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Journal Abstract Search

331 related items for PubMed ID: 6799331

  • 21. In situ injection of kainic acid: a new method for selectively lesioning neural cell bodies while sparing axons of passage.
    Coyle JT, Molliver ME, Kuhar MJ.
    J Comp Neurol; 1978 Jul 15; 180(2):301-23. PubMed ID: 659663
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  • 22. [Effects of different doses of apomorphine on the glutamate decarboxylase activity of the substantia nigra and the medial basal hypothalamus].
    Giammona G, Pattí F, Sambataro V, Reggio A, Rampello L, Di Giorgio RM, Maccagnano C, Condorelli DF, Nicoletti F.
    Boll Soc Ital Biol Sper; 1981 Oct 30; 57(20):2074-9. PubMed ID: 7317201
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  • 23. Postsynaptic receptors are not essential for dopaminergic feedback regulation.
    Di Chiara G, Porceddu ML, Fratta W, Gessa GL.
    Nature; 1977 May 19; 267(5608):270-2. PubMed ID: 865621
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  • 29. Synaptic reorganisation in the rat striatum after dopaminergic deafferentation: an ultrastructural study using glutamate decarboxylase immunocytochemistry.
    Nitsch C, Riesenberg R.
    Synapse; 1995 Apr 19; 19(4):247-63. PubMed ID: 7792720
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  • 30. Binding of [3H]apomorphine to striatal membranes prepared from rat brain after 6-hydroxydopamine and kainic acid lesions.
    Fujita N, Saito K, Iwatsubo K, Hirata A, Noguchi Y, Yoshida H.
    Brain Res; 1980 May 26; 190(2):593-6. PubMed ID: 7370810
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  • 31. Long-term sequelae of striatal kainate lesion.
    Zaczek R, Schwarcz R, Coyle JT.
    Brain Res; 1978 Sep 08; 152(3):626-32. PubMed ID: 28812
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  • 32. Effects of intrastriatal kainic acid injection on [3H]dopamine metabolism in rat striatal slices: evidence for postsynaptic glial cell metabolism by both the type A and B forms of monoamine oxidase.
    Schoepp DD, Azzaro AJ.
    J Neurochem; 1983 May 08; 40(5):1340-8. PubMed ID: 6131940
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  • 34. Loss of striatal dopaminergic receptors after intrastriatal kainic acid injection.
    Fields JZ, Reisine TD, Yamamura HI.
    Life Sci; 1978 Aug 14; 23(6):569-73. PubMed ID: 692281
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  • 35. Bilateral injection of kainic acid into the rat striatum potentiates morphine, arecoline and pilocarpine but not haloperidol catalepsy.
    Turski W, Czuczwar SJ, Turski L, Kleinrok Z.
    Methods Find Exp Clin Pharmacol; 1982 Aug 14; 4(5):287-91. PubMed ID: 6889661
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  • 36. Evidence for dopamine autoreceptors controlling dopamine synthesis in the substantia nigra.
    Argiolas A, Melis MR, Fadda F, Gessa GL.
    Brain Res; 1982 Feb 18; 234(1):177-81. PubMed ID: 6800566
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  • 37. Persistence of beta-adrenergic receptors in rat striatum following kainic acid administration.
    Zahniser NR, Minneman KP, Molinoff PB.
    Brain Res; 1979 Dec 14; 178(2-3):589-95. PubMed ID: 228797
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  • 38. Influence of lithium on biochemical manifestations of striatal dopamine target cell supersensitivity induced by prolonged haloperidol treatment.
    Le Douarin C, Oblin A, Fage D, Scatton B.
    Eur J Pharmacol; 1983 Sep 16; 93(1-2):55-62. PubMed ID: 6628547
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  • 39. Chronic modification of dopaminergic transmission and its effects on binding characteristics of dopamine transporter ligand 3H-GBR 12783 in the striatum.
    Valchár M, Bonnet JJ, Costentin J.
    Act Nerv Super (Praha); 1989 Dec 16; 31(4):273-4. PubMed ID: 2561622
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