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Journal Abstract Search


132 related items for PubMed ID: 36378362

  • 1. InsP3R-RyR Ca2+ channel crosstalk facilitates arrhythmias in the failing human ventricle.
    Jin X, Amoni M, Gilbert G, Dries E, Doñate Puertas R, Tomar A, Nagaraju CK, Pradhan A, Yule DI, Martens T, Menten R, Vanden Berghe P, Rega F, Sipido K, Roderick HL.
    Basic Res Cardiol; 2022 Nov 15; 117(1):60. PubMed ID: 36378362
    [Abstract] [Full Text] [Related]

  • 2. InsP3R-RyR channel crosstalk augments sarcoplasmic reticulum Ca2+ release and arrhythmogenic activity in post-MI pig cardiomyocytes.
    Jin X, Meletiou A, Chung J, Tilunaite A, Demydenko K, Dries E, Puertas RD, Amoni M, Tomar A, Gilbert G, Claus P, Soeller C, Rajagopal V, Sipido K, Roderick HL.
    J Mol Cell Cardiol; 2023 Jun 15; 179():47-59. PubMed ID: 37003353
    [Abstract] [Full Text] [Related]

  • 3. [Effect of carvedilol on ryanodine receptor in heart failure].
    Li R, Yi QJ, Qian YR, Liu XY.
    Zhonghua Er Ke Za Zhi; 2005 Aug 15; 43(8):603-7. PubMed ID: 16191273
    [Abstract] [Full Text] [Related]

  • 4. T-tubule disruption promotes calcium alternans in failing ventricular myocytes: mechanistic insights from computational modeling.
    Nivala M, Song Z, Weiss JN, Qu Z.
    J Mol Cell Cardiol; 2015 Feb 15; 79():32-41. PubMed ID: 25450613
    [Abstract] [Full Text] [Related]

  • 5. Hyperactive ryanodine receptors in human heart failure and ischaemic cardiomyopathy reside outside of couplons.
    Dries E, Santiago DJ, Gilbert G, Lenaerts I, Vandenberk B, Nagaraju CK, Johnson DM, Holemans P, Roderick HL, Macquaide N, Claus P, Sipido KR.
    Cardiovasc Res; 2018 Sep 01; 114(11):1512-1524. PubMed ID: 29668881
    [Abstract] [Full Text] [Related]

  • 6. Decreased inward rectifying K+ current and increased ryanodine receptor sensitivity synergistically contribute to sustained focal arrhythmia in the intact rabbit heart.
    Myles RC, Wang L, Bers DM, Ripplinger CM.
    J Physiol; 2015 Mar 15; 593(6):1479-93. PubMed ID: 25772297
    [Abstract] [Full Text] [Related]

  • 7. Ca2+ release via InsP3Rs enhances RyR recruitment during Ca2+ transients by increasing dyadic [Ca2+] in cardiomyocytes.
    Demydenko K, Sipido KR, Roderick HL.
    J Cell Sci; 2021 Jul 15; 134(14):. PubMed ID: 34125209
    [Abstract] [Full Text] [Related]

  • 8. Oxidation of ryanodine receptor after ischemia-reperfusion increases propensity of Ca2+ waves during β-adrenergic receptor stimulation.
    Bovo E, Mazurek SR, Zima AV.
    Am J Physiol Heart Circ Physiol; 2018 Oct 01; 315(4):H1032-H1040. PubMed ID: 30028204
    [Abstract] [Full Text] [Related]

  • 9. Inositol 1,4,5-trisphosphate receptors in cardiomyocyte physiology and disease.
    Demydenko K, Ekhteraei-Tousi S, Roderick HL.
    Philos Trans R Soc Lond B Biol Sci; 2022 Nov 21; 377(1864):20210319. PubMed ID: 36189803
    [Abstract] [Full Text] [Related]

  • 10. Elevated InsP3R expression underlies enhanced calcium fluxes and spontaneous extra-systolic calcium release events in hypertrophic cardiac myocytes.
    Harzheim D, Talasila A, Movassagh M, Foo RS, Figg N, Bootman MD, Roderick HL.
    Channels (Austin); 2010 Nov 21; 4(1):67-71. PubMed ID: 19934645
    [Abstract] [Full Text] [Related]

  • 11. Inositol-1,4,5-trisphosphate induced Ca2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts.
    Hohendanner F, Walther S, Maxwell JT, Kettlewell S, Awad S, Smith GL, Lonchyna VA, Blatter LA.
    J Physiol; 2015 Mar 15; 593(6):1459-77. PubMed ID: 25416623
    [Abstract] [Full Text] [Related]

  • 12. Functional local crosstalk of inositol 1,4,5-trisphosphate receptor- and ryanodine receptor-dependent Ca2+ release in atrial cardiomyocytes.
    Wullschleger M, Blanch J, Egger M.
    Cardiovasc Res; 2017 Apr 01; 113(5):542-552. PubMed ID: 28158491
    [Abstract] [Full Text] [Related]

  • 13. Prolonged β-adrenergic stimulation disperses ryanodine receptor clusters in cardiomyocytes and has implications for heart failure.
    Shen X, van den Brink J, Bergan-Dahl A, Kolstad TR, Norden ES, Hou Y, Laasmaa M, Aguilar-Sanchez Y, Quick AP, Espe EKS, Sjaastad I, Wehrens XHT, Edwards AG, Soeller C, Louch WE.
    Elife; 2022 Aug 01; 11():. PubMed ID: 35913125
    [Abstract] [Full Text] [Related]

  • 14. Ryanodine receptor dispersion disrupts Ca2+ release in failing cardiac myocytes.
    Kolstad TR, van den Brink J, MacQuaide N, Lunde PK, Frisk M, Aronsen JM, Norden ES, Cataliotti A, Sjaastad I, Sejersted OM, Edwards AG, Lines GT, Louch WE.
    Elife; 2018 Oct 30; 7():. PubMed ID: 30375974
    [Abstract] [Full Text] [Related]

  • 15. Cardiomyocyte substructure reverts to an immature phenotype during heart failure.
    Lipsett DB, Frisk M, Aronsen JM, Nordén ES, Buonarati OR, Cataliotti A, Hell JW, Sjaastad I, Christensen G, Louch WE.
    J Physiol; 2019 Apr 30; 597(7):1833-1853. PubMed ID: 30707448
    [Abstract] [Full Text] [Related]

  • 16. Dantrolene prevents arrhythmogenic Ca2+ release in heart failure.
    Maxwell JT, Domeier TL, Blatter LA.
    Am J Physiol Heart Circ Physiol; 2012 Feb 15; 302(4):H953-63. PubMed ID: 22180651
    [Abstract] [Full Text] [Related]

  • 17. Inositol 1,4,5-trisphosphate receptor - reactive oxygen signaling domain regulates excitation-contraction coupling in atrial myocytes.
    Varma D, Almeida JFQ, DeSantiago J, Blatter LA, Banach K.
    J Mol Cell Cardiol; 2022 Feb 15; 163():147-155. PubMed ID: 34755642
    [Abstract] [Full Text] [Related]

  • 18. A novel mechanism of tandem activation of ryanodine receptors by cytosolic and SR luminal Ca2+ during excitation-contraction coupling in atrial myocytes.
    Maxwell JT, Blatter LA.
    J Physiol; 2017 Jun 15; 595(12):3835-3845. PubMed ID: 28028837
    [Abstract] [Full Text] [Related]

  • 19. 'Eventless' InsP3-dependent SR-Ca2+ release affecting atrial Ca2+ sparks.
    Horn T, Ullrich ND, Egger M.
    J Physiol; 2013 Apr 15; 591(8):2103-11. PubMed ID: 23381902
    [Abstract] [Full Text] [Related]

  • 20. Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmias.
    Houser SR.
    Circ Res; 2014 Apr 11; 114(8):1320-7; discussion 1327. PubMed ID: 24723657
    [Abstract] [Full Text] [Related]


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