    Int J Biochem Cell Biol; 2020 Sep 25; 126():105821. PubMed ID: 32758633
    [Abstract] [Full Text] [Related]

  • 20. The RNA-binding motif 45 (RBM45) protein accumulates in inclusion bodies in amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration with TDP-43 inclusions (FTLD-TDP) patients.
    Collins M, Riascos D, Kovalik T, An J, Krupa K, Krupa K, Hood BL, Conrads TP, Renton AE, Traynor BJ, Bowser R.
    Acta Neuropathol; 2012 Nov 25; 124(5):717-32. PubMed ID: 22993125
    [Abstract] [Full Text] [Related]

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