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Journal Abstract Search

112 related items for PubMed ID: 36757138

  • 1. Potentiation of neuromuscular transmission by a small molecule calcium channel gating modifier improves motor function in a severe spinal muscular atrophy mouse model.
    Ojala KS, Kaufhold CJ, Davey MR, Yang D, Liang M, Wipf P, Badawi Y, Meriney SD.
    Hum Mol Genet; 2023 May 18; 32(11):1901-1911. PubMed ID: 36757138
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  • 2. Chronic treatment with lithium does not improve neuromuscular phenotype in a mouse model of severe spinal muscular atrophy.
    Dachs E, Piedrafita L, Hereu M, Esquerda JE, Calderó J.
    Neuroscience; 2013 Oct 10; 250():417-33. PubMed ID: 23876328
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  • 5. Motor transmission defects with sex differences in a new mouse model of mild spinal muscular atrophy.
    Deguise MO, De Repentigny Y, Tierney A, Beauvais A, Michaud J, Chehade L, Thabet M, Paul B, Reilly A, Gagnon S, Renaud JM, Kothary R.
    EBioMedicine; 2020 May 10; 55():102750. PubMed ID: 32339936
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  • 6. Hyperexcitability precedes motoneuron loss in the Smn2B/- mouse model of spinal muscular atrophy.
    Quinlan KA, Reedich EJ, Arnold WD, Puritz AC, Cavarsan CF, Heckman CJ, DiDonato CJ.
    J Neurophysiol; 2019 Oct 01; 122(4):1297-1311. PubMed ID: 31365319
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  • 17. Synaptic withdrawal following nerve injury is influenced by postnatal maturity, muscle-specific properties, and the presence of underlying pathology in mice.
    Mole AJ, Bell S, Thomson AK, Dissanayake KN, Ribchester RR, Murray LM.
    J Anat; 2020 Aug 01; 237(2):263-274. PubMed ID: 32311115
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