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438 related items for PubMed ID: 6334697
1. Normal human alveolar macrophages obtained by bronchoalveolar lavage have a limited capacity to release interleukin-1. Wewers MD, Rennard SI, Hance AJ, Bitterman PB, Crystal RG. J Clin Invest; 1984 Dec; 74(6):2208-18. PubMed ID: 6334697 [Abstract] [Full Text] [Related]
9. Disparate cytochemical characteristics and production of cytokines and prostaglandin E2 by human mononuclear phagocytes from the blood, lung, and peritoneal cavity. Friedlander MA, Hilbert CM, Wu YC, Finegan CK, Rich EA. J Lab Clin Med; 1994 Apr 15; 123(4):574-84. PubMed ID: 8145006 [Abstract] [Full Text] [Related]
12. Release of interleukin-1 by alveolar macrophages of patients with active pulmonary sarcoidosis. Hunninghake GW. Am Rev Respir Dis; 1984 Apr 15; 129(4):569-72. PubMed ID: 6608889 [Abstract] [Full Text] [Related]
19. Neutrophil chemotactic factor produced by lipopolysaccharide (LPS)-stimulated human blood mononuclear leukocytes: partial characterization and separation from interleukin 1 (IL 1). Yoshimura T, Matsushima K, Oppenheim JJ, Leonard EJ. J Immunol; 1987 Aug 01; 139(3):788-93. PubMed ID: 3298433 [Abstract] [Full Text] [Related]
20. Pulmonary alveolar proteinosis is a disease of decreased availability of GM-CSF rather than an intrinsic cellular defect. Thomassen MJ, Yi T, Raychaudhuri B, Malur A, Kavuru MS. Clin Immunol; 2000 May 01; 95(2):85-92. PubMed ID: 10779401 [Abstract] [Full Text] [Related] Page: [Next] [New Search]