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Journal Abstract Search

135 related items for PubMed ID: 3489143

  • 1. Comparative studies on B cell reactivities in two X-linked immunodeficient mice to the B cell-stimulating factors.
    Hayashi S, Tomita S, Kanatani T, Takatsu K, Ono S, Hamaoka T.
    Lymphokine Res; 1986; 5(3):229-38. PubMed ID: 3489143
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  • 2. Polyclonal B cell activation by a B cell differentiation factor, B151-TRF2. III. B151-TRF2 as a B cell differentiation factor closely associated with autoimmune disease.
    Dobashi K, Ono S, Murakami S, Takahama Y, Katoh Y, Hamaoka T.
    J Immunol; 1987 Feb 01; 138(3):780-7. PubMed ID: 3543118
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  • 3. Identification of two distinct factors, B151-TRF1 and B151-TRF2, inducing differentiation of activated B cells and small resting B cells into antibody-producing cells.
    Ono S, Hayashi S, Takahama Y, Dobashi K, Katoh Y, Nakanishi K, Paul WE, Hamaoka T.
    J Immunol; 1986 Jul 01; 137(1):187-96. PubMed ID: 3519774
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  • 4. Different effect of prostaglandin E2 on B-cell activation by two distinct B-cell differentiation factors, B151-TRF1/IL-5 and B151-TRF2: selective inhibition of B151-TRF2-induced antibody response through increases in intracellular cyclic AMP levels.
    Ishihara K, Ono S, Takahama Y, Hirayama F, Hirano H, Itoh K, Dobashi K, Murakami S, Katoh Y, Yamaguchi M.
    Immunology; 1989 Oct 01; 68(2):154-62. PubMed ID: 2553585
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  • 6. Polyclonal B cell activation by a B cell differentiation factor, B151-TRF2. II. Evidence for interaction of B151-TRF2 with glycoprotein on B cell membrane via recognition of terminal N-acetyl-D-glucosamine residue(s).
    Katoh Y, Ono S, Takahama Y, Miyake K, Hamaoka T.
    J Immunol; 1986 Nov 01; 137(9):2871-7. PubMed ID: 3489778
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  • 7. Evidence for existence of two distinct TNP-Ficoll-responsive B cell subpopulations preferentially reactive either to a T cell-replacing factor (B151-TRF) or to a signal from accessory cells.
    Hayashi S, Ono S, Miyake K, Tsukada S, Takatsu K, Hamaoka T.
    J Immunol; 1986 Mar 15; 136(6):2069-75. PubMed ID: 3485139
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  • 9. Polyclonal B-cell activation by a B-cell differentiation factor B151-TRF2. IV. B151-TRF2-responsive F1 B cells consist of two separate populations capable of recognizing only one of the parental I-A products expressed on B cells.
    Takaháma Y, Ono S, Glimcher LH, Hamaoka T.
    J Mol Cell Immunol; 1987 Mar 15; 3(3):177-94. PubMed ID: 3509923
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  • 10. Ia-restricted B-B cell interaction. I. The MHC haplotype of bone marrow cells present during B cell ontogeny dictates the self-recognition specificity of B cells in the polyclonal B cell activation by a B cell differentiation factor, B151-TRF2.
    Ono S, Takahama Y, Hamaoka T.
    J Immunol; 1987 Nov 15; 139(10):3213-23. PubMed ID: 3316381
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  • 11. LPS-activated CBA/N mouse B cells respond to anti-Ig and a BSF-1-like factor.
    Howard M, Pesavento P, Stein P.
    J Immunol; 1986 Jun 15; 136(12):4531-7. PubMed ID: 3486901
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  • 13. In vitro studies of the genetically determined unresponsiveness to thymus-independent antigens in CBA/N mice.
    Cohen PL, Scher I, Mosier DE.
    J Immunol; 1976 Feb 15; 116(2):301-4. PubMed ID: 55435
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  • 17. Establishment of three PPD-reactive helper T cell clones with distinct functions in B cell activation.
    Sano Y, Yamada G, Dobashi K, Mizuochi T, Hamaoka T, Takatsu K.
    J Immunol; 1984 Aug 15; 133(2):629-35. PubMed ID: 6234353
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  • 20. Role of N-acetyl-D-galactosamine residue on B151K12-derived T cell-replacing factor (B151-TRF) molecule in B cell-receptor binding and -stimulating activity.
    Takahama Y, Ono S, Hara Y, Hayashi S, Dobashi K, Hamaoka T.
    J Immunol; 1985 Oct 15; 135(4):2534-40. PubMed ID: 3897376
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