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Journal Abstract Search


265 related items for PubMed ID: 3543637

  • 1.
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  • 2. 1alpha(OH)D3 One-alpha-hydroxy-cholecalciferol--an active vitamin D analog. Clinical studies on prophylaxis and treatment of secondary hyperparathyroidism in uremic patients on chronic dialysis.
    Brandi L.
    Dan Med Bull; 2008 Nov; 55(4):186-210. PubMed ID: 19232159
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  • 3. Dialysis vintage and parathyroid hormone level, not fibroblast growth factor-23, determines chronic-phase phosphate wasting after renal transplantation.
    Tomida K, Hamano T, Ichimaru N, Fujii N, Matsui I, Nonomura N, Tsubakihara Y, Rakugi H, Takahara S, Isaka Y.
    Bone; 2012 Oct; 51(4):729-36. PubMed ID: 22796419
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  • 4. Absence of effect of 24,25-dihydroxycholecalciferol on serum immunoreactive PTH in patients with persistent hyperparathyroidism after renal transplantation.
    Friedlander MA, Horst RL, Hawker CD.
    Clin Nephrol; 1984 Oct; 22(4):206-10. PubMed ID: 6391769
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  • 7. Dissociation between the effects of endogenous parathyroid hormone on adenosine 3',5'-monophosphate generation and phosphate reabsorption in hypocalcemia due to vitamin D depletion: an acquired disorder resembling pseudohypoparathyroidism type II.
    Rao DS, Parfitt AM, Kleerekoper M, Pumo BS, Frame B.
    J Clin Endocrinol Metab; 1985 Aug; 61(2):285-90. PubMed ID: 2989317
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  • 10. Metabolic aspects of phosphate replacement therapy for hypophosphatemia after renal transplantation: impact on muscular phosphate content, mineral metabolism, and acid/base homeostasis.
    Ambühl PM, Meier D, Wolf B, Dydak U, Boesiger P, Binswanger U.
    Am J Kidney Dis; 1999 Nov; 34(5):875-83. PubMed ID: 10561144
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  • 11. Handling of phosphate by the transplanted kidney.
    Graf H, Kovarik J, Stummvoll HK, Wolf A, Pinggera WF.
    Proc Eur Dial Transplant Assoc; 1979 Nov; 16():624-9. PubMed ID: 398523
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  • 13. Changes in blood pressure and renal function following subtotal parathyroidectomy in renal transplant patients presenting with persistent hypercalcemic hyperparathyroidism.
    Rostaing L, Moreau-Gaudry X, Baron E, Cisterne JM, Monroziès-Bernadet P, Durand D.
    Clin Nephrol; 1997 Apr; 47(4):248-55. PubMed ID: 9128792
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  • 14. Phosphate metabolism in renal stone formers. (II): Relation to renal tubular functions and calcium metabolism.
    Wikström B, Backman U, Danielson BG, Fellström B, Johansson G, Ljunghall S, Wide L.
    Scand J Urol Nephrol Suppl; 1981 Apr; 61():II:1-26. PubMed ID: 6274002
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  • 15. Metabolic studies in a patient with idiopathic hypophosphatemic osteomalacia.
    Weiss M, Schechter P, Bab I, Mekhmandarov S, Bank H, Eisenstein Z.
    Isr J Med Sci; 1988 Jan; 24(1):46-50. PubMed ID: 3346150
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  • 17. Reelevation of parathyroid hormone level after parathyroidectomy in patients with primary hyperparathyroidism: importance of decreased renal parathyroid hormone sensitivity.
    Yamashita H, Noguchi S, Moriyama T, Takamatsu Y, Sadanaga K, Uchino S, Watanabe S, Ogawa T.
    Surgery; 2005 Apr; 137(4):419-25. PubMed ID: 15800489
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  • 18. In vivo studies of parathyroid gland function in secondary hyperparathyroidism.
    Salusky IB, Goodman WG.
    Adv Nephrol Necker Hosp; 1996 Apr; 25():289-302. PubMed ID: 8717632
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  • 19. Acute oral calcium load decreases parathyroid secretion and suppresses tubular phosphate loss in long-term renal transplant recipients.
    Dumoulin G, Hory B, Nguyen NU, Henriet MT, Bresson C, Regnard J, Saint-Hillier Y.
    Am J Nephrol; 1995 Apr; 15(3):238-44. PubMed ID: 7618649
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