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Journal Abstract Search

166 related items for PubMed ID: 7364046

  • 1.
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  • 2. Ionic changes and alterations in the size of the extracellular space during epileptic activity.
    Lux HD, Heinemann U, Dietzel I.
    Adv Neurol; 1986; 44():619-39. PubMed ID: 3518349
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  • 5. Analysis of K+ accumulation reveals privileged extracellular region in the vicinity of glial cells in situ.
    Chvátal A, Anderová M, Syková E.
    J Neurosci Res; 2004 Dec 01; 78(5):668-82. PubMed ID: 15478195
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  • 6. [The role of the glial cells in the maintenance of the ionic environment of the photoreceptors of the retina of the drone (author's transl)].
    Tsacopoulos M, Coles JA.
    Klin Monbl Augenheilkd; 1978 Apr 01; 172(4):449-51. PubMed ID: 651211
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  • 7. Regulation of extracellular potassium concentration in epileptogenesis.
    Pedley TA, Fisher RS, Futamachi KJ, Prince DA.
    Fed Proc; 1976 May 01; 35(6):1254-9. PubMed ID: 816678
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  • 8. Relations between slow extracellular potential changes, glial potassium buffering, and electrolyte and cellular volume changes during neuronal hyperactivity in cat brain.
    Dietzel I, Heinemann U, Lux HD.
    Glia; 1989 May 01; 2(1):25-44. PubMed ID: 2523337
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  • 9. Do neuronal signals regulate potassium flow in glial cells? Evidence from an invertebrate central nervous system.
    Walz W.
    J Neurosci Res; 1982 May 01; 7(1):71-9. PubMed ID: 7069800
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  • 10. [Reactions of nervous elements to K+ accumulation in the medium and functional potassium feedback at the synapse].
    Matiushkin DP.
    Fiziol Zh SSSR Im I M Sechenova; 1976 Dec 01; 62(12):1834-41. PubMed ID: 1010069
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  • 13. Synaptic feed-backs mediated by potassium ions.
    Matyushkin DP, Krivoi II, Drabkina TM.
    Gen Physiol Biophys; 1995 Oct 01; 14(5):369-81. PubMed ID: 8786037
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  • 15. Model of potassium dynamics in the central nervous system.
    Odette LL, Newman EA.
    Glia; 1988 Oct 01; 1(3):198-210. PubMed ID: 2976039
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