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Journal Abstract Search

192 related items for PubMed ID: 35912995

  • 1. Dissociation of SH3 and cysteine-rich domain 3 and junctophilin 1 from dihydropyridine receptor in dystrophin-deficient muscles.
    Ashida Y, Himori K, Tokuda N, Naito A, Yamauchi N, Takenaka-Ninagawa N, Aoki Y, Sakurai H, Yamada T.
    Am J Physiol Cell Physiol; 2022 Sep 01; 323(3):C885-C895. PubMed ID: 35912995
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  • 2. Role of calpain in eccentric contraction-induced proteolysis of Ca2+-regulatory proteins and force depression in rat fast-twitch skeletal muscle.
    Kanzaki K, Watanabe D, Kuratani M, Yamada T, Matsunaga S, Wada M.
    J Appl Physiol (1985); 2017 Feb 01; 122(2):396-405. PubMed ID: 27979982
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  • 3. Ca2+-dependent proteolysis of junctophilin-1 and junctophilin-2 in skeletal and cardiac muscle.
    Murphy RM, Dutka TL, Horvath D, Bell JR, Delbridge LM, Lamb GD.
    J Physiol; 2013 Feb 01; 591(3):719-29. PubMed ID: 23148318
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  • 4. Preconditioning contractions prevent prolonged force depression and Ca2+-dependent proteolysis of STAC3 after damaging eccentric contractions.
    Ashida Y, Himori K, Tamai K, Kimura I, Yamada T.
    J Appl Physiol (1985); 2021 Nov 01; 131(5):1399-1407. PubMed ID: 34590910
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  • 5. STAC3 incorporation into skeletal muscle triads occurs independent of the dihydropyridine receptor.
    Campiglio M, Kaplan MM, Flucher BE.
    J Cell Physiol; 2018 Dec 01; 233(12):9045-9051. PubMed ID: 30071129
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  • 6. Cell transplantation-mediated dystrophin supplementation efficacy in Duchenne muscular dystrophy mouse motor function improvement demonstrated by enhanced skeletal muscle fatigue tolerance.
    Bourgeois Yoshioka CK, Takenaka-Ninagawa N, Goto M, Miki M, Watanabe D, Yamamoto M, Aoyama T, Sakurai H.
    Stem Cell Res Ther; 2024 Sep 19; 15(1):313. PubMed ID: 39300595
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  • 8. Transport of the alpha subunit of the voltage gated L-type calcium channel through the sarcoplasmic reticulum occurs prior to localization to triads and requires the beta subunit but not Stac3 in skeletal muscles.
    Linsley JW, Hsu IU, Wang W, Kuwada JY.
    Traffic; 2017 Sep 19; 18(9):622-632. PubMed ID: 28697281
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  • 9. Alterations in Notch signalling in skeletal muscles from mdx and dko dystrophic mice and patients with Duchenne muscular dystrophy.
    Church JE, Trieu J, Chee A, Naim T, Gehrig SM, Lamon S, Angelini C, Russell AP, Lynch GS.
    Exp Physiol; 2014 Apr 19; 99(4):675-87. PubMed ID: 24443351
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  • 10. Congenital myopathy results from misregulation of a muscle Ca2+ channel by mutant Stac3.
    Linsley JW, Hsu IU, Groom L, Yarotskyy V, Lavorato M, Horstick EJ, Linsley D, Wang W, Franzini-Armstrong C, Dirksen RT, Kuwada JY.
    Proc Natl Acad Sci U S A; 2017 Jan 10; 114(2):E228-E236. PubMed ID: 28003463
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  • 11. Muscle-specific overexpression of IGF-I improves E-C coupling in skeletal muscle fibers from dystrophic mdx mice.
    Schertzer JD, van der Poel C, Shavlakadze T, Grounds MD, Lynch GS.
    Am J Physiol Cell Physiol; 2008 Jan 10; 294(1):C161-8. PubMed ID: 17989207
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  • 13. Isometric resistance training increases strength and alters histopathology of dystrophin-deficient mouse skeletal muscle.
    Lindsay A, Larson AA, Verma M, Ervasti JM, Lowe DA.
    J Appl Physiol (1985); 2019 Feb 01; 126(2):363-375. PubMed ID: 30571283
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  • 18. l-arginine ingestion inhibits eccentric contraction-induced proteolysis and force deficit via S-nitrosylation of calpain.
    Kanzaki K, Watanabe D, Aibara C, Kawakami Y, Yamada T, Takahashi Y, Wada M.
    Physiol Rep; 2018 Jan 01; 6(2):. PubMed ID: 29368397
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  • 19. Junctophilin damage contributes to early strength deficits and EC coupling failure after eccentric contractions.
    Corona BT, Balog EM, Doyle JA, Rupp JC, Luke RC, Ingalls CP.
    Am J Physiol Cell Physiol; 2010 Feb 01; 298(2):C365-76. PubMed ID: 19940065
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