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Journal Abstract Search

196 related items for PubMed ID: 36940705

  • 1.
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  • 2. DNA single-strand break repair is impaired in aprataxin-related ataxia.
    Hirano M, Yamamoto A, Mori T, Lan L, Iwamoto TA, Aoki M, Shimada K, Furiya Y, Kariya S, Asai H, Yasui A, Nishiwaki T, Imoto K, Kobayashi N, Kiriyama T, Nagata T, Konishi N, Itoyama Y, Ueno S.
    Ann Neurol; 2007 Feb; 61(2):162-74. PubMed ID: 17315206
    [Abstract] [Full Text] [Related]

  • 3. XRCC1 phosphorylation affects aprataxin recruitment and DNA deadenylation activity.
    Horton JK, Stefanick DF, Çağlayan M, Zhao ML, Janoshazi AK, Prasad R, Gassman NR, Wilson SH.
    DNA Repair (Amst); 2018 Apr; 64():26-33. PubMed ID: 29477978
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  • 4. Early-onset ataxia with ocular motor apraxia and hypoalbuminemia/ataxia with oculomotor apraxia 1.
    Tada M, Yokoseki A, Sato T, Makifuchi T, Onodera O.
    Adv Exp Med Biol; 2010 Apr; 685():21-33. PubMed ID: 20687492
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  • 5. The FHA domain of PNKP is essential for its recruitment to DNA damage sites and maintenance of genome stability.
    Tsukada K, Shimada M, Imamura R, Saikawa K, Ishiai M, Matsumoto Y.
    Mutat Res; 2021 Apr; 822():111727. PubMed ID: 33220551
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  • 7. The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates.
    Ahel I, Rass U, El-Khamisy SF, Katyal S, Clements PM, McKinnon PJ, Caldecott KW, West SC.
    Nature; 2006 Oct 12; 443(7112):713-6. PubMed ID: 16964241
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  • 8. Species variations in XRCC1 recruitment strategies for FHA domain-containing proteins.
    London RE.
    DNA Repair (Amst); 2022 Feb 12; 110():103263. PubMed ID: 35026705
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  • 9. Synergistic decrease of DNA single-strand break repair rates in mouse neural cells lacking both Tdp1 and aprataxin.
    El-Khamisy SF, Katyal S, Patel P, Ju L, McKinnon PJ, Caldecott KW.
    DNA Repair (Amst); 2009 Jun 04; 8(6):760-6. PubMed ID: 19303373
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  • 12. DNA repair mechanisms in dividing and non-dividing cells.
    Iyama T, Wilson DM.
    DNA Repair (Amst); 2013 Aug 04; 12(8):620-36. PubMed ID: 23684800
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  • 13. XRCC4 and XLF form long helical protein filaments suitable for DNA end protection and alignment to facilitate DNA double strand break repair.
    Mahaney BL, Hammel M, Meek K, Tainer JA, Lees-Miller SP.
    Biochem Cell Biol; 2013 Feb 04; 91(1):31-41. PubMed ID: 23442139
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  • 14. The FHA domain of aprataxin interacts with the C-terminal region of XRCC1.
    Date H, Igarashi S, Sano Y, Takahashi T, Takahashi T, Takano H, Tsuji S, Nishizawa M, Onodera O.
    Biochem Biophys Res Commun; 2004 Dec 24; 325(4):1279-85. PubMed ID: 15555565
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  • 16. Deficiency of XLF and PAXX prevents DNA double-strand break repair by non-homologous end joining in lymphocytes.
    Hung PJ, Chen BR, George R, Liberman C, Morales AJ, Colon-Ortiz P, Tyler JK, Sleckman BP, Bredemeyer AL.
    Cell Cycle; 2017 Feb 24; 16(3):286-295. PubMed ID: 27830975
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  • 18. Remodeling and spacing factor 1 (RSF1) deposits centromere proteins at DNA double-strand breaks to promote non-homologous end-joining.
    Helfricht A, Wiegant WW, Thijssen PE, Vertegaal AC, Luijsterburg MS, van Attikum H.
    Cell Cycle; 2013 Sep 15; 12(18):3070-82. PubMed ID: 23974106
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  • 19. C-Terminal region of DNA ligase IV drives XRCC4/DNA ligase IV complex to chromatin.
    Liu S, Liu X, Kamdar RP, Wanotayan R, Sharma MK, Adachi N, Matsumoto Y.
    Biochem Biophys Res Commun; 2013 Sep 20; 439(2):173-8. PubMed ID: 23994631
    [Abstract] [Full Text] [Related]

  • 20. Molecular underpinnings of Aprataxin RNA/DNA deadenylase function and dysfunction in neurological disease.
    Schellenberg MJ, Tumbale PP, Williams RS.
    Prog Biophys Mol Biol; 2015 Mar 20; 117(2-3):157-165. PubMed ID: 25637650
    [Abstract] [Full Text] [Related]

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