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Journal Abstract Search

203 related items for PubMed ID: 3789183

  • 1. Four brief periods of myocardial ischemia cause no cumulative ATP loss or necrosis.
    Reimer KA, Murry CE, Yamasawa I, Hill ML, Jennings RB.
    Am J Physiol; 1986 Dec; 251(6 Pt 2):H1306-15. PubMed ID: 3789183
    [Abstract] [Full Text] [Related]

  • 2. Prolonged derangements of canine myocardial purine metabolism after a brief coronary artery occlusion not associated with anatomic evidence of necrosis.
    DeBoer LW, Ingwall JS, Kloner RA, Braunwald E.
    Proc Natl Acad Sci U S A; 1980 Sep; 77(9):5471-5. PubMed ID: 6933566
    [Abstract] [Full Text] [Related]

  • 3. Intermittent v continuous ischemia decelerates adenylate breakdown and prevents norepinephrine release in reperfused rabbit heart.
    de Jong JW, Cargnoni A, Bradamante S, Curello S, Janssen M, Pasini E, Ceconi C, Bünger R, Ferrari R.
    J Mol Cell Cardiol; 1995 Jan; 27(1):659-71. PubMed ID: 7760385
    [Abstract] [Full Text] [Related]

  • 4. Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium.
    Murry CE, Jennings RB, Reimer KA.
    Circulation; 1986 Nov; 74(5):1124-36. PubMed ID: 3769170
    [Abstract] [Full Text] [Related]

  • 5. Ischemic preconditioning slows energy metabolism and delays ultrastructural damage during a sustained ischemic episode.
    Murry CE, Richard VJ, Reimer KA, Jennings RB.
    Circ Res; 1990 Apr; 66(4):913-31. PubMed ID: 2317895
    [Abstract] [Full Text] [Related]

  • 6. Pathobiology of acute myocardial ischemia: metabolic, functional and ultrastructural studies.
    Reimer KA, Jennings RB, Tatum AH.
    Am J Cardiol; 1983 Jul 20; 52(2):72A-81A. PubMed ID: 6869259
    [Abstract] [Full Text] [Related]

  • 7. Prolonged depletion of ATP because of delayed repletion of the adenine nucleotide pool following reversible myocardial ischemic injury in dogs.
    Reimer KA, Hill ML, Jennings RB.
    Adv Myocardiol; 1983 Jul 20; 4():395-407. PubMed ID: 6856966
    [Abstract] [Full Text] [Related]

  • 8. Energy metabolism in the reversible and irreversible phases of severe myocardial ischemia.
    Reimer KA, Jennings RB.
    Acta Med Scand Suppl; 1981 Jul 20; 651():19-27. PubMed ID: 6948501
    [Abstract] [Full Text] [Related]

  • 9. Salutary action of nicorandil, a new antianginal drug, on myocardial metabolism during ischemia and on postischemic function in a canine preparation of brief, repetitive coronary artery occlusions: comparison with isosorbide dinitrate.
    Pieper GM, Gross GJ.
    Circulation; 1987 Oct 20; 76(4):916-28. PubMed ID: 2958176
    [Abstract] [Full Text] [Related]

  • 10. Repetitive episodes of brief ischaemia (12 min) do not produce a cumulative depletion of high energy phosphate compounds.
    Swain JL, Sabina RL, Hines JJ, Greenfield JC, Holmes EW.
    Cardiovasc Res; 1984 May 20; 18(5):264-9. PubMed ID: 6733731
    [Abstract] [Full Text] [Related]

  • 11. Effects of recurrent ischemia on myocardial high energy phosphate content in canine hearts.
    Lange R, Ingwall JS, Hale SL, Alker KJ, Kloner RA.
    Basic Res Cardiol; 1984 May 20; 79(4):469-78. PubMed ID: 6487240
    [Abstract] [Full Text] [Related]

  • 12. Influence of repetitive coronary occlusions on myocardial adenine nucleosides, high energy phosphates and ultrastructure.
    Henrichs KJ, Matsuoka H, Schaper J.
    Basic Res Cardiol; 1987 May 20; 82(6):557-65. PubMed ID: 3435401
    [Abstract] [Full Text] [Related]

  • 13. Repeated short periods of regional myocardial ischemia: effect on local function and high energy phosphate levels.
    Hoffmeister HM, Mauser M, Schaper W.
    Basic Res Cardiol; 1986 May 20; 81(4):361-72. PubMed ID: 3778416
    [Abstract] [Full Text] [Related]

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  • 15. Nucleotide metabolism and cellular damage in myocardial ischemia.
    Jennings RB, Steenbergen C.
    Annu Rev Physiol; 1985 May 20; 47():727-49. PubMed ID: 2581508
    [Abstract] [Full Text] [Related]

  • 16. Effects of calcium-channel blockers on myocardial preservation during experimental acute myocardial infarction.
    Reimer KA, Jennings RB.
    Am J Cardiol; 1985 Jan 25; 55(3):107B-115B. PubMed ID: 3881903
    [Abstract] [Full Text] [Related]

  • 17. Intermittent aortic crossclamping prevents cumulative adenosine triphosphate depletion, ventricular fibrillation, and dysfunction (stunning): is it preconditioning?
    Abd-Elfattah AS, Ding M, Wechsler AS.
    J Thorac Cardiovasc Surg; 1995 Aug 25; 110(2):328-39. PubMed ID: 7637350
    [Abstract] [Full Text] [Related]

  • 18. Is adenosine 5'-triphosphate derangement or free-radical-mediated injury the major cause of ventricular dysfunction during reperfusion? Role of adenine nucleoside transport in myocardial reperfusion injury.
    Abd-Elfattah AS, Jessen ME, Hanan SA, Tuchy G, Wechsler AS.
    Circulation; 1990 Nov 25; 82(5 Suppl):IV341-50. PubMed ID: 2225426
    [Abstract] [Full Text] [Related]

  • 19. Myocardial metabolism and regional myocardial blood flow in the canine left ventricle following twenty minutes of circumflex artery occlusion and reperfusion.
    Allison TB, Holsinger JW.
    J Mol Cell Cardiol; 1983 Mar 25; 15(3):151-61. PubMed ID: 6864811
    [Abstract] [Full Text] [Related]

  • 20. [-Myocardial protection by preconditioning. Experimental and clinical significance-].
    Strasser R, Vogt A, Schaper W.
    Z Kardiol; 1996 Feb 25; 85(2):79-89. PubMed ID: 8650986
    [Abstract] [Full Text] [Related]

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