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178 related items for PubMed ID: 3674464

  • 1. Effects of halothane on myocardial high-energy phosphate metabolism and intracellular pH utilizing 31P NMR spectroscopy.
    Murray PA, Blanck TJ, Rogers MC, Jacobus WE.
    Anesthesiology; 1987 Nov; 67(5):649-53. PubMed ID: 3674464
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  • 2. The effect of halothane on the steady-state levels of high-energy phosphates in the neonatal heart.
    McAuliffe JJ, Hickey PR.
    Anesthesiology; 1987 Aug; 67(2):231-5. PubMed ID: 3605750
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  • 8. High-energy phosphate responses to tachycardia and inotropic stimulation in left ventricular hypertrophy.
    Bache RJ, Zhang J, Path G, Merkle H, Hendrich K, From AH, Ugurbil K.
    Am J Physiol; 1994 May; 266(5 Pt 2):H1959-70. PubMed ID: 8203595
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  • 9. Effect of a novel cardioprotective agent, JTV-519, on metabolism, contraction and relaxation in the ischemia-reperfused rabbit heart.
    Kawabata H, Ryomoto T, Ishikawa K.
    Jpn Circ J; 2000 Oct; 64(10):772-6. PubMed ID: 11059618
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  • 10. 31P-NMR measurements of pHi and high-energy phosphates in isolated turtle hearts during anoxia and acidosis.
    Wasser JS, Inman KC, Arendt EA, Lawler RG, Jackson DC.
    Am J Physiol; 1990 Sep; 259(3 Pt 2):R521-30. PubMed ID: 2396711
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  • 11. Use of gated perfusion to study early effects of anoxia on cardiac energy metabolism: a new 31P NMR method.
    Barbour RL, Sotak CH, Levy GC, Chan SH.
    Biochemistry; 1984 Dec 04; 23(25):6053-62. PubMed ID: 6525343
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  • 12. Influence of propranolol on acidosis and high energy phosphates in ischaemic myocardium of the rabbit.
    Malloy CR, Matthews PM, Smith MB, Radda GK.
    Cardiovasc Res; 1986 Oct 04; 20(10):710-20. PubMed ID: 3791338
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  • 13. Differences in nucleotide compartmentation and energy state in isolated and in situ rat heart: assessment by 31P-NMR spectroscopy.
    Williams JP, Headrick JP.
    Biochim Biophys Acta; 1996 Aug 07; 1276(1):71-9. PubMed ID: 8764892
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  • 14. Improvement in myocardial performance without a decrease in high-energy phosphate metabolites after isoproterenol in Syrian cardiomyopathic hamsters.
    Camacho SA, Wikman-Coffelt J, Wu ST, Watters TA, Botvinick EH, Sievers R, James TL, Jasmin G, Parmley WW.
    Circulation; 1988 Mar 07; 77(3):712-9. PubMed ID: 3342496
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  • 15. Brain energy metabolism and blood flow during sevoflurane and halothane anesthesia: effects of hypocapnia and blood pressure fluctuations.
    Fujibayashi T, Sugiura Y, Yanagimoto M, Harada J, Goto Y.
    Acta Anaesthesiol Scand; 1994 May 07; 38(4):413-8. PubMed ID: 8067234
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  • 16. Glycolysis is necessary to preserve myocardial Ca2+ homeostasis during beta-adrenergic stimulation.
    Nakamura K, Kusuoka H, Ambrosio G, Becker LC.
    Am J Physiol; 1993 Mar 07; 264(3 Pt 2):H670-8. PubMed ID: 8384419
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  • 17. Contractile dysfunction during metabolic acidosis: role of impaired energy metabolism.
    Zhou HZ, Malhotra D, Shapiro JI.
    Am J Physiol; 1991 Nov 07; 261(5 Pt 2):H1481-6. PubMed ID: 1951735
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  • 18. Cyclical changes in high-energy phosphates during the cardiac cycle by pacing-Gated 31P nuclear magnetic resonance.
    Honda H, Tanaka K, Akita N, Haneda T.
    Circ J; 2002 Jan 07; 66(1):80-6. PubMed ID: 11999671
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  • 19. The effects of halothane and isoflurane on the phosphoenergetic state of the liver during hemorrhagic shock in rats: an in vivo 31P nuclear magnetic resonance spectroscopic study.
    Takahashi K, Shigemori S, Nosaka S, Morikawa S, Inubushi T.
    Anesth Analg; 1997 Aug 07; 85(2):347-52. PubMed ID: 9249112
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  • 20. 31P-NMR of high-energy phosphates in perfused rat heart during metabolic acidosis.
    Jelicks LA, Gupta R.
    Am J Physiol; 1992 Sep 07; 263(3 Pt 2):H903-9. PubMed ID: 1415618
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