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Journal Abstract Search

249 related items for PubMed ID: 6436037

  • 1. Quantification of in vivo spiperone binding in the rat striatum after lesions produced by kainate or decortication.
    Van der Werf JF, Van het Schip F, Sebens JB, Korf J.
    Eur J Pharmacol; 1984 Jul 20; 102(3-4):387-99. PubMed ID: 6436037
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  • 2. In vivo binding of N-n-propylnorapomorphine in the rat striatum: quantification after lesions produced by kainate, 6-hydroxydopamine and decortication.
    Van der Werf JF, Sebens JB, Korf J.
    Eur J Pharmacol; 1984 Jul 13; 102(2):251-9. PubMed ID: 6434329
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  • 3. Kainic acid lesions dissociate [3H] spiperone and [3H]cis-flupenthixol binding sites in rat striatum.
    Cross AJ, Waddington JL.
    Eur J Pharmacol; 1981 May 08; 71(2-3):327-32. PubMed ID: 7250192
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  • 4. Kainic acid lesions of striatum and decortication reduce specific [3H]sulpiride binding in rats, so D-2 receptors exist post-synaptically on corticostriate afferents and striatal neurons.
    Theodorou A, Reavill C, Jenner P, Marsden CD.
    J Pharm Pharmacol; 1981 Jul 08; 33(7):439-44. PubMed ID: 6115030
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  • 5. Relationship between dopamine receptor occupation by spiperone and acetylcholine levels in the rat striatum after long-term haloperidol treatment depends on dopamine innervation.
    Korf J, Sebens JB.
    J Neurochem; 1987 Feb 08; 48(2):516-21. PubMed ID: 2878979
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  • 8. 6-hydroxydopamine treatments enhance behavioral responses to intracerebral microinjection of D1- and D2-dopamine agonists into nucleus accumbens and striatum without changing dopamine antagonist binding.
    Breese GR, Duncan GE, Napier TC, Bondy SC, Iorio LC, Mueller RA.
    J Pharmacol Exp Ther; 1987 Jan 08; 240(1):167-76. PubMed ID: 3100767
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  • 9. Changes of the striatal 3H-spiperone binding 3-6 weeks after nigrostriatal denervation and after two years.
    Guerin B, Silice C, Mouchet P, Feuerstein C, Demenge P.
    Life Sci; 1985 Sep 09; 37(10):953-61. PubMed ID: 3162072
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  • 10. Dopamine denervation does not alter in vivo 3H-spiperone binding in rat striatum: implications for external imaging of dopamine receptors in Parkinson's disease.
    Bennett JP, Wooten GF.
    Ann Neurol; 1986 Apr 09; 19(4):378-83. PubMed ID: 3707090
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  • 11. The relationship between loss of dopamine nerve terminals, striatal [3H]spiroperidol binding and rotational behavior in unilaterally 6-hydroxydopamine-lesioned rats.
    Heikkila RE, Shapiro BS, Duvoisin RC.
    Brain Res; 1981 May 04; 211(2):285-92. PubMed ID: 7237124
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  • 12. Reduction of 3H-spiroperidol binding in rat striatum and frontal cortex by chronic amphetamine: dose response, time course and role of sustained dopamine release.
    Nielsen EB, Nielsen M, Braestrup C.
    Psychopharmacology (Berl); 1983 May 04; 81(1):81-5. PubMed ID: 6415736
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  • 14. Tracer and maximal specific binding of tritiated spiperone or N-n-propylnorapomorphine to quantify dopamine receptors in rat brain regions in vivo.
    Van der Werf JF, Sebens JB, Korf J.
    Life Sci; 1986 Jul 14; 39(2):155-60. PubMed ID: 3724366
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  • 15. Occupation of dopamine receptors by N-n-propylnorapomorphine or spiperone and acetylcholine levels in the rat striatum.
    Korf J, Sebens JB, Flentge F, van der Werf JF.
    J Neurochem; 1985 Jan 14; 44(1):314-8. PubMed ID: 3964835
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  • 18. Nigrostriatal lesions enhance striatal 3H-apomorphine and 3H-spiroperidol binding.
    Creese I, Snyder SH.
    Eur J Pharmacol; 1979 Jun 15; 56(3):277-81. PubMed ID: 38973
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  • 20. Dissociation between the presynaptic dopamine-sensitive adenylate cyclase and [3H]spiperone binding sites in rat substantia nigra.
    Quik M, Emson PC, Joyce E.
    Brain Res; 1979 May 11; 167(2):355-65. PubMed ID: 36204
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