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163 related items for PubMed ID: 6656764

  • 1. Properties of single-step mutants of Syrian hamster cell lines resistant to N-(phosphonacetyl)-L-aspartate.
    Zieg J, Clayton CE, Ardeshir F, Giulotto E, Swyryd EA, Stark GR.
    Mol Cell Biol; 1983 Nov; 3(11):2089-98. PubMed ID: 6656764
    [Abstract] [Full Text] [Related]

  • 2. Structure of amplified DNA in different Syrian hamster cell lines resistant to N-(phosphonacetyl)-L-aspartate.
    Ardeshir F, Giulotto E, Zieg J, Brison O, Liao WS, Stark GR.
    Mol Cell Biol; 1983 Nov; 3(11):2076-88. PubMed ID: 6656763
    [Abstract] [Full Text] [Related]

  • 3. Co-amplification of rRNA genes with CAD genes in N-(phosphonacetyl)-L-aspartate-resistant Syrian hamster cells.
    Wahl GM, Vitto L, Rubnitz J.
    Mol Cell Biol; 1983 Nov; 3(11):2066-75. PubMed ID: 6318080
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  • 8. The cloning and reintroduction into animal cells of a functional CAD gene, a dominant amplifiable genetic marker.
    de Saint Vincent BR, Delbrück S, Eckhart W, Meinkoth J, Vitto L, Wahl G.
    Cell; 1981 Dec; 27(2 Pt 1):267-77. PubMed ID: 6277500
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  • 9. Induction of CAD gene amplification by restriction endonucleases in V79,B7 Chinese hamster cells.
    Cavolina P, Agnese C, Maddalena A, Sciandrello G, Di Leonardo A.
    Mutat Res; 1989 Dec; 225(1-2):61-4. PubMed ID: 2563307
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  • 11. Structure of DNA formed in the first step of CAD gene amplification.
    Giulotto E, Saito I, Stark GR.
    EMBO J; 1986 Sep; 5(9):2115-21. PubMed ID: 3023056
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  • 12. The presence of amplified regions affects the stability of chromosomes in drug-resistant Chinese hamster cells.
    Miele M, Bonatti S, Menichini P, Ottaggio L, Abbondandolo A.
    Mutat Res; 1989 May; 219(3):171-8. PubMed ID: 2567961
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  • 13. Selection of N-(phosphonacetyl)-L-aspartate resistant Chinese hamster mutants in the presence of the uridine uptake inhibitor dipyridamole.
    Tessera L, Mucciolo E, Bertoni L, Giulotto E.
    Anticancer Res; 1995 May; 15(1):189-92. PubMed ID: 7733632
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  • 14. MYC abrogates p53-mediated cell cycle arrest in N-(phosphonacetyl)-L-aspartate-treated cells, permitting CAD gene amplification.
    Chernova OB, Chernov MV, Ishizaka Y, Agarwal ML, Stark GR.
    Mol Cell Biol; 1998 Jan; 18(1):536-45. PubMed ID: 9418900
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  • 15. The propensity for gene amplification: a comparison of protocols, cell lines, and selection agents.
    Sharma RC, Schimke RT.
    Mutat Res; 1994 Jan 16; 304(2):243-60. PubMed ID: 7506368
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  • 16. Single-copy and amplified CAD genes in Syrian hamster chromosomes localized by a highly sensitive method for in situ hybridization.
    Wahl GM, Vitto L, Padgett RA, Stark GR.
    Mol Cell Biol; 1982 Mar 16; 2(3):308-19. PubMed ID: 6180304
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  • 17. Gene amplification and gene deletion are induced at different rates in V79/AP4 Chinese hamster cells.
    Vatteroni L, Piras A, Moretti A, Rainaldi G.
    Mutagenesis; 1993 Sep 16; 8(5):411-5. PubMed ID: 7901735
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  • 18. Inefficient growth arrest in response to dNTP starvation stimulates gene amplification through bridge-breakage-fusion cycles.
    Poupon MF, Smith KA, Chernova OB, Gilbert C, Stark GR.
    Mol Biol Cell; 1996 Mar 16; 7(3):345-54. PubMed ID: 8868464
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  • 19. General method for cloning amplified DNA by differential screening with genomic probes.
    Brison O, Ardeshir F, Stark GR.
    Mol Cell Biol; 1982 May 16; 2(5):578-87. PubMed ID: 6896736
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  • 20. Protein alterations associated with gene amplification in cultured human and rodent cells.
    Conner EA, Wirth PJ.
    Electrophoresis; 1996 Jul 16; 17(7):1257-64. PubMed ID: 8855414
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