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Journal Abstract Search

186 related items for PubMed ID: 38880127

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  • 3. Carboxyl-terminal sequences in APOA5 are important for suppressing ANGPTL3/8 activity.
    Chen YQ, Yang Y, Zhen EY, Beyer TP, Li H, Wen Y, Ehsani M, Jackson N, Xie K, Jung H, Scheithauer JL, Kumari A, Birrane G, Russell AM, Balasubramaniam D, Liao Z, Siegel RW, Qian Y, Ploug M, Young SG, Konrad RJ.
    Proc Natl Acad Sci U S A; 2024 Apr 23; 121(17):e2322332121. PubMed ID: 38625948
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  • 4. ApoA5 lowers triglyceride levels via suppression of ANGPTL3/8-mediated LPL inhibition.
    Chen YQ, Pottanat TG, Zhen EY, Siegel RW, Ehsani M, Qian YW, Konrad RJ.
    J Lipid Res; 2021 Apr 23; 62():100068. PubMed ID: 33762177
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  • 6. Rare and common variants in LPL and APOA5 in Thai subjects with severe hypertriglyceridemia: A resequencing approach.
    Khovidhunkit W, Charoen S, Kiateprungvej A, Chartyingcharoen P, Muanpetch S, Plengpanich W.
    J Clin Lipidol; 2016 Apr 23; 10(3):505-511.e1. PubMed ID: 27206937
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  • 9. Magnolol-mediated regulation of plasma triglyceride through affecting lipoprotein lipase activity in apolipoprotein A5 knock-in mice.
    Chang CK, Lin XR, Lin YL, Fang WH, Lin SW, Chang SY, Kao JT.
    PLoS One; 2018 Apr 23; 13(2):e0192740. PubMed ID: 29425239
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  • 12. Identification of a new functional domain in angiopoietin-like 3 (ANGPTL3) and angiopoietin-like 4 (ANGPTL4) involved in binding and inhibition of lipoprotein lipase (LPL).
    Lee EC, Desai U, Gololobov G, Hong S, Feng X, Yu XC, Gay J, Wilganowski N, Gao C, Du LL, Chen J, Hu Y, Zhao S, Kirkpatrick L, Schneider M, Zambrowicz BP, Landes G, Powell DR, Sonnenburg WK.
    J Biol Chem; 2009 May 15; 284(20):13735-13745. PubMed ID: 19318355
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  • 13. Severe hypertriglyceridaemia and pancreatitis in a patient with lipoprotein lipase deficiency based on mutations in lipoprotein lipase (LPL) and apolipoprotein A5 (APOA5) genes.
    Koopal C, Bemelmans R, Marais AD, Visseren FL.
    BMJ Case Rep; 2019 Apr 03; 12(4):. PubMed ID: 30948399
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  • 14. ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis.
    Schaap FG, Rensen PC, Voshol PJ, Vrins C, van der Vliet HN, Chamuleau RA, Havekes LM, Groen AK, van Dijk KW.
    J Biol Chem; 2004 Jul 02; 279(27):27941-7. PubMed ID: 15090553
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  • 17. Incidental finding of severe hypertriglyceridemia in children. Role of multiple rare variants in genes affecting plasma triglyceride.
    Buonuomo PS, Rabacchi C, Macchiaiolo M, Trenti C, Fasano T, Tarugi P, Bartuli A, Bertolini S, Calandra S.
    J Clin Lipidol; 2017 Jul 02; 11(6):1329-1337.e3. PubMed ID: 28951076
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  • 19. Familial hypertriglyceridemia: an entity with distinguishable features from other causes of hypertriglyceridemia.
    Cruz-Bautista I, Huerta-Chagoya A, Moreno-Macías H, Rodríguez-Guillén R, Ordóñez-Sánchez ML, Segura-Kato Y, Mehta R, Almeda-Valdés P, Gómez-Munguía L, Ruiz-De Chávez X, Rosas-Flota X, Andrade-Amado A, Bernal-Barroeta B, López-Carrasco MG, Guillén-Pineda LE, López-Estrada A, Elías-López D, Martagón-Rosado AJ, Gómez-Velasco D, Lam-Chung CE, Bello-Chavolla OY, Del Razo-Olvera F, Cetina-Pérez LD, Acosta-Rodríguez JL, Tusié-Luna MT, Aguilar-Salinas CA.
    Lipids Health Dis; 2021 Feb 15; 20(1):14. PubMed ID: 33588820
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