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216 related items for PubMed ID: 36004695
1. Analysis of Hypoxia Inducible Factor-1α Expression and Its Effects on Glycolysis of Esophageal Carcinoma. Chen X, Luo C, Bai Y, Yao L, Shanzhou Q, Xie Y, Wang S, Xu L, Guo X, Zhong X, Wu Q. Crit Rev Eukaryot Gene Expr; 2022; 32(7):47-66. PubMed ID: 36004695 [Abstract] [Full Text] [Related]
2. Wortmannin influences hypoxia-inducible factor-1 alpha expression and glycolysis in esophageal carcinoma cells. Zeng L, Zhou HY, Tang NN, Zhang WF, He GJ, Hao B, Feng YD, Zhu H. World J Gastroenterol; 2016 May 28; 22(20):4868-80. PubMed ID: 27239113 [Abstract] [Full Text] [Related]
4. Hypoxia-inducible transcription factor-1alpha promotes hypoxia-induced A549 apoptosis via a mechanism that involves the glycolysis pathway. Luo F, Liu X, Yan N, Li S, Cao G, Cheng Q, Xia Q, Wang H. BMC Cancer; 2006 Jan 27; 6():26. PubMed ID: 16438736 [Abstract] [Full Text] [Related]
5. Excess glucose induces hypoxia-inducible factor-1α in pancreatic cancer cells and stimulates glucose metabolism and cell migration. Liu Z, Jia X, Duan Y, Xiao H, Sundqvist KG, Permert J, Wang F. Cancer Biol Ther; 2013 May 27; 14(5):428-35. PubMed ID: 23377827 [Abstract] [Full Text] [Related]
7. HIF-1α induces VE-cadherin expression and modulates vasculogenic mimicry in esophageal carcinoma cells. Tang NN, Zhu H, Zhang HJ, Zhang WF, Jin HL, Wang L, Wang P, He GJ, Hao B, Shi RH. World J Gastroenterol; 2014 Dec 21; 20(47):17894-904. PubMed ID: 25548487 [Abstract] [Full Text] [Related]
8. FBP1 modulates cell metabolism of breast cancer cells by inhibiting the expression of HIF-1α. Shi L, He C, Li Z, Wang Z, Zhang Q. Neoplasma; 2017 Dec 21; 64(4):535-542. PubMed ID: 28485159 [Abstract] [Full Text] [Related]
9. Hypoxia-Induced Ephrin-B2 Facilitates Proliferation and Induces Glycolytic Metabolism in Cutaneous Squamous Cell Carcinoma by Modulating PKM2/HIF-1α Axis. Wang X, Zhang Z, Hu G, Zhou X. Discov Med; 2024 Aug 21; 36(187):1692-1702. PubMed ID: 39190384 [Abstract] [Full Text] [Related]
10. Hypoxia-inducible Factor-1α Mediates Hyperglycemia-induced Pancreatic Cancer Glycolysis. Cheng L, Qin T, Ma J, Duan W, Xu Q, Li X, Han L, Li W, Wang Z, Zhang D, Ma Q, Lei J. Anticancer Agents Med Chem; 2019 Aug 21; 19(12):1503-1512. PubMed ID: 31241439 [Abstract] [Full Text] [Related]
15. LINC00365 functions as a tumor suppressor by inhibiting HIF-1α-mediated glucose metabolism reprogramming in breast cancer. Liu B, Qu X, Wang J, Xu L, Zhang L, Xu B, Su J, Bian X. Exp Cell Res; 2023 Apr 01; 425(1):113514. PubMed ID: 36804531 [Abstract] [Full Text] [Related]
16. Qiliqiangxin attenuates hypoxia-induced injury in primary rat cardiac microvascular endothelial cells via promoting HIF-1α-dependent glycolysis. Wang Y, Han X, Fu M, Wang J, Song Y, Liu Y, Zhang J, Zhou J, Ge J. J Cell Mol Med; 2018 May 01; 22(5):2791-2803. PubMed ID: 29502357 [Abstract] [Full Text] [Related]
17. Effect of heat-reinforcing needling on hypoxia-inducible factor 1α and glycolysis activity in rabbits with cold syndrome of rheumatoid arthritis. Li XJ, Li FX, Jing WY, Liu C, Liu LM, Zhang XH, Zhang FF, Chen P, DU XZ. Zhen Ci Yan Jiu; 2024 Jun 25; 49(6):585-593. PubMed ID: 38897802 [Abstract] [Full Text] [Related]
18. The Promoting Effect of Radiation on Glucose Metabolism in Breast Cancer Cells under the Treatment of Cobalt Chloride. Zhao CB, Shi L, Pu HH, Zhang QY. Pathol Oncol Res; 2017 Jan 25; 23(1):47-53. PubMed ID: 27342248 [Abstract] [Full Text] [Related]
19. Role of hypoxia-inducible factor (HIF)-1alpha versus HIF-2alpha in the regulation of HIF target genes in response to hypoxia, insulin-like growth factor-I, or loss of von Hippel-Lindau function: implications for targeting the HIF pathway. Carroll VA, Ashcroft M. Cancer Res; 2006 Jun 15; 66(12):6264-70. PubMed ID: 16778202 [Abstract] [Full Text] [Related]
20. Orexin A affects HepG2 human hepatocellular carcinoma cells glucose metabolism via HIF-1α-dependent and -independent mechanism. Wan X, Liu Y, Zhao Y, Sun X, Fan D, Guo L. PLoS One; 2017 Jun 15; 12(9):e0184213. PubMed ID: 28886081 [Abstract] [Full Text] [Related] Page: [Next] [New Search]