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Journal Abstract Search

655 related items for PubMed ID: 36008843

  • 1. Co-deposition of SOD1, TDP-43 and p62 proteinopathies in ALS: evidence for multifaceted pathways underlying neurodegeneration.
    Trist BG, Fifita JA, Hogan A, Grima N, Smith B, Troakes C, Vance C, Shaw C, Al-Sarraj S, Blair IP, Double KL.
    Acta Neuropathol Commun; 2022 Aug 25; 10(1):122. PubMed ID: 36008843
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  • 2. TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutations.
    Maekawa S, Leigh PN, King A, Jones E, Steele JC, Bodi I, Shaw CE, Hortobagyi T, Al-Sarraj S.
    Neuropathology; 2009 Dec 25; 29(6):672-83. PubMed ID: 19496940
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  • 3. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ, Kwong LK, Forman MS, Ravits J, Stewart H, Eisen A, McClusky L, Kretzschmar HA, Monoranu CM, Highley JR, Kirby J, Siddique T, Shaw PJ, Lee VM, Trojanowski JQ.
    Ann Neurol; 2007 May 25; 61(5):427-34. PubMed ID: 17469116
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  • 4. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS, Shim YM, Lee DY, Kim JS, Kang M, Ahn SH, Shin JY, Geum D, Hong YH, Sung JJ.
    Mol Neurobiol; 2019 Mar 25; 56(3):2007-2021. PubMed ID: 29982983
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  • 6. Altered SOD1 maturation and post-translational modification in amyotrophic lateral sclerosis spinal cord.
    Trist BG, Genoud S, Roudeau S, Rookyard A, Abdeen A, Cottam V, Hare DJ, White M, Altvater J, Fifita JA, Hogan A, Grima N, Blair IP, Kysenius K, Crouch PJ, Carmona A, Rufin Y, Claverol S, Van Malderen S, Falkenberg G, Paterson DJ, Smith B, Troakes C, Vance C, Shaw CE, Al-Sarraj S, Cordwell S, Halliday G, Ortega R, Double KL.
    Brain; 2022 Sep 14; 145(9):3108-3130. PubMed ID: 35512359
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  • 7. The Overexpression of TDP-43 Protein in the Neuron and Oligodendrocyte Cells Causes the Progressive Motor Neuron Degeneration in the SOD1 G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis.
    Lu Y, Tang C, Zhu L, Li J, Liang H, Zhang J, Xu R.
    Int J Biol Sci; 2016 Sep 14; 12(9):1140-9. PubMed ID: 27570488
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  • 8. Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis.
    Pokrishevsky E, Grad LI, Yousefi M, Wang J, Mackenzie IR, Cashman NR.
    PLoS One; 2012 Sep 14; 7(4):e35050. PubMed ID: 22493728
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  • 11. MicroRNA-183-5p regulates TAR DNA-binding protein 43 neurotoxicity via SQSTM1/p62 in amyotrophic lateral sclerosis.
    Kim HC, Zhang Y, King PH, Lu L.
    J Neurochem; 2023 Mar 14; 164(5):643-657. PubMed ID: 36527420
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  • 12. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
    Soo KY, Halloran M, Sundaramoorthy V, Parakh S, Toth RP, Southam KA, McLean CA, Lock P, King A, Farg MA, Atkin JD.
    Acta Neuropathol; 2015 Nov 14; 130(5):679-97. PubMed ID: 26298469
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  • 13. Nuclear TAR DNA binding protein 43 expression in spinal cord neurons correlates with the clinical course in amyotrophic lateral sclerosis.
    Sumi H, Kato S, Mochimaru Y, Fujimura H, Etoh M, Sakoda S.
    J Neuropathol Exp Neurol; 2009 Jan 14; 68(1):37-47. PubMed ID: 19104447
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  • 14. Protein misdirection inside and outside motor neurons in Amyotrophic Lateral Sclerosis (ALS): a possible clue for therapeutic strategies.
    Ido A, Fukuyama H, Urushitani M.
    Int J Mol Sci; 2011 Jan 14; 12(10):6980-7003. PubMed ID: 22072931
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  • 15. Functional links between SQSTM1 and ALS2 in the pathogenesis of ALS: cumulative impact on the protection against mutant SOD1-mediated motor dysfunction in mice.
    Hadano S, Mitsui S, Pan L, Otomo A, Kubo M, Sato K, Ono S, Onodera W, Abe K, Chen X, Koike M, Uchiyama Y, Aoki M, Warabi E, Yamamoto M, Ishii T, Yanagawa T, Shang HF, Yoshii F.
    Hum Mol Genet; 2016 Aug 01; 25(15):3321-3340. PubMed ID: 27439389
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  • 16. Lack of TDP-43 abnormalities in mutant SOD1 transgenic mice shows disparity with ALS.
    Robertson J, Sanelli T, Xiao S, Yang W, Horne P, Hammond R, Pioro EP, Strong MJ.
    Neurosci Lett; 2007 Jun 13; 420(2):128-32. PubMed ID: 17543992
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  • 17. An integrative miRNA-mRNA expression analysis identifies miRNA signatures associated with SOD1 and TARDBP patient-derived motor neurons.
    Dash BP, Freischmidt A, Weishaupt JH, Hermann A.
    Hum Mol Genet; 2024 Jul 22; 33(15):1300-1314. PubMed ID: 38676626
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  • 18. Amyotrophic lateral sclerosis is a non-amyloid disease in which extensive misfolding of SOD1 is unique to the familial form.
    Kerman A, Liu HN, Croul S, Bilbao J, Rogaeva E, Zinman L, Robertson J, Chakrabartty A.
    Acta Neuropathol; 2010 Mar 22; 119(3):335-44. PubMed ID: 20111867
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  • 19. ERp57 is protective against mutant SOD1-induced cellular pathology in amyotrophic lateral sclerosis.
    Parakh S, Jagaraj CJ, Vidal M, Ragagnin AMG, Perri ER, Konopka A, Toth RP, Galper J, Blair IP, Thomas CJ, Walker AK, Yang S, Spencer DM, Atkin JD.
    Hum Mol Genet; 2018 Apr 15; 27(8):1311-1331. PubMed ID: 29409023
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  • 20. TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy.
    Turner BJ, Bäumer D, Parkinson NJ, Scaber J, Ansorge O, Talbot K.
    BMC Neurosci; 2008 Oct 28; 9():104. PubMed ID: 18957104
    [Abstract] [Full Text] [Related]

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