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Journal Abstract Search

138 related items for PubMed ID: 6318905

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  • 2. Tetanic stimulation increases the frequency of miniature end-plate potentials at the frog neuromuscular junction in Mn2+-, CO2+-, and Ni2+-saline solutions.
    Kita H, Narita K, Van der Kloot W.
    Brain Res; 1981 Jan 26; 205(1):111-21. PubMed ID: 6258705
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  • 3. [Tetanic potentiation of miniature end-plate potential frequency at frog neuromuscular junction in manganese solutions].
    Narita K.
    Nihon Seirigaku Zasshi; 1985 Jan 26; 47(12):746-55. PubMed ID: 3007749
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  • 4. The thiol-oxidizing agent diamide increases transmitter release by decreasing calcium requirements for neuromuscular transmission in the frog.
    Carlen PL, Kosower EM, Werman R.
    Brain Res; 1976 Nov 26; 117(2):257-76. PubMed ID: 186154
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  • 8. Time course and magnitude of effects of changes in tonicity on acetylcholine release at frog neuromuscular junction.
    Kita H, van der Kloot W.
    J Neurophysiol; 1977 Mar 26; 40(2):212-24. PubMed ID: 300428
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  • 13. Mn and Mg influxes through Ca channels of motor nerve terminals are prevented by verapamil in frogs.
    Narita K, Kawasaki F, Kita H.
    Brain Res; 1990 Mar 05; 510(2):289-95. PubMed ID: 2158851
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  • 16. Dependence of spontaneous release at frog junctions on synaptic strength, external calcium and terminal length.
    Grinnell AD, Pawson PA.
    J Physiol; 1989 Nov 05; 418():397-410. PubMed ID: 2576068
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  • 17. Changes in MEPP frequency during depression of evoked release at the frog neuromuscular junction.
    Zengel JE, Sosa MA.
    J Physiol; 1994 Jun 01; 477(Pt 2):267-77. PubMed ID: 7932218
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  • 19. Mechanism of action of lead on neuromuscular junctions.
    Atchison WD, Narahashi T.
    Neurotoxicology; 1984 Jun 01; 5(3):267-82. PubMed ID: 6097847
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  • 20. Effect of alteration of nerve terminal Ca2+ regulation on increased spontaneous quantal release of acetylcholine by methyl mercury.
    Levesque PC, Atchison WD.
    Toxicol Appl Pharmacol; 1988 Jun 15; 94(1):55-65. PubMed ID: 3376114
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