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Journal Abstract Search

211 related items for PubMed ID: 7646476

  • 1. Residues in the synuclein consensus motif of the alpha-synuclein fragment, NAC, participate in transglutaminase-catalysed cross-linking to Alzheimer-disease amyloid beta A4 peptide.
    Jensen PH, Sørensen ES, Petersen TE, Gliemann J, Rasmussen LK.
    Biochem J; 1995 Aug 15; 310 ( Pt 1)(Pt 1):91-4. PubMed ID: 7646476
    [Abstract] [Full Text] [Related]

  • 2. Identification of glutamine and lysine residues in Alzheimer amyloid beta A4 peptide responsible for transglutaminase-catalysed homopolymerization and cross-linking to alpha 2M receptor.
    Rasmussen LK, Sørensen ES, Petersen TE, Gliemann J, Jensen PH.
    FEBS Lett; 1994 Jan 31; 338(2):161-6. PubMed ID: 7905838
    [Abstract] [Full Text] [Related]

  • 3. Covalent blocking of fibril formation and aggregation of intracellular amyloidgenic proteins by transglutaminase-catalyzed intramolecular cross-linking.
    Konno T, Morii T, Hirata A, Sato S, Oiki S, Ikura K.
    Biochemistry; 2005 Feb 15; 44(6):2072-9. PubMed ID: 15697232
    [Abstract] [Full Text] [Related]

  • 4. Cross-linking of a synthetic partial-length (1-28) peptide of the Alzheimer beta/A4 amyloid protein by transglutaminase.
    Ikura K, Takahata K, Sasaki R.
    FEBS Lett; 1993 Jul 12; 326(1-3):109-11. PubMed ID: 8100780
    [Abstract] [Full Text] [Related]

  • 5. Non-Abeta component of Alzheimer's disease amyloid (NAC) revisited. NAC and alpha-synuclein are not associated with Abeta amyloid.
    Culvenor JG, McLean CA, Cutt S, Campbell BC, Maher F, Jäkälä P, Hartmann T, Beyreuther K, Masters CL, Li QX.
    Am J Pathol; 1999 Oct 12; 155(4):1173-81. PubMed ID: 10514400
    [Abstract] [Full Text] [Related]

  • 6. Formation of hydrogen peroxide and hydroxyl radicals from A(beta) and alpha-synuclein as a possible mechanism of cell death in Alzheimer's disease and Parkinson's disease.
    Tabner BJ, Turnbull S, El-Agnaf OM, Allsop D.
    Free Radic Biol Med; 2002 Jun 01; 32(11):1076-83. PubMed ID: 12031892
    [Abstract] [Full Text] [Related]

  • 7. Transglutaminase facilitates the formation of polymers of the beta-amyloid peptide.
    Dudek SM, Johnson GV.
    Brain Res; 1994 Jul 18; 651(1-2):129-33. PubMed ID: 7922559
    [Abstract] [Full Text] [Related]

  • 8. Oligopeptide-mediated acceleration of amyloid fibril formation of amyloid beta(Abeta) and alpha-synuclein fragment peptide (NAC).
    Kuroda Y, Maeda Y, Hanaoka H, Miyamoto K, Nakagawa T.
    J Pept Sci; 2004 Jan 18; 10(1):8-17. PubMed ID: 14959887
    [Abstract] [Full Text] [Related]

  • 9. NACP, the precursor protein of the non-amyloid beta/A4 protein (A beta) component of Alzheimer disease amyloid, binds A beta and stimulates A beta aggregation.
    Yoshimoto M, Iwai A, Kang D, Otero DA, Xia Y, Saitoh T.
    Proc Natl Acad Sci U S A; 1995 Sep 26; 92(20):9141-5. PubMed ID: 7568089
    [Abstract] [Full Text] [Related]

  • 10. Copper(II)-induced self-oligomerization of alpha-synuclein.
    Paik SR, Shin HJ, Lee JH, Chang CS, Kim J.
    Biochem J; 1999 Jun 15; 340 ( Pt 3)(Pt 3):821-8. PubMed ID: 10359669
    [Abstract] [Full Text] [Related]

  • 11. Self-oligomerization of NACP, the precursor protein of the non-amyloid beta/A4 protein (A beta) component of Alzheimer's disease amyloid, observed in the presence of a C-terminal A beta fragment (residues 25-35).
    Paik SR, Lee JH, Kim DH, Chang CS, Kim YS.
    FEBS Lett; 1998 Jan 02; 421(1):73-6. PubMed ID: 9462843
    [Abstract] [Full Text] [Related]

  • 12. Toxicity of non-abeta component of Alzheimer's disease amyloid, and N-terminal fragments thereof, correlates to formation of beta-sheet structure and fibrils.
    Bodles AM, Guthrie DJ, Harriott P, Campbell P, Irvine GB.
    Eur J Biochem; 2000 Apr 02; 267(8):2186-94. PubMed ID: 10759841
    [Abstract] [Full Text] [Related]

  • 13. Binding of Abeta to alpha- and beta-synucleins: identification of segments in alpha-synuclein/NAC precursor that bind Abeta and NAC.
    Jensen PH, Hojrup P, Hager H, Nielsen MS, Jacobsen L, Olesen OF, Gliemann J, Jakes R.
    Biochem J; 1997 Apr 15; 323 ( Pt 2)(Pt 2):539-46. PubMed ID: 9163350
    [Abstract] [Full Text] [Related]

  • 14. In vitro preparation of prefibrillar intermediates of amyloid-beta and alpha-synuclein.
    Lashuel HA, Grillo-Bosch D.
    Methods Mol Biol; 2005 Apr 15; 299():19-33. PubMed ID: 15980593
    [Abstract] [Full Text] [Related]

  • 15. Identification of the region of non-Abeta component (NAC) of Alzheimer's disease amyloid responsible for its aggregation and toxicity.
    Bodles AM, Guthrie DJ, Greer B, Irvine GB.
    J Neurochem; 2001 Jul 15; 78(2):384-95. PubMed ID: 11461974
    [Abstract] [Full Text] [Related]

  • 16. Non-Amyloid-β Component of Human α-Synuclein Oligomers Induces Formation of New Aβ Oligomers: Insight into the Mechanisms That Link Parkinson's and Alzheimer's Diseases.
    Atsmon-Raz Y, Miller Y.
    ACS Chem Neurosci; 2016 Jan 20; 7(1):46-55. PubMed ID: 26479553
    [Abstract] [Full Text] [Related]

  • 17. beta-amyloid peptides enhance alpha-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's disease.
    Masliah E, Rockenstein E, Veinbergs I, Sagara Y, Mallory M, Hashimoto M, Mucke L.
    Proc Natl Acad Sci U S A; 2001 Oct 09; 98(21):12245-50. PubMed ID: 11572944
    [Abstract] [Full Text] [Related]

  • 18. Molecular cloning of cDNA encoding an unrecognized component of amyloid in Alzheimer disease.
    Uéda K, Fukushima H, Masliah E, Xia Y, Iwai A, Yoshimoto M, Otero DA, Kondo J, Ihara Y, Saitoh T.
    Proc Natl Acad Sci U S A; 1993 Dec 01; 90(23):11282-6. PubMed ID: 8248242
    [Abstract] [Full Text] [Related]

  • 19. NACP/alpha-synuclein, NAC, and beta-amyloid pathology of familial Alzheimer's disease with the E184D presenilin-1 mutation: a clinicopathological study of two autopsy cases.
    Yokota O, Terada S, Ishizu H, Ujike H, Ishihara T, Nakashima H, Yasuda M, Kitamura Y, Uéda K, Checler F, Kuroda S.
    Acta Neuropathol; 2002 Dec 01; 104(6):637-48. PubMed ID: 12410385
    [Abstract] [Full Text] [Related]

  • 20. Transglutaminase-mediated intramolecular cross-linking of membrane-bound alpha-synuclein promotes amyloid formation in Lewy bodies.
    Nemes Z, Petrovski G, Aerts M, Sergeant K, Devreese B, Fésüs L.
    J Biol Chem; 2009 Oct 02; 284(40):27252-64. PubMed ID: 19651786
    [Abstract] [Full Text] [Related]

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