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PUBMED FOR HANDHELDS

Journal Abstract Search


248 related items for PubMed ID: 38575959

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  • 2. Prominent tauopathy and intracellular β-amyloid accumulation triggered by genetic deletion of cathepsin D: Implications for Alzheimer disease pathogenesis.
    Terron HM, Parikh SJ, Abdul-Hay SO, Sahara T, Kang D, Dickson DW, Saftig P, LaFerla FM, Lane S, Leissring MA.
    Res Sq; 2023 Oct 23. PubMed ID: 37961253
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  • 7. Chemogenetic attenuation of neuronal activity in the entorhinal cortex reduces Aβ and tau pathology in the hippocampus.
    Rodriguez GA, Barrett GM, Duff KE, Hussaini SA.
    PLoS Biol; 2020 Aug 23; 18(8):e3000851. PubMed ID: 32822389
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  • 9. Cathepsin B degrades amyloid-β in mice expressing wild-type human amyloid precursor protein.
    Wang C, Sun B, Zhou Y, Grubb A, Gan L.
    J Biol Chem; 2012 Nov 16; 287(47):39834-41. PubMed ID: 23024364
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  • 11. Soluble Conformers of Aβ and Tau Alter Selective Proteins Governing Axonal Transport.
    Sherman MA, LaCroix M, Amar F, Larson ME, Forster C, Aguzzi A, Bennett DA, Ramsden M, Lesné SE.
    J Neurosci; 2016 Sep 14; 36(37):9647-58. PubMed ID: 27629715
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  • 12. Increased tauopathy drives microglia-mediated clearance of beta-amyloid.
    Chen W, Abud EA, Yeung ST, Lakatos A, Nassi T, Wang J, Blum D, Buée L, Poon WW, Blurton-Jones M.
    Acta Neuropathol Commun; 2016 Jun 23; 4(1):63. PubMed ID: 27339073
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  • 13. The fluorescent ligand bTVBT2 reveals increased p-tau uptake by retinal microglia in Alzheimer's disease patients and AppNL-F/NL-F mice.
    Nuñez-Diaz C, Andersson E, Schultz N, Pocevičiūtė D, Hansson O, Netherlands Brain Bank, Nilsson KPR, Wennström M.
    Alzheimers Res Ther; 2024 Jan 02; 16(1):4. PubMed ID: 38167557
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  • 15. Tau Protein Mediates APP Intracellular Domain (AICD)-Induced Alzheimer's-Like Pathological Features in Mice.
    Ghosal K, Fan Q, Dawson HN, Pimplikar SW.
    PLoS One; 2016 Jan 02; 11(7):e0159435. PubMed ID: 27459671
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  • 17. Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalities.
    Tran HT, LaFerla FM, Holtzman DM, Brody DL.
    J Neurosci; 2011 Jun 29; 31(26):9513-25. PubMed ID: 21715616
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  • 18. Dietary supplementation with S-adenosyl methionine delayed amyloid-β and tau pathology in 3xTg-AD mice.
    Lee S, Lemere CA, Frost JL, Shea TB.
    J Alzheimers Dis; 2012 Jun 29; 28(2):423-31. PubMed ID: 22045486
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  • 20. CCL2 Overexpression in the Brain Promotes Glial Activation and Accelerates Tau Pathology in a Mouse Model of Tauopathy.
    Joly-Amado A, Hunter J, Quadri Z, Zamudio F, Rocha-Rangel PV, Chan D, Kesarwani A, Nash K, Lee DC, Morgan D, Gordon MN, Selenica MB.
    Front Immunol; 2020 Jun 29; 11():997. PubMed ID: 32508844
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