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Journal Abstract Search
114 related items for PubMed ID: 4297475
1. Adrenal hyperplasia--a case report of delayed onset of the congenital form or an acquired form. Mahesh VB, Greenblatt RB, Coniff RF. J Clin Endocrinol Metab; 1968 May; 28(5):619-23. PubMed ID: 4297475 [No Abstract] [Full Text] [Related]
2. Secretion rates of cortisol and aldosterone precursors in various forms of congenital adrenal hyperplasia. New MI, Seaman MP. J Clin Endocrinol Metab; 1970 Mar; 30(3):361-71. PubMed ID: 4312795 [No Abstract] [Full Text] [Related]
3. Congenital adrenal hyperplasia due to deficiency of 11 -hydroxylation of 17 -hydroxylated steroids. Zachmann M, Völlmin JA, New MI, Curtius HC, Prader A. J Clin Endocrinol Metab; 1971 Sep; 33(3):501-8. PubMed ID: 4328339 [No Abstract] [Full Text] [Related]
4. Congenital adrenal hyperplasia due to a deficiency of one of the enzymes involved in the biosynthesis of pregnenolone. Camacho AM, Kowarski A, Migeon CJ, Brough AJ. J Clin Endocrinol Metab; 1968 Feb; 28(2):153-61. PubMed ID: 4295130 [No Abstract] [Full Text] [Related]
5. Adrenal virilism due to 21-hydroxylase deficiency in the postmenarchial female. Riddick DH, Hammond CB. Obstet Gynecol; 1975 Jan; 45(1):21-4. PubMed ID: 163016 [Abstract] [Full Text] [Related]
6. [Adrenal hirsutism (3beta-hydroxysteroid dehydrogenase deficiency). Studies using a chromatographic separation of the 17-ketosteroid fraction in the urine. 1. Dehydroepiandrosterone-forming adrenocortical adenoma]. Göbel P. Endokrinologie; 1967 Jan; 52(1):22-36. PubMed ID: 4296633 [No Abstract] [Full Text] [Related]
15. [Clinical variants of virile form of congenital adrenocortical dysfunction]. Dzenis IG, Latypova NKh, Kuznetsova MN. Akush Ginekol (Mosk); 1990 Apr; (4):25-8. PubMed ID: 2165743 [No Abstract] [Full Text] [Related]
16. [21-hydroxylase deficiency detected late in a man with chronic asthenia]. Guenel J, Bureau L, Orieux J. Ann Endocrinol (Paris); 1967 Apr; 28(1):71-7. PubMed ID: 4293957 [No Abstract] [Full Text] [Related]
18. [Congenital adrenal hyperplasia due to lack of 21-hydroxylation. Study of 3 cases, 2 of them familial, at the adult stage]. Vague J, Boyer J, Nicolino J, Latrabe J, Picard D, Luciani J, Fouilloux C, Lieutaud B, Cavaleri F. Ann Endocrinol (Paris); 1967 Dec; 28(2):149-76. PubMed ID: 6069154 [No Abstract] [Full Text] [Related]