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Journal Abstract Search

360 related items for PubMed ID: 35033142

  • 1. Anti-high-mobility group box-1 (HMGB1) mediates the apoptosis of alveolar epithelial cells (AEC) by receptor of advanced glycation end-products (RAGE)/c-Jun N-terminal kinase (JNK) pathway in the rats of crush injuries.
    Zhang BF, Song W, Wang J, Wen PF, Zhang YM.
    J Orthop Surg Res; 2022 Jan 15; 17(1):20. PubMed ID: 35033142
    [Abstract] [Full Text] [Related]

  • 2. HMGB1/RAGE pro-inflammatory axis promotes vascular endothelial cell apoptosis in limb ischemia/reperfusion injury.
    Mi L, Zhang Y, Xu Y, Zheng X, Zhang X, Wang Z, Xue M, Jin X.
    Biomed Pharmacother; 2019 Aug 15; 116():109005. PubMed ID: 31136947
    [Abstract] [Full Text] [Related]

  • 3. Anti-high mobility group box-1 (HMGB1) antibody attenuates kidney damage following experimental crush injury and the possible role of the tumor necrosis factor-α and c-Jun N-terminal kinase pathway.
    Zhang BF, Wang PF, Cong YX, Lei JL, Wang H, Huang H, Han S, Zhuang Y.
    J Orthop Surg Res; 2017 Jul 12; 12(1):110. PubMed ID: 28701229
    [Abstract] [Full Text] [Related]

  • 4. HMGB1 may act via RAGE to promote angiogenesis in the later phase after intracerebral hemorrhage.
    Lei C, Zhang S, Cao T, Tao W, Liu M, Wu B.
    Neuroscience; 2015 Jun 04; 295():39-47. PubMed ID: 25813710
    [Abstract] [Full Text] [Related]

  • 5. The HMGB1-RAGE axis induces apoptosis in acute respiratory distress syndrome through PERK/eIF2α/ATF4-mediated endoplasmic reticulum stress.
    He F, Gu L, Cai N, Ni J, Liu Y, Zhang Q, Wu C.
    Inflamm Res; 2022 Nov 04; 71(10-11):1245-1260. PubMed ID: 35871648
    [Abstract] [Full Text] [Related]

  • 6. Activation of the high-mobility group box 1 protein-receptor for advanced glycation end-products signaling pathway in rats during neurogenesis after intracerebral hemorrhage.
    Lei C, Wu B, Cao T, Zhang S, Liu M.
    Stroke; 2015 Feb 04; 46(2):500-6. PubMed ID: 25538203
    [Abstract] [Full Text] [Related]

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  • 8. The receptor for advanced glycation end-products enhances lung epithelial wound repair: An in vitro study.
    Zhai R, Blondonnet R, Ebrahimi E, Belville C, Audard J, Gross C, Choltus H, Henrioux F, Constantin JM, Pereira B, Blanchon L, Sapin V, Jabaudon M.
    Exp Cell Res; 2020 Jun 15; 391(2):112030. PubMed ID: 32330509
    [Abstract] [Full Text] [Related]

  • 9. HMGB1 promotes neurovascular remodeling via Rage in the late phase of subarachnoid hemorrhage.
    Tian X, Sun L, Feng D, Sun Q, Dou Y, Liu C, Zhou F, Li H, Shen H, Wang Z, Chen G.
    Brain Res; 2017 Sep 01; 1670():135-145. PubMed ID: 28606778
    [Abstract] [Full Text] [Related]

  • 10. High-mobility group box 1 protein is implicated in advanced glycation end products-induced vascular endothelial growth factor A production in the rat retinal ganglion cell line RGC-5.
    Lee JJ, Hsiao CC, Yang IH, Chou MH, Wu CL, Wei YC, Chen CH, Chuang JH.
    Mol Vis; 2012 Sep 01; 18():838-50. PubMed ID: 22511847
    [Abstract] [Full Text] [Related]

  • 11. Inhibition of the receptor for advanced glycation inhibits lipopolysaccharide-mediated High mobility group protein B1 and Interleukin-6 synthesis in human gingival fibroblasts through the NF-κB signaling pathway.
    Huang J, Xiong T, Zhang Z, Tan Y, Guo L.
    Arch Oral Biol; 2019 Sep 01; 105():81-87. PubMed ID: 31288145
    [Abstract] [Full Text] [Related]

  • 12. HMGB1 contributes to the irradiation-induced endothelial barrier injury through receptor for advanced glycation endproducts (RAGE).
    Zhou H, Jin C, Cui L, Xing H, Liu J, Liao W, Liao H, Yu Y.
    J Cell Physiol; 2018 Sep 01; 233(9):6714-6721. PubMed ID: 29215715
    [Abstract] [Full Text] [Related]

  • 13. Dietary advanced glycation end-products, its pulmonary receptor, and high mobility group box 1 in aspiration lung injury.
    Smit PJ, Guo WA, Davidson BA, Mullan BA, Helinski JD, Knight PR.
    J Surg Res; 2014 Sep 01; 191(1):214-23. PubMed ID: 24814199
    [Abstract] [Full Text] [Related]

  • 14. The activation of HMGB1 as a progression factor on inflammation response in normal human bronchial epithelial cells through RAGE/JNK/NF-κB pathway.
    Wu X, Mi Y, Yang H, Hu A, Zhang Q, Shang C.
    Mol Cell Biochem; 2013 Aug 01; 380(1-2):249-57. PubMed ID: 23712703
    [Abstract] [Full Text] [Related]

  • 15. Inhibition of the HMGB1/RAGE axis protects against cisplatin-induced ototoxicity via suppression of inflammation and oxidative stress.
    Qiao X, Li W, Zheng Z, Liu C, Zhao L, He Y, Li H.
    Int J Biol Sci; 2024 Aug 01; 20(2):784-800. PubMed ID: 38169643
    [Abstract] [Full Text] [Related]

  • 16. Electroacupuncture protects against the striatum of ischemia stroke by inhibiting the HMGB1/RAGE/p-JNK signaling pathways.
    Nie Z, Hu C, Miao H, Wu F.
    J Chem Neuroanat; 2024 Mar 01; 136():102376. PubMed ID: 38123001
    [Abstract] [Full Text] [Related]

  • 17. High-mobility group box 1 released from astrocytes promotes the proliferation of cultured neural stem/progenitor cells.
    Li M, Sun L, Luo Y, Xie C, Pang Y, Li Y.
    Int J Mol Med; 2014 Sep 01; 34(3):705-14. PubMed ID: 24970310
    [Abstract] [Full Text] [Related]

  • 18. Ethyl Pyruvate Alleviates Pulmonary Hypertension through the Suppression of Pulmonary Artery Smooth Muscle Cell Proliferation via the High Mobility Group Protein B1/Receptor for Advanced Glycation End-Products Axis.
    Liu C, Sun H, Tang M, Li J, Zhang X, Cao G.
    Ann Thorac Cardiovasc Surg; 2021 Dec 20; 27(6):380-388. PubMed ID: 34011805
    [Abstract] [Full Text] [Related]

  • 19. High-mobility group box 1 facilitates migration of neural stem cells via receptor for advanced glycation end products signaling pathway.
    Xue X, Chen X, Fan W, Wang G, Zhang L, Chen Z, Liu P, Liu M, Zhao J.
    Sci Rep; 2018 Mar 14; 8(1):4513. PubMed ID: 29540727
    [Abstract] [Full Text] [Related]

  • 20. Systemic involvement of high-mobility group box 1 protein and therapeutic effect of anti-high-mobility group box 1 protein antibody in a rat model of crush injury.
    Shimazaki J, Matsumoto N, Ogura H, Muroya T, Kuwagata Y, Nakagawa J, Yamakawa K, Hosotsubo H, Imamura Y, Shimazu T.
    Shock; 2012 Jun 14; 37(6):634-8. PubMed ID: 22392147
    [Abstract] [Full Text] [Related]

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