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Journal Abstract Search

156 related items for PubMed ID: 36325756

  • 1. 1,3-Diarylpyrazolyl-acylsulfonamides Target HadAB/BC Complex in Mycobacterium tuberculosis.
    Singh V, Grzegorzewicz AE, Fienberg S, Müller R, Khonde LP, Sanz O, Alfonso S, Urones B, Drewes G, Bantscheff M, Ghidelli-Disse S, Ioerger TR, Angala B, Liu J, Lee RE, Sacchettini JC, Krieger IV, Jackson M, Chibale K, Ghorpade SR.
    ACS Infect Dis; 2022 Nov 11; 8(11):2315-2326. PubMed ID: 36325756
    [Abstract] [Full Text] [Related]

  • 2. The missing piece of the type II fatty acid synthase system from Mycobacterium tuberculosis.
    Sacco E, Covarrubias AS, O'Hare HM, Carroll P, Eynard N, Jones TA, Parish T, Daffé M, Bäckbro K, Quémard A.
    Proc Natl Acad Sci U S A; 2007 Sep 11; 104(37):14628-33. PubMed ID: 17804795
    [Abstract] [Full Text] [Related]

  • 3. Mechanisms of Resistance Associated with the Inhibition of the Dehydration Step of Type II Fatty Acid Synthase in Mycobacterium tuberculosis.
    Grzegorzewicz AE, Gee C, Das S, Liu J, Belardinelli JM, Jones V, McNeil MR, Lee RE, Jackson M.
    ACS Infect Dis; 2020 Feb 14; 6(2):195-204. PubMed ID: 31775512
    [Abstract] [Full Text] [Related]

  • 4. A common mechanism of inhibition of the Mycobacterium tuberculosis mycolic acid biosynthetic pathway by isoxyl and thiacetazone.
    Grzegorzewicz AE, Korduláková J, Jones V, Born SE, Belardinelli JM, Vaquié A, Gundi VA, Madacki J, Slama N, Laval F, Vaubourgeix J, Crew RM, Gicquel B, Daffé M, Morbidoni HR, Brennan PJ, Quémard A, McNeil MR, Jackson M.
    J Biol Chem; 2012 Nov 09; 287(46):38434-41. PubMed ID: 23002234
    [Abstract] [Full Text] [Related]

  • 5. Fluorescently labelled thioacetazone for detecting the interaction with Mycobacterium dehydratases HadAB and HadBC.
    Singh BK, Singha M, Basak S, Biswas R, Das AK, Basak A.
    Org Biomol Chem; 2022 Feb 16; 20(7):1444-1452. PubMed ID: 35084426
    [Abstract] [Full Text] [Related]

  • 6. Molecular basis for the inhibition of β-hydroxyacyl-ACP dehydratase HadAB complex from Mycobacterium tuberculosis by flavonoid inhibitors.
    Dong Y, Qiu X, Shaw N, Xu Y, Sun Y, Li X, Li J, Rao Z.
    Protein Cell; 2015 Jul 16; 6(7):504-17. PubMed ID: 26081470
    [Abstract] [Full Text] [Related]

  • 7. Synthesis, antitubercular activity and mechanism of resistance of highly effective thiacetazone analogues.
    Coxon GD, Craig D, Corrales RM, Vialla E, Gannoun-Zaki L, Kremer L.
    PLoS One; 2013 Jul 16; 8(1):e53162. PubMed ID: 23301038
    [Abstract] [Full Text] [Related]

  • 8. Point mutations within the fatty acid synthase type II dehydratase components HadA or HadC contribute to isoxyl resistance in Mycobacterium tuberculosis.
    Gannoun-Zaki L, Alibaud L, Kremer L.
    Antimicrob Agents Chemother; 2013 Jan 16; 57(1):629-32. PubMed ID: 23114755
    [Abstract] [Full Text] [Related]

  • 9. The C-terminal end of mycobacterial HadBC regulates AcpM interaction during the FAS-II pathway: a structural perspective.
    Singh BK, Biswas R, Bhattacharyya S, Basak A, Das AK.
    FEBS J; 2022 Aug 16; 289(16):4963-4980. PubMed ID: 35175661
    [Abstract] [Full Text] [Related]

  • 10. A new dehydratase conferring innate resistance to thiacetazone and intra-amoebal survival of Mycobacterium smegmatis.
    Carrère-Kremer S, Blaise M, Singh VK, Alibaud L, Tuaillon E, Halloum I, van de Weerd R, Guérardel Y, Drancourt M, Takiff H, Geurtsen J, Kremer L.
    Mol Microbiol; 2015 Jun 16; 96(5):1085-102. PubMed ID: 25754266
    [Abstract] [Full Text] [Related]

  • 11. Mycobacterial crypto-AcpM as a tool to investigate the consequence of drug binding on its key FAS II partner enzyme HadAB.
    Singh BK, Biswas R, Basak A, Das AK.
    Biochim Biophys Acta Gen Subj; 2021 Oct 16; 1865(10):129964. PubMed ID: 34252514
    [Abstract] [Full Text] [Related]

  • 12.
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  • 13. Expression, purification and crystallization of the (3R)-hydroxyacyl-ACP dehydratase HadAB complex from Mycobacterium tuberculosis.
    Dong Y, Li J, Qiu X, Yan C, Li X.
    Protein Expr Purif; 2015 Oct 16; 114():115-20. PubMed ID: 26118698
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  • 15. Mutations in the essential FAS II β-hydroxyacyl ACP dehydratase complex confer resistance to thiacetazone in Mycobacterium tuberculosis and Mycobacterium kansasii.
    Belardinelli JM, Morbidoni HR.
    Mol Microbiol; 2012 Nov 16; 86(3):568-79. PubMed ID: 22994892
    [Abstract] [Full Text] [Related]

  • 16. HadBD dehydratase from Mycobacterium tuberculosis fatty acid synthase type II: A singular structure for a unique function.
    Bories P, Rima J, Tranier S, Marcoux J, Grimoire Y, Tomaszczyk M, Launay A, Fata K, Marrakchi H, Burlet-Schiltz O, Mourey L, Ducoux-Petit M, Bardou F, Bon C, Quémard A.
    Protein Sci; 2024 Apr 16; 33(4):e4964. PubMed ID: 38501584
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  • 18. Crystal structure of dehydratase component HadAB complex of mycobacterial FAS-II pathway.
    Biswas R, Dutta A, Dutta D, Hazra D, Banerjee DR, Basak A, Das AK.
    Biochem Biophys Res Commun; 2015 Mar 06; 458(2):369-74. PubMed ID: 25656575
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  • 20. The changes in mycolic acid structures caused by hadC mutation have a dramatic effect on the virulence of Mycobacterium tuberculosis.
    Slama N, Jamet S, Frigui W, Pawlik A, Bottai D, Laval F, Constant P, Lemassu A, Cam K, Daffé M, Brosch R, Eynard N, Quémard A.
    Mol Microbiol; 2016 Feb 06; 99(4):794-807. PubMed ID: 26538472
    [Abstract] [Full Text] [Related]

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