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Journal Abstract Search

106 related items for PubMed ID: 7470030

  • 1. Isolated rat heart mitochondria are able to metabolize pent-4-enoate to tricarboxylic acid-cycle intermediates.
    Hiltunen JK, Kauppinen RA, Nuutinen EM, Peuhkurinen KJ, Hassinen IE.
    Biochem J; 1980 Jun 15; 188(3):725-9. PubMed ID: 7470030
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  • 2. Metabolism of pent-4-enoate in rat heart. Reduction of the double bond.
    Hiltunen JK, Davis EJ.
    Biochem J; 1981 Feb 15; 194(2):427-32. PubMed ID: 7305999
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  • 3. [Substrate inhibition in the tricarboxylic acid cycle].
    Dynnik VV, Maevskiĭ EI, Grigorenko EV, Kim IuV.
    Biofizika; 1984 Feb 15; 29(6):954-8. PubMed ID: 6518172
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  • 4. Biochemical effects of the hypoglycaemic compound pent-4-enoic acid and related non-hypoglycaemic fatty acids. Effects of the free acids and their carnitine esters on coenzyme A-dependent oxidations in rat liver mitochondria.
    Holland PC, Sherratt HS.
    Biochem J; 1973 Sep 15; 136(1):157-71. PubMed ID: 4772622
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  • 11. In vivo induction of 4-enoyl-CoA reductase by clofibrate in liver mitochondria and its effect on pent-4-enoate metabolism.
    Borrebaek B, Osmundsen H, Bremer J.
    Biochem Biophys Res Commun; 1980 Apr 29; 93(4):1173-80. PubMed ID: 7396904
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  • 12. The disposition of citric acid cycle intermediates by isolated rat heart mitochondria.
    Hiltunen JK, Davis EJ.
    Biochim Biophys Acta; 1981 Nov 18; 678(1):115-21. PubMed ID: 7306575
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  • 13. Accumulation and disposal of tricarboxylic acid cycle intermediates during propionate oxidation in the isolated perfused rat heart.
    Peuhkurinen KJ.
    Biochim Biophys Acta; 1982 Oct 11; 721(2):124-34. PubMed ID: 7138913
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  • 14. Inhibition of urea synthesis by pent-4-enoate associated with decrease in N-acetyl-L-glutamate concentration in isolated rat hepatocytes.
    Aoyagi K, Mori M, Tatibana M.
    Biochim Biophys Acta; 1979 Nov 01; 587(4):515-21. PubMed ID: 508801
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  • 15. Protection of rats by clofibrate against the hypoglycaemic and toxic effects of hypoglycin and pent-4-enoate. An ultrastructural and biochemical study.
    Van Hoof F, Hue L, Vamecq J, Sherratt HS.
    Biochem J; 1985 Jul 15; 229(2):387-97. PubMed ID: 4038275
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  • 18. Elimination and replenishment of tricarboxylic acid-cycle intermediates in myocardium.
    Nuutinen EM, Peuhkurinen KJ, Pietiläinen EP, Hiltunen JK, Hassinen IE.
    Biochem J; 1981 Mar 15; 194(3):867-75. PubMed ID: 6796067
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  • 19. Effects of mitochondria concentration and age, energy and inhibitors of mitochondrial anion transport on malate transport in isolated rat heart mitochondria.
    Barritt GJ.
    Int J Biochem; 1978 Mar 15; 9(5):317-22. PubMed ID: 566683
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  • 20. Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
    Jespersen NR, Yokota T, Støttrup NB, Bergdahl A, Paelestik KB, Povlsen JA, Dela F, Bøtker HE.
    J Physiol; 2017 Jun 15; 595(12):3765-3780. PubMed ID: 28093764
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