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Journal Abstract Search

366 related items for PubMed ID: 38411501

  • 1. SNAP25 disease mutations change the energy landscape for synaptic exocytosis due to aberrant SNARE interactions.
    Kádková A, Murach J, Østergaard M, Malsam A, Malsam J, Lolicato F, Nickel W, Söllner TH, Sørensen JB.
    Elife; 2024 Feb 27; 12():. PubMed ID: 38411501
    [Abstract] [Full Text] [Related]

  • 2. Ca2+-dependent release of synaptotagmin-1 from the SNARE complex on phosphatidylinositol 4,5-bisphosphate-containing membranes.
    Voleti R, Jaczynska K, Rizo J.
    Elife; 2020 Aug 18; 9():. PubMed ID: 32808925
    [Abstract] [Full Text] [Related]

  • 3. Neurotransmitter release is triggered by a calcium-induced rearrangement in the Synaptotagmin-1/SNARE complex primary interface.
    Toulmé E, Salazar Lázaro A, Trimbuch T, Rizo J, Rosenmund C.
    Proc Natl Acad Sci U S A; 2024 Oct 15; 121(42):e2409636121. PubMed ID: 39374398
    [Abstract] [Full Text] [Related]

  • 4. Interactions Between SNAP-25 and Synaptotagmin-1 Are Involved in Vesicle Priming, Clamping Spontaneous and Stimulating Evoked Neurotransmission.
    Schupp M, Malsam J, Ruiter M, Scheutzow A, Wierda KD, Söllner TH, Sørensen JB.
    J Neurosci; 2016 Nov 23; 36(47):11865-11880. PubMed ID: 27881774
    [Abstract] [Full Text] [Related]

  • 5. Allosteric stabilization of calcium and phosphoinositide dual binding engages several synaptotagmins in fast exocytosis.
    Kobbersmed JRL, Berns MMM, Ditlevsen S, Sørensen JB, Walter AM.
    Elife; 2022 Aug 05; 11():. PubMed ID: 35929728
    [Abstract] [Full Text] [Related]

  • 6. A Post-Docking Role of Synaptotagmin 1-C2B Domain Bottom Residues R398/399 in Mouse Chromaffin Cells.
    Kedar GH, Munch AS, van Weering JR, Malsam J, Scheutzow A, de Wit H, Houy S, Tawfik B, Söllner TH, Sørensen JB, Verhage M.
    J Neurosci; 2015 Oct 21; 35(42):14172-82. PubMed ID: 26490858
    [Abstract] [Full Text] [Related]

  • 7. Polybasic Patches in Both C2 Domains of Synaptotagmin-1 Are Required for Evoked Neurotransmitter Release.
    Wu Z, Ma L, Courtney NA, Zhu J, Landajuela A, Zhang Y, Chapman ER, Karatekin E.
    J Neurosci; 2022 Jul 27; 42(30):5816-5829. PubMed ID: 35701163
    [Abstract] [Full Text] [Related]

  • 8.
    Liedtke WB, Heller S, Planells-Cases R, Ferrer-Montiel A.
    ; 2007 Jul 27. PubMed ID: 21204515
    [Abstract] [Full Text] [Related]

  • 9. Exploring the structural dynamics of the vesicle priming machinery.
    An D, Lindau M.
    Biochem Soc Trans; 2024 Aug 28; 52(4):1715-1725. PubMed ID: 39082978
    [Abstract] [Full Text] [Related]

  • 10. Models of synaptotagmin-1 to trigger Ca2+ -dependent vesicle fusion.
    Park Y, Ryu JK.
    FEBS Lett; 2018 Nov 28; 592(21):3480-3492. PubMed ID: 30004579
    [Abstract] [Full Text] [Related]

  • 11. Synaptotagmin-1 and -7 Are Redundantly Essential for Maintaining the Capacity of the Readily-Releasable Pool of Synaptic Vesicles.
    Bacaj T, Wu D, Burré J, Malenka RC, Liu X, Südhof TC.
    PLoS Biol; 2015 Oct 28; 13(10):e1002267. PubMed ID: 26437117
    [Abstract] [Full Text] [Related]

  • 12. Molecular mechanisms of COMPLEXIN fusion clamp function in synaptic exocytosis revealed in a new Drosophila mutant.
    Iyer J, Wahlmark CJ, Kuser-Ahnert GA, Kawasaki F.
    Mol Cell Neurosci; 2013 Sep 28; 56():244-54. PubMed ID: 23769723
    [Abstract] [Full Text] [Related]

  • 13. The primed SNARE-complexin-synaptotagmin complex for neuronal exocytosis.
    Zhou Q, Zhou P, Wang AL, Wu D, Zhao M, Südhof TC, Brunger AT.
    Nature; 2017 Aug 24; 548(7668):420-425. PubMed ID: 28813412
    [Abstract] [Full Text] [Related]

  • 14. synaptotagmin mutants reveal essential functions for the C2B domain in Ca2+-triggered fusion and recycling of synaptic vesicles in vivo.
    Littleton JT, Bai J, Vyas B, Desai R, Baltus AE, Garment MB, Carlson SD, Ganetzky B, Chapman ER.
    J Neurosci; 2001 Mar 01; 21(5):1421-33. PubMed ID: 11222632
    [Abstract] [Full Text] [Related]

  • 15. Synaptic vesicle pool-specific modification of neurotransmitter release by intravesicular free radical generation.
    Afuwape OA, Wasser CR, Schikorski T, Kavalali ET.
    J Physiol; 2017 Feb 15; 595(4):1223-1238. PubMed ID: 27723113
    [Abstract] [Full Text] [Related]

  • 16. Complexin controls spontaneous and evoked neurotransmitter release by regulating the timing and properties of synaptotagmin activity.
    Jorquera RA, Huntwork-Rodriguez S, Akbergenova Y, Cho RW, Littleton JT.
    J Neurosci; 2012 Dec 12; 32(50):18234-45. PubMed ID: 23238737
    [Abstract] [Full Text] [Related]

  • 17. The Transmembrane Domain of Synaptobrevin Influences Neurotransmitter Flux through Synaptic Fusion Pores.
    Chiang CW, Chang CW, Jackson MB.
    J Neurosci; 2018 Aug 08; 38(32):7179-7191. PubMed ID: 30012692
    [Abstract] [Full Text] [Related]

  • 18. Deconstructing Synaptotagmin-1's Distinct Roles in Synaptic Vesicle Priming and Neurotransmitter Release.
    Bouazza-Arostegui B, Camacho M, Brockmann MM, Zobel S, Rosenmund C.
    J Neurosci; 2022 Apr 06; 42(14):2856-2871. PubMed ID: 35193927
    [Abstract] [Full Text] [Related]

  • 19. Neurotransmitter release is triggered by a calcium-induced rearrangement in the Synaptotagmin-1/SNARE complex primary interface.
    Toulmé E, Lázaro AS, Trimbuch T, Rizo J, Rosenmund C.
    bioRxiv; 2024 Jun 21. PubMed ID: 38948868
    [Abstract] [Full Text] [Related]

  • 20. The stability of the primed pool of synaptic vesicles and the clamping of spontaneous neurotransmitter release rely on the integrity of the C-terminal half of the SNARE domain of syntaxin-1A.
    Salazar Lázaro A, Trimbuch T, Vardar G, Rosenmund C.
    Elife; 2024 Mar 21; 12():. PubMed ID: 38512129
    [Abstract] [Full Text] [Related]

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