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137 related items for PubMed ID: 37961889
1. Nitric Oxide Modulates Ca2+ Leak and Arrhythmias via S-Nitrosylation of CaMKII. Power AS, Asamudo EU, Worthington LPI, Alim CC, Parackal RE, Wallace RS, Ebenebe OV, Heller Brown J, Kohr MJ, Bers DM, Erickson JR. Circ Res; 2023 Dec 08; 133(12):1040-1055. PubMed ID: 37961889 [Abstract] [Full Text] [Related]
2. Nitric Oxide modulates spontaneous Ca2+ release and ventricular arrhythmias during β-adrenergic signalling through S-nitrosylation of Calcium/Calmodulin dependent kinase II. Power AS, Asamudo E, Worthington LPI, Alim CC, Parackal R, Wallace RS, Ebenebe OV, Brown JH, Kohr MJ, Bers DM, Erickson JR. bioRxiv; 2023 Aug 24. PubMed ID: 37662205 [Abstract] [Full Text] [Related]
3. Nitrosylation of cardiac CaMKII at Cys290 mediates mechanical afterload-induced increases in Ca2+ transient and Ca2+ sparks. Alim CC, Ko CY, Mira Hernandez J, Shen EY, Baidar S, Chen-Izu Y, Bers DM, Bossuyt J. J Physiol; 2022 Nov 24; 600(22):4865-4879. PubMed ID: 36227145 [Abstract] [Full Text] [Related]
4. CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation. Grimm M, Ling H, Willeford A, Pereira L, Gray CB, Erickson JR, Sarma S, Respress JL, Wehrens XH, Bers DM, Brown JH. J Mol Cell Cardiol; 2015 Aug 24; 85():282-91. PubMed ID: 26080362 [Abstract] [Full Text] [Related]
5. Nitric oxide-dependent activation of CaMKII increases diastolic sarcoplasmic reticulum calcium release in cardiac myocytes in response to adrenergic stimulation. Curran J, Tang L, Roof SR, Velmurugan S, Millard A, Shonts S, Wang H, Santiago D, Ahmad U, Perryman M, Bers DM, Mohler PJ, Ziolo MT, Shannon TR. PLoS One; 2014 Aug 24; 9(2):e87495. PubMed ID: 24498331 [Abstract] [Full Text] [Related]
6. Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure. Sag CM, Wadsack DP, Khabbazzadeh S, Abesser M, Grefe C, Neumann K, Opiela MK, Backs J, Olson EN, Brown JH, Neef S, Maier SK, Maier LS. Circ Heart Fail; 2009 Nov 24; 2(6):664-75. PubMed ID: 19919992 [Abstract] [Full Text] [Related]
7. Autonomous activation of CaMKII exacerbates diastolic calcium leak during beta-adrenergic stimulation in cardiomyocytes of metabolic syndrome rats. Romero-García T, Landa-Galvan HV, Pavón N, Mercado-Morales M, Valdivia HH, Rueda A. Cell Calcium; 2020 Nov 24; 91():102267. PubMed ID: 32920522 [Abstract] [Full Text] [Related]
8. CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factor. Haugsten Hansen M, Sadredini M, Hasic A, Anderson ME, Sjaastad I, Korseberg Stokke M. J Mol Cell Cardiol; 2023 Feb 24; 175():49-61. PubMed ID: 36528076 [Abstract] [Full Text] [Related]
9. Calcium/calmodulin-dependent kinase II and nitric oxide synthase 1-dependent modulation of ryanodine receptors during β-adrenergic stimulation is restricted to the dyadic cleft. Dries E, Santiago DJ, Johnson DM, Gilbert G, Holemans P, Korte SM, Roderick HL, Sipido KR. J Physiol; 2016 Oct 15; 594(20):5923-5939. PubMed ID: 27121757 [Abstract] [Full Text] [Related]
10. Oxidation of ryanodine receptor after ischemia-reperfusion increases propensity of Ca2+ waves during β-adrenergic receptor stimulation. Bovo E, Mazurek SR, Zima AV. Am J Physiol Heart Circ Physiol; 2018 Oct 01; 315(4):H1032-H1040. PubMed ID: 30028204 [Abstract] [Full Text] [Related]
11. Epac2 mediates cardiac β1-adrenergic-dependent sarcoplasmic reticulum Ca2+ leak and arrhythmia. Pereira L, Cheng H, Lao DH, Na L, van Oort RJ, Brown JH, Wehrens XH, Chen J, Bers DM. Circulation; 2013 Feb 26; 127(8):913-22. PubMed ID: 23363625 [Abstract] [Full Text] [Related]
12. CaMKIIδ Met281/282 oxidation is not required for recovery of calcium transients during acidosis. Kreitmeier KG, Tarnowski D, Nanadikar MS, Baier MJ, Wagner S, Katschinski DM, Maier LS, Sag CM. Am J Physiol Heart Circ Physiol; 2021 Mar 01; 320(3):H1199-H1212. PubMed ID: 33449853 [Abstract] [Full Text] [Related]
13. Overexpression of adenylyl cyclase type 5 (AC5) confers a proarrhythmic substrate to the heart. Zhao Z, Babu GJ, Wen H, Fefelova N, Gordan R, Sui X, Yan L, Vatner DE, Vatner SF, Xie LH. Am J Physiol Heart Circ Physiol; 2015 Feb 01; 308(3):H240-9. PubMed ID: 25485900 [Abstract] [Full Text] [Related]
15. Reactive oxygen species-activated Ca/calmodulin kinase IIδ is required for late I(Na) augmentation leading to cellular Na and Ca overload. Wagner S, Ruff HM, Weber SL, Bellmann S, Sowa T, Schulte T, Anderson ME, Grandi E, Bers DM, Backs J, Belardinelli L, Maier LS. Circ Res; 2011 Mar 04; 108(5):555-65. PubMed ID: 21252154 [Abstract] [Full Text] [Related]
17. S-Nitrosylation of Calcium-Handling Proteins in Cardiac Adrenergic Signaling and Hypertrophy. Irie T, Sips PY, Kai S, Kida K, Ikeda K, Hirai S, Moazzami K, Jiramongkolchai P, Bloch DB, Doulias PT, Armoundas AA, Kaneki M, Ischiropoulos H, Kranias E, Bloch KD, Stamler JS, Ichinose F. Circ Res; 2015 Oct 09; 117(9):793-803. PubMed ID: 26259881 [Abstract] [Full Text] [Related]
18. S-Nitrosylation Induces Both Autonomous Activation and Inhibition of Calcium/Calmodulin-dependent Protein Kinase II δ. Erickson JR, Nichols CB, Uchinoumi H, Stein ML, Bossuyt J, Bers DM. J Biol Chem; 2015 Oct 16; 290(42):25646-56. PubMed ID: 26316536 [Abstract] [Full Text] [Related]
19. Phospholamban ablation rescues the enhanced propensity to arrhythmias of mice with CaMKII-constitutive phosphorylation of RyR2 at site S2814. Mazzocchi G, Sommese L, Palomeque J, Felice JI, Di Carlo MN, Fainstein D, Gonzalez P, Contreras P, Skapura D, McCauley MD, Lascano EC, Negroni JA, Kranias EG, Wehrens XH, Valverde CA, Mattiazzi A. J Physiol; 2016 Jun 01; 594(11):3005-30. PubMed ID: 26695843 [Abstract] [Full Text] [Related]
20. β-Adrenergic induced SR Ca2+ leak is mediated by an Epac-NOS pathway. Pereira L, Bare DJ, Galice S, Shannon TR, Bers DM. J Mol Cell Cardiol; 2017 Jul 01; 108():8-16. PubMed ID: 28476660 [Abstract] [Full Text] [Related] Page: [Next] [New Search]