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8. α2δ-1 couples to NMDA receptors in the hypothalamus to sustain sympathetic vasomotor activity in hypertension. Ma H, Chen SR, Chen H, Zhou JJ, Li DP, Pan HL. J Physiol; 2018 Sep 05; 596(17):4269-4283. PubMed ID: 29971791 [Abstract] [Full Text] [Related]
9. Nerve injury increases GluA2-lacking AMPA receptor prevalence in spinal cords: functional significance and signaling mechanisms. Chen SR, Zhou HY, Byun HS, Pan HL. J Pharmacol Exp Ther; 2013 Dec 05; 347(3):765-72. PubMed ID: 24030012 [Abstract] [Full Text] [Related]
10. CaMKII Regulates Synaptic NMDA Receptor Activity of Hypothalamic Presympathetic Neurons and Sympathetic Outflow in Hypertension. Li DP, Zhou JJ, Zhang J, Pan HL. J Neurosci; 2017 Nov 01; 37(44):10690-10699. PubMed ID: 28972129 [Abstract] [Full Text] [Related]
11. α2δ-1 Is Essential for Sympathetic Output and NMDA Receptor Activity Potentiated by Angiotensin II in the Hypothalamus. Ma H, Chen SR, Chen H, Li L, Li DP, Zhou JJ, Pan HL. J Neurosci; 2018 Jul 11; 38(28):6388-6398. PubMed ID: 29921713 [Abstract] [Full Text] [Related]
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17. Calcineurin Inhibition Causes α2δ-1-Mediated Tonic Activation of Synaptic NMDA Receptors and Pain Hypersensitivity. Huang Y, Chen SR, Chen H, Luo Y, Pan HL. J Neurosci; 2020 May 06; 40(19):3707-3719. PubMed ID: 32269108 [Abstract] [Full Text] [Related]
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