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Journal Abstract Search

160 related items for PubMed ID: 7668333

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  • 6. Mitochondrial damage results in a reversible increase in lysosomal storage material in lymphoblasts from patients with juvenile neuronal ceroid-lipofuscinosis (Batten Disease).
    Boriack RL, Cortinas E, Bennett MJ.
    Am J Med Genet; 1995 Jun 05; 57(2):301-3. PubMed ID: 7668350
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  • 7. Progress in neuropathology of the neuronal ceroid lipofuscinoses.
    Goebel HH, Schochet SS, Jaynes M, Brück W, Kohlschütter A, Hentati F.
    Mol Genet Metab; 1999 Apr 05; 66(4):367-72. PubMed ID: 10191130
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  • 8. Decreased lysosomal subunit c-degrading activity in fibroblasts from patients with late infantile neuronal ceroid lipofuscinosis.
    Ezaki J, Wolfe LS, Kominami E.
    Neuropediatrics; 1997 Feb 05; 28(1):53-5. PubMed ID: 9151323
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  • 9. Sanfilippo disease, type A with some features of ceroid lipofuscinosis.
    Wisniewski K, Rudelli R, Laure-Kamionowska M, Sklower S, Houck GE, Kieras F, Ramos P, Wisniewski HM, Braak H.
    Neuropediatrics; 1985 May 05; 16(2):98-105. PubMed ID: 3925366
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  • 13. Diagnosis of neuronal ceroid lipofuscinosis (Batten disease) by electron microscopy in peripheral blood specimens.
    Anderson GW, Smith VV, Brooke I, Malone M, Sebire NJ.
    Ultrastruct Pathol; 2006 May 05; 30(5):373-8. PubMed ID: 17090516
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  • 19. Late infantile neuronal ceroid lipofuscinosis: a new mutation in Arabs.
    Goldberg-Stern H, Halevi A, Marom D, Straussberg R, Mimouni-Bloch A.
    Pediatr Neurol; 2009 Oct 05; 41(4):297-300. PubMed ID: 19748052
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  • 20. Lysosomal degradation of cholecystokinin-(29-33)-amide in mouse brain is dependent on tripeptidyl peptidase-I: implications for the degradation and storage of peptides in classical late-infantile neuronal ceroid lipofuscinosis.
    Bernardini F, Warburton MJ.
    Biochem J; 2002 Sep 01; 366(Pt 2):521-9. PubMed ID: 12038963
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