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Journal Abstract Search

166 related items for PubMed ID: 6389367

  • 1. Difference in susceptibility to gram-negative urinary tract infection between C3H/HeJ and C3H/HeN mice.
    Hagberg L, Hull R, Hull S, McGhee JR, Michalek SM, Svanborg Edén C.
    Infect Immun; 1984 Dec; 46(3):839-44. PubMed ID: 6389367
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  • 2. Bacterial virulence versus host resistance in the urinary tracts of mice.
    Svanborg-Edén C, Hagberg L, Hull R, Hull S, Magnusson KE, Ohman L.
    Infect Immun; 1987 May; 55(5):1224-32. PubMed ID: 3552994
    [Abstract] [Full Text] [Related]

  • 3. Inverse relationship between severity of experimental pyelonephritis and nitric oxide production in C3H/HeJ mice.
    Nowicki B, Singhal J, Fang L, Nowicki S, Yallampalli C.
    Infect Immun; 1999 May; 67(5):2421-7. PubMed ID: 10225904
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  • 4. Evidence for separate genetic defects in C3H/HeJ and C3HeB/FeJ mice, that affect susceptibility to gram-negative infections.
    Hagberg L, Briles DE, Edén CS.
    J Immunol; 1985 Jun; 134(6):4118-22. PubMed ID: 3886795
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  • 6. Murine immune responses to Salmonella lipopolysaccharide: oral administration of whole bacteria to C3H/HeJ mice induces secondary anti-LPS responses, especially of the IgA isotype.
    Jirillo E, Kiyono H, Michalek SM, McGhee JR.
    J Immunol; 1984 Apr; 132(4):1702-11. PubMed ID: 6366051
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  • 7. LPS regulation of the immune response: separate mechanisms for murine B cell activation by lipid A (direct) and polysaccharide (macrophage-dependent) derived from Bacteroides LPS.
    Williamson SI, Wannemuehler MJ, Jirillo E, Pritchard DG, Michalek SM, McGhee JR.
    J Immunol; 1984 Nov; 133(5):2294-300. PubMed ID: 6332842
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  • 8. Neutrophil recruitment and resistance to urinary tract infection.
    Haraoka M, Hang L, Frendéus B, Godaly G, Burdick M, Strieter R, Svanborg C.
    J Infect Dis; 1999 Oct; 180(4):1220-9. PubMed ID: 10479151
    [Abstract] [Full Text] [Related]

  • 9. Induction of inflammation by Escherichia coli on the mucosal level: requirement for adherence and endotoxin.
    Linder H, Engberg I, Baltzer IM, Jann K, Svanborg-Edén C.
    Infect Immun; 1988 May; 56(5):1309-13. PubMed ID: 2895744
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  • 10. Lack of association between the Tlr4 (Lpsd/Lpsd) genotype and increased susceptibility to Escherichia coli bladder infections in female C3H/HeJ mice.
    Suhs KA, Marthaler BR, Welch RA, Hopkins WJ.
    mBio; 2011 May; 2(3):e00094-11. PubMed ID: 21628500
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  • 12. The importance of a lipopolysaccharide-initiated, cytokine-mediated host defense mechanism in mice against extraintestinally invasive Escherichia coli.
    Cross A, Asher L, Seguin M, Yuan L, Kelly N, Hammack C, Sadoff J, Gemski P.
    J Clin Invest; 1995 Aug; 96(2):676-86. PubMed ID: 7635960
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  • 13. Ascending, unobstructed urinary tract infection in mice caused by pyelonephritogenic Escherichia coli of human origin.
    Hagberg L, Engberg I, Freter R, Lam J, Olling S, Svanborg Edén C.
    Infect Immun; 1983 Apr; 40(1):273-83. PubMed ID: 6339403
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  • 15. LPS regulation of the immune response: Bacteroides endotoxin induces mitogenic, polyclonal, and antibody responses in classical LPS responsive but not C3H/HeJ mice.
    Wannemuehler MJ, Michalek SM, Jirillo E, Williamson SI, Hirasawa M, McGhee JR.
    J Immunol; 1984 Jul; 133(1):299-305. PubMed ID: 6202784
    [Abstract] [Full Text] [Related]

  • 16. Lipopolysaccharide-responder and nonresponder C3H mouse strains are equally susceptible to an induced Escherichia coli urinary tract infection.
    Hopkins W, Gendron-Fitzpatrick A, McCarthy DO, Haine JE, Uehling DT.
    Infect Immun; 1996 Apr; 64(4):1369-72. PubMed ID: 8606102
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  • 18. Immunologic properties of bacterial lipopolysaccharide (LPS). II. The unresponsiveness of C3H/HeJ Mouse spleen cells to LPS-induced mitogenesis is dependent on the method used to extract LPS.
    Skidmore BJ, Morrison DC, Chiller JM, Weigle WO.
    J Exp Med; 1975 Dec 01; 142(6):1488-1508. PubMed ID: 1104747
    [Abstract] [Full Text] [Related]

  • 19. Functional role of interleukin 1 in periodontal disease: induction of interleukin 1 production by Bacteroides gingivalis lipopolysaccharide in peritoneal macrophages from C3H/HeN and C3H/HeJ mice.
    Hanazawa S, Nakada K, Ohmori Y, Miyoshi T, Amano S, Kitano S.
    Infect Immun; 1985 Oct 01; 50(1):262-70. PubMed ID: 3876285
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  • 20. Monoclonal antibodies to Salmonella lipopolysaccharide: anti-O-polysaccharide antibodies protect C3H mice against challenge with virulent Salmonella typhimurium.
    Colwell DE, Michalek SM, Briles DE, Jirillo E, McGhee JR.
    J Immunol; 1984 Aug 01; 133(2):950-7. PubMed ID: 6203984
    [Abstract] [Full Text] [Related]

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