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Journal Abstract Search


394 related items for PubMed ID: 7685791

  • 1. Dissection of the function of HLA class II and costimulation in B cell-mediated and toxic shock syndrome toxin-1-induced T cell proliferation.
    Dennig D, O'Reilly RJ.
    J Immunol; 1993 Jun 15; 150(12):5231-40. PubMed ID: 7685791
    [Abstract] [Full Text] [Related]

  • 2. A novel HLA class II-independent TCR-mediated T cell activation mechanism is distinguished by the V beta specificity of the proliferating oligoclones and their capacity to generate interleukin-2.
    Dennig D, Yan Y, Ferguson K, O'Reilly RJ.
    Cell Immunol; 1996 Aug 01; 171(2):200-10. PubMed ID: 8806788
    [Abstract] [Full Text] [Related]

  • 3. Activation of human T cells by toxic shock syndrome toxin-1: the toxin-binding structures expressed on human lymphoid cells acting as accessory cells are HLA class II molecules.
    Uchiyama T, Imanishi K, Saito S, Araake M, Yan XJ, Fujikawa H, Igarashi H, Kato H, Obata F, Kashiwagi N.
    Eur J Immunol; 1989 Oct 01; 19(10):1803-9. PubMed ID: 2583222
    [Abstract] [Full Text] [Related]

  • 4. Intercellular adhesion molecule-1 and leukocyte function-associated antigen-3 provide costimulation for superantigen-induced T lymphocyte proliferation in the absence of a specific presenting molecule.
    Lamphear JG, Stevens KR, Rich RR.
    J Immunol; 1998 Jan 15; 160(2):615-23. PubMed ID: 9551895
    [Abstract] [Full Text] [Related]

  • 5. Activation of murine T cells by toxic shock syndrome toxin-1. The toxin-binding structures expressed on murine accessory cells are MHC class II molecules.
    Uchiyama T, Tadakuma T, Imanishi K, Araake M, Saito S, Yan XJ, Fujikawa H, Igarashi H, Yamaura N.
    J Immunol; 1989 Nov 15; 143(10):3175-82. PubMed ID: 2509554
    [Abstract] [Full Text] [Related]

  • 6. Accessory cell function of keratinocytes for superantigens. Dependence on lymphocyte function-associated antigen-1/intercellular adhesion molecule-1 interaction.
    Nickoloff BJ, Mitra RS, Green J, Zheng XG, Shimizu Y, Thompson C, Turka LA.
    J Immunol; 1993 Mar 15; 150(6):2148-59. PubMed ID: 8450207
    [Abstract] [Full Text] [Related]

  • 7. Mechanism of Staphylococcus aureus exotoxin A inhibition of Ig production by human B cells.
    Moseley AB, Huston DP.
    J Immunol; 1991 Feb 01; 146(3):826-32. PubMed ID: 1988499
    [Abstract] [Full Text] [Related]

  • 8. Proliferation of human T lymphocytes induced with superantigens is not dependent on costimulation by the CD28 counter-receptor B7.
    Damle NK, Klussman K, Leytze G, Linsley PS.
    J Immunol; 1993 Feb 01; 150(3):726-35. PubMed ID: 7678619
    [Abstract] [Full Text] [Related]

  • 9. Superantigen and HLA-DR ligation induce phospholipase-C gamma 1 activation in class II+ T cells.
    Kanner SB, Odum N, Grosmaire L, Masewicz S, Svejgaard A, Ledbetter JA.
    J Immunol; 1992 Dec 01; 149(11):3482-8. PubMed ID: 1385526
    [Abstract] [Full Text] [Related]

  • 10. Binding of toxic shock syndrome toxin-1 to murine major histocompatibility complex class II molecules.
    Scholl PR, Sekaly RP, Diez A, Glimcher LH, Geha RS.
    Eur J Immunol; 1990 Sep 01; 20(9):1911-6. PubMed ID: 2209697
    [Abstract] [Full Text] [Related]

  • 11. Engagement of MHC-class II molecules by staphylococcal exotoxins delivers a comitogenic signal to human B cells.
    Fuleihan R, Mourad W, Geha RS, Chatila T.
    J Immunol; 1991 Mar 01; 146(5):1661-6. PubMed ID: 1847171
    [Abstract] [Full Text] [Related]

  • 12. IL-7 induces surface expression of B7/BB1 on pre-B cells and an associated increase in their costimulatory effects on T cell proliferation.
    Dennig D, O'Reilly RJ.
    Cell Immunol; 1994 Jan 01; 153(1):227-38. PubMed ID: 7507002
    [Abstract] [Full Text] [Related]

  • 13. Staphylococcal enterotoxin B and toxic shock syndrome toxin-1 bind to distinct sites on HLA-DR and HLA-DQ molecules.
    Scholl PR, Diez A, Geha RS.
    J Immunol; 1989 Oct 15; 143(8):2583-8. PubMed ID: 2551962
    [Abstract] [Full Text] [Related]

  • 14. Molecular requirements for T cell activation by the staphylococcal toxic shock syndrome toxin-1.
    Norton SD, Schlievert PM, Novick RP, Jenkins MK.
    J Immunol; 1990 Mar 15; 144(6):2089-95. PubMed ID: 2313089
    [Abstract] [Full Text] [Related]

  • 15. The alpha 1 domain of the HLA-DR molecule is essential for high-affinity binding of the toxic shock syndrome toxin-1.
    Karp DR, Teletski CL, Scholl P, Geha R, Long EO.
    Nature; 1990 Aug 02; 346(6283):474-6. PubMed ID: 2377209
    [Abstract] [Full Text] [Related]

  • 16. Major histocompatibility complex class II-associated peptides determine the binding of the superantigen toxic shock syndrome toxin-1.
    von Bonin A, Ehrlich S, Malcherek G, Fleischer B.
    Eur J Immunol; 1995 Oct 02; 25(10):2894-8. PubMed ID: 7589089
    [Abstract] [Full Text] [Related]

  • 17. The staphylococcal toxic shock syndrome toxin 1 triggers B cell proliferation and differentiation via major histocompatibility complex-unrestricted cognate T/B cell interaction.
    Mourad W, Scholl P, Diaz A, Geha R, Chatila T.
    J Exp Med; 1989 Dec 01; 170(6):2011-22. PubMed ID: 2584933
    [Abstract] [Full Text] [Related]

  • 18. Autologous B lymphoblastoid cell lines and long-term cultured T cells as stimulators in the mixed lymphocyte reaction: analysis of the role of HLA class II antigens as stimulatory molecules.
    Santoli D, Radka SF, Igarashi M, Kreider BL, Ferrone S.
    J Immunol; 1986 Jul 15; 137(2):400-7. PubMed ID: 2941479
    [Abstract] [Full Text] [Related]

  • 19. Regulation of superantigen-induced T cell activation in the absence and the presence of MHC class II.
    Lando PA, Olsson C, Kalland T, Newton D, Kotb M, Dohlsten M.
    J Immunol; 1996 Oct 01; 157(7):2857-63. PubMed ID: 8816390
    [Abstract] [Full Text] [Related]

  • 20. Staphylococcal toxins bind to different sites on HLA-DR.
    Chintagumpala MM, Mollick JA, Rich RR.
    J Immunol; 1991 Dec 01; 147(11):3876-81. PubMed ID: 1940373
    [Abstract] [Full Text] [Related]


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