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Journal Abstract Search

164 related items for PubMed ID: 7156231

  • 1. Area postrema lesions increase drinking to angiotensin and extracellular dehydration.
    Edwards GL, Ritter RC.
    Physiol Behav; 1982 Nov; 29(5):943-7. PubMed ID: 7156231
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  • 2. Preoptic-hypothalamic periventricular lesions: thirst deficits and hypernatremia.
    Buggy J, Jonhson AK.
    Am J Physiol; 1977 Jul; 233(1):R44-52. PubMed ID: 879343
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  • 4. Brain stem mechanisms and the inhibition of angiotensin-induced drinking.
    Ohman LE, Johnson AK.
    Am J Physiol; 1989 Jan; 256(1 Pt 2):R264-9. PubMed ID: 2912220
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  • 9. Localization of central nervous system structures mediating extracellular thirst in the female rat.
    Kucharczyk J.
    J Endocrinol; 1984 Feb; 100(2):183-8. PubMed ID: 6537965
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  • 12. Area postrema lesions produce feeding deficits in the rat: effects of preoperative dieting and 2-deoxy-D-glucose.
    Contreras RJ, Fox E, Drugovich ML.
    Physiol Behav; 1982 Nov; 29(5):875-84. PubMed ID: 7156225
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  • 13. Differential effects of brain lesions on thirst induced by the administration of angiotensin-II to the preoptic region, subfornical organ and anterior third ventricle.
    Kucharczyk J, Assaf SY, Mogenson GJ.
    Brain Res; 1976 May 28; 108(2):327-37. PubMed ID: 1276900
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  • 17. Rats with area postrema lesions have lengthy eating and drinking bouts when fed ad libitum: implications for feedback inhibition of ingestive behavior.
    Stricker EM, Curtis KS, Peacock KA, Smith JC.
    Behav Neurosci; 1997 Jun 28; 111(3):623-32. PubMed ID: 9189277
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  • 18. Lesions in lateral parabrachial nucleus enhance drinking to angiotensin II and isoproterenol.
    Ohman LE, Johnson AK.
    Am J Physiol; 1986 Sep 28; 251(3 Pt 2):R504-9. PubMed ID: 3752284
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  • 20. Brain mechanisms and drinking: the role of lamina terminalis-associated systems in extracellular thirst.
    Johnson AK, Cunningham JT.
    Kidney Int Suppl; 1987 Aug 28; 21():S35-42. PubMed ID: 3306105
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