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Journal Abstract Search

134 related items for PubMed ID: 7622355

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  • 5. Neutrophils are required for endotoxin-induced myocardial cross-tolerance to ischemia-reperfusion injury.
    Meldrum DR, Sheridan BC, Cleveland JC, Fullerton DA, Banerjee A, Harken AH.
    Arch Surg; 1996 Nov; 131(11):1203-8. PubMed ID: 8911261
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  • 6. Changes in P-selectin expression on cardiac microvessels in blood-perfused rat hearts subjected to ischemia-reperfusion.
    Chukwuemeka AO, Brown KA, Venn GE, Chambers DJ.
    Ann Thorac Surg; 2005 Jan; 79(1):204-11. PubMed ID: 15620944
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  • 7. IL-10 is induced in the reperfused myocardium and may modulate the reaction to injury.
    Frangogiannis NG, Mendoza LH, Lindsey ML, Ballantyne CM, Michael LH, Smith CW, Entman ML.
    J Immunol; 2000 Sep 01; 165(5):2798-808. PubMed ID: 10946312
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  • 10. Opposing effects mediated by the chemokine receptor CXCR2 on myocardial ischemia-reperfusion injury: recruitment of potentially damaging neutrophils and direct myocardial protection.
    Tarzami ST, Miao W, Mani K, Lopez L, Factor SM, Berman JW, Kitsis RN.
    Circulation; 2003 Nov 11; 108(19):2387-92. PubMed ID: 14568904
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  • 12. Attenuation of interleukin-8 expression in C6-deficient rabbits after myocardial ischemia/reperfusion.
    Kilgore KS, Park JL, Tanhehco EJ, Booth EA, Marks RM, Lucchesi BR.
    J Mol Cell Cardiol; 1998 Jan 11; 30(1):75-85. PubMed ID: 9500866
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  • 19. The blood contribution to early myocardial reperfusion injury is amplified in diabetes.
    McDonagh PF, Hokama JY, Copeland JG, Reynolds JM.
    Diabetes; 1997 Nov 11; 46(11):1859-67. PubMed ID: 9356037
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