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Journal Abstract Search

315 related items for PubMed ID: 34834966

  • 1. The Stem-Loop I of Senecavirus A IRES Is Essential for Cap-Independent Translation Activity and Virus Recovery.
    Wang N, Wang H, Shi J, Li C, Liu X, Fan J, Sun C, Cameron CE, Qi H, Yu L.
    Viruses; 2021 Oct 26; 13(11):. PubMed ID: 34834966
    [Abstract] [Full Text] [Related]

  • 2. Modification of the internal ribosome entry site element impairs the growth of foot-and-mouth disease virus in porcine-derived cells.
    Sun C, Yang D, Gao R, Liang T, Wang H, Zhou G, Yu L.
    J Gen Virol; 2016 Apr 26; 97(4):901-911. PubMed ID: 26795299
    [Abstract] [Full Text] [Related]

  • 3. Heterogeneous Nuclear Ribonucleoprotein L Negatively Regulates Foot-and-Mouth Disease Virus Replication through Inhibition of Viral RNA Synthesis by Interacting with the Internal Ribosome Entry Site in the 5' Untranslated Region.
    Sun C, Liu M, Chang J, Yang D, Zhao B, Wang H, Zhou G, Weng C, Yu L.
    J Virol; 2020 May 04; 94(10):. PubMed ID: 32161169
    [Abstract] [Full Text] [Related]

  • 4. Nucleolin Promotes IRES-Driven Translation of Foot-and-Mouth Disease Virus by Supporting the Assembly of Translation Initiation Complexes.
    Han S, Wang X, Guan J, Wu J, Zhang Y, Li P, Liu Z, Abdullah SW, Zhang Z, Jin Y, Sun S, Guo H.
    J Virol; 2021 Jun 10; 95(13):e0023821. PubMed ID: 33853964
    [Abstract] [Full Text] [Related]

  • 5. Experimental evidence for occurrence of putative copy-choice recombination between two Senecavirus A genomes.
    Liu F, Wang Q, Meng H, Zhao D, Hao X, Zhang S, Lu J, Shan H.
    Vet Microbiol; 2022 Aug 10; 271():109487. PubMed ID: 35714527
    [Abstract] [Full Text] [Related]

  • 6. Advances and Breakthroughs in IRES-Directed Translation and Replication of Picornaviruses.
    Abdullah SW, Wu J, Wang X, Guo H, Sun S.
    mBio; 2023 Apr 25; 14(2):e0035823. PubMed ID: 36939331
    [Abstract] [Full Text] [Related]

  • 7. Stem II-disrupting pseudoknot does not abolish ability of Senecavirus A IRES to initiate protein expression, but inhibits recovery of virus from cDNA clone.
    Liu F, Wang N, Huang Y, Wang Q, Shan H.
    Vet Microbiol; 2021 Apr 25; 255():109024. PubMed ID: 33713975
    [Abstract] [Full Text] [Related]

  • 8. VP3 protein of Senecavirus A promotes viral IRES-driven translation and attenuates innate immunity by specifically relocalizing hnRNPA2B1.
    Li L, Li X, Zhong H, Li M, Wan B, He W, Zhang Y, Du Y, Chen D, Zhang W, Ji P, Jiang D, Han S.
    J Virol; 2024 Sep 17; 98(9):e0122724. PubMed ID: 39207136
    [Abstract] [Full Text] [Related]

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  • 10. hnRNP K Is a Novel Internal Ribosomal Entry Site-Transacting Factor That Negatively Regulates Foot-and-Mouth Disease Virus Translation and Replication and Is Antagonized by Viral 3C Protease.
    Liu W, Yang D, Sun C, Wang H, Zhao B, Zhou G, Yu L.
    J Virol; 2020 Aug 17; 94(17):. PubMed ID: 32581104
    [Abstract] [Full Text] [Related]

  • 11. Mutational Analysis of the Bovine Hepacivirus Internal Ribosome Entry Site.
    Baron AL, Schoeniger A, Becher P, Baechlein C.
    J Virol; 2018 Aug 01; 92(15):. PubMed ID: 29769341
    [Abstract] [Full Text] [Related]

  • 12. Impacts of single nucleotide deletions from the 3' end of Senecavirus A 5' untranslated region on activity of viral IRES and on rescue of recombinant virus.
    Liu F, Wang Q, Wang N, Shan H.
    Virology; 2021 Nov 01; 563():126-133. PubMed ID: 34530232
    [Abstract] [Full Text] [Related]

  • 13. Poly(C)-binding protein 2 interacts with sequences required for viral replication in the hepatitis C virus (HCV) 5' untranslated region and directs HCV RNA replication through circularizing the viral genome.
    Wang L, Jeng KS, Lai MM.
    J Virol; 2011 Aug 01; 85(16):7954-64. PubMed ID: 21632751
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  • 16. A Temperature-Dependent Translation Defect Caused by Internal Ribosome Entry Site Mutation Attenuates Foot-and-Mouth Disease Virus: Implications for Rational Vaccine Design.
    Yang D, Sun C, Gao R, Wang H, Liu W, Yu K, Zhou G, Zhao B, Yu L.
    J Virol; 2020 Jul 30; 94(16):. PubMed ID: 32493820
    [Abstract] [Full Text] [Related]

  • 17. Eukaryotic initiation factor 4G-poly(A) binding protein interaction is required for poly(A) tail-mediated stimulation of picornavirus internal ribosome entry segment-driven translation but not for X-mediated stimulation of hepatitis C virus translation.
    Michel YM, Borman AM, Paulous S, Kean KM.
    Mol Cell Biol; 2001 Jul 30; 21(13):4097-109. PubMed ID: 11390639
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  • 19. Hepatitis C virus internal ribosome entry site (IRES) stem loop IIId contains a phylogenetically conserved GGG triplet essential for translation and IRES folding.
    Jubin R, Vantuno NE, Kieft JS, Murray MG, Doudna JA, Lau JY, Baroudy BM.
    J Virol; 2000 Nov 30; 74(22):10430-7. PubMed ID: 11044087
    [Abstract] [Full Text] [Related]

  • 20. Distinct roles for the IIId2 sub-domain in pestivirus and picornavirus internal ribosome entry sites.
    Willcocks MM, Zaini S, Chamond N, Ulryck N, Allouche D, Rajagopalan N, Davids NA, Fahnøe U, Hadsbjerg J, Rasmussen TB, Roberts LO, Sargueil B, Belsham GJ, Locker N.
    Nucleic Acids Res; 2017 Dec 15; 45(22):13016-13028. PubMed ID: 29069411
    [Abstract] [Full Text] [Related]

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