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Journal Abstract Search

208 related items for PubMed ID: 39424798

  • 1. Post-transcriptional methylation of mitochondrial-tRNA differentially contributes to mitochondrial pathology.
    Maharjan S, Gamper H, Yamaki Y, Christian T, Henley RY, Li NS, Suzuki T, Suzuki T, Piccirilli JA, Wanunu M, Seifert E, Wallace DC, Hou YM.
    Nat Commun; 2024 Oct 18; 15(1):9008. PubMed ID: 39424798
    [Abstract] [Full Text] [Related]

  • 2. Post-Transcriptional Methylation of Mitochondrial-tRNA Differentially Contributes to Mitochondrial Pathology.
    Maharjan S, Gamper H, Yamaki Y, Henley RY, Li NS, Suzuki T, Suzuki T, Piccirilli JA, Wanunu M, Seifert E, Wallace DC, Hou YM.
    bioRxiv; 2023 Dec 10. PubMed ID: 38106193
    [Abstract] [Full Text] [Related]

  • 3. Acquisition of the wobble modification in mitochondrial tRNALeu(CUN) bearing the G12300A mutation suppresses the MELAS molecular defect.
    Kirino Y, Yasukawa T, Marjavaara SK, Jacobs HT, Holt IJ, Watanabe K, Suzuki T.
    Hum Mol Genet; 2006 Mar 15; 15(6):897-904. PubMed ID: 16446307
    [Abstract] [Full Text] [Related]

  • 4. Codon-specific translational defect caused by a wobble modification deficiency in mutant tRNA from a human mitochondrial disease.
    Kirino Y, Yasukawa T, Ohta S, Akira S, Ishihara K, Watanabe K, Suzuki T.
    Proc Natl Acad Sci U S A; 2004 Oct 19; 101(42):15070-5. PubMed ID: 15477592
    [Abstract] [Full Text] [Related]

  • 5. Disease-associated mutations in mitochondrial precursor tRNAs affect binding, m1R9 methylation, and tRNA processing by mtRNase P.
    Karasik A, Wilhelm CA, Fierke CA, Koutmos M.
    RNA; 2021 Apr 19; 27(4):420-432. PubMed ID: 33380464
    [Abstract] [Full Text] [Related]

  • 6. Human mitochondrial diseases associated with tRNA wobble modification deficiency.
    Kirino Y, Suzuki T.
    RNA Biol; 2005 Apr 19; 2(2):41-4. PubMed ID: 17132941
    [Abstract] [Full Text] [Related]

  • 7. Unraveling RNA Conformation Dynamics in Mitochondrial Encephalomyopathy, Lactic Acidosis, and Stroke-like Episode Syndrome with Solid-State Nanopores.
    Namani S, Kavetsky K, Lin CY, Maharjan S, Gamper HB, Li NS, Piccirilli JA, Hou YM, Drndic M.
    ACS Nano; 2024 Jul 02; 18(26):17240-17250. PubMed ID: 38906834
    [Abstract] [Full Text] [Related]

  • 8. The MELAS mutation m.3243A>G alters the expression of mitochondrial tRNA fragments.
    Meseguer S, Navarro-González C, Panadero J, Villarroya M, Boutoual R, Sánchez-Alcázar JA, Armengod ME.
    Biochim Biophys Acta Mol Cell Res; 2019 Sep 02; 1866(9):1433-1449. PubMed ID: 31195049
    [Abstract] [Full Text] [Related]

  • 9. Human mitochondrial diseases caused by lack of taurine modification in mitochondrial tRNAs.
    Suzuki T, Nagao A, Suzuki T.
    Wiley Interdiscip Rev RNA; 2011 Sep 02; 2(3):376-86. PubMed ID: 21957023
    [Abstract] [Full Text] [Related]

  • 10. Correction of the consequences of mitochondrial 3243A>G mutation in the MT-TL1 gene causing the MELAS syndrome by tRNA import into mitochondria.
    Karicheva OZ, Kolesnikova OA, Schirtz T, Vysokikh MY, Mager-Heckel AM, Lombès A, Boucheham A, Krasheninnikov IA, Martin RP, Entelis N, Tarassov I.
    Nucleic Acids Res; 2011 Oct 02; 39(18):8173-86. PubMed ID: 21724600
    [Abstract] [Full Text] [Related]

  • 11. The ROS-sensitive microRNA-9/9* controls the expression of mitochondrial tRNA-modifying enzymes and is involved in the molecular mechanism of MELAS syndrome.
    Meseguer S, Martínez-Zamora A, García-Arumí E, Andreu AL, Armengod ME.
    Hum Mol Genet; 2015 Jan 01; 24(1):167-84. PubMed ID: 25149473
    [Abstract] [Full Text] [Related]

  • 12. Pathogenic mitochondrial tRNA point mutations: nine novel mutations affirm their importance as a cause of mitochondrial disease.
    Blakely EL, Yarham JW, Alston CL, Craig K, Poulton J, Brierley C, Park SM, Dean A, Xuereb JH, Anderson KN, Compston A, Allen C, Sharif S, Enevoldson P, Wilson M, Hammans SR, Turnbull DM, McFarland R, Taylor RW.
    Hum Mutat; 2013 Sep 01; 34(9):1260-8. PubMed ID: 23696415
    [Abstract] [Full Text] [Related]

  • 13. Restoration of mitochondrial function through activation of hypomodified tRNAs with pathogenic mutations associated with mitochondrial diseases.
    Tomoda E, Nagao A, Shirai Y, Asano K, Suzuki T, Battersby BJ, Suzuki T.
    Nucleic Acids Res; 2023 Aug 11; 51(14):7563-7579. PubMed ID: 36928678
    [Abstract] [Full Text] [Related]

  • 14. Modification defect at anticodon wobble nucleotide of mitochondrial tRNAs(Leu)(UUR) with pathogenic mutations of mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes.
    Yasukawa T, Suzuki T, Ueda T, Ohta S, Watanabe K.
    J Biol Chem; 2000 Feb 11; 275(6):4251-7. PubMed ID: 10660592
    [Abstract] [Full Text] [Related]

  • 15. Mammalian NSUN2 introduces 5-methylcytidines into mitochondrial tRNAs.
    Shinoda S, Kitagawa S, Nakagawa S, Wei FY, Tomizawa K, Araki K, Araki M, Suzuki T, Suzuki T.
    Nucleic Acids Res; 2019 Sep 19; 47(16):8734-8745. PubMed ID: 31287866
    [Abstract] [Full Text] [Related]

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  • 18. Human mitochondrial leucyl-tRNA synthetase corrects mitochondrial dysfunctions due to the tRNALeu(UUR) A3243G mutation, associated with mitochondrial encephalomyopathy, lactic acidosis, and stroke-like symptoms and diabetes.
    Li R, Guan MX.
    Mol Cell Biol; 2010 May 19; 30(9):2147-54. PubMed ID: 20194621
    [Abstract] [Full Text] [Related]

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  • 20. Human mitochondrial tRNAs: biogenesis, function, structural aspects, and diseases.
    Suzuki T, Nagao A, Suzuki T.
    Annu Rev Genet; 2011 May 19; 45():299-329. PubMed ID: 21910628
    [Abstract] [Full Text] [Related]

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