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9. Accumulation of amyloid-β in the brain of mouse models of Alzheimer's disease is modified by altered gene expression in the presence of human apoE isoforms during aging. Honda K, Saito Y, Saito H, Toyoda M, Abe R, Saito T, Saido TC, Michikawa M, Taru H, Sobu Y, Hata S, Nakaya T, Suzuki T. Neurobiol Aging; 2023 Mar 07; 123():63-74. PubMed ID: 36638682 [Abstract] [Full Text] [Related]
13. APOE4-mediated amyloid-β pathology depends on its neuronal receptor LRP1. Tachibana M, Holm ML, Liu CC, Shinohara M, Aikawa T, Oue H, Yamazaki Y, Martens YA, Murray ME, Sullivan PM, Weyer K, Glerup S, Dickson DW, Bu G, Kanekiyo T. J Clin Invest; 2019 Mar 01; 129(3):1272-1277. PubMed ID: 30741718 [Abstract] [Full Text] [Related]
14. Differential Signaling Mediated by ApoE2, ApoE3, and ApoE4 in Human Neurons Parallels Alzheimer's Disease Risk. Huang YA, Zhou B, Nabet AM, Wernig M, Südhof TC. J Neurosci; 2019 Sep 11; 39(37):7408-7427. PubMed ID: 31331998 [Abstract] [Full Text] [Related]
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19. Apolipoprotein E4 influences amyloid deposition but not cell loss after traumatic brain injury in a mouse model of Alzheimer's disease. Hartman RE, Laurer H, Longhi L, Bales KR, Paul SM, McIntosh TK, Holtzman DM. J Neurosci; 2002 Dec 01; 22(23):10083-7. PubMed ID: 12451108 [Abstract] [Full Text] [Related]
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