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Journal Abstract Search

134 related items for PubMed ID: 7735144

  • 1. Spontaneous overexpression of heat-shock proteins in Ehrlich ascites carcinoma cells during in vivo growth.
    Gabai VL, Mosina VA, Budagova KR, Kabakov AE.
    Biochem Mol Biol Int; 1995 Jan; 35(1):95-102. PubMed ID: 7735144
    [Abstract] [Full Text] [Related]

  • 2. Adaptation of Ehrlich ascites carcinoma cells to energy deprivation in vivo can be associated with heat shock protein accumulation.
    Kabakov AE, Molotkov AO, Budagova KR, Makarova YuM, Mosin AF, Gabai VL.
    J Cell Physiol; 1995 Oct; 165(1):1-6. PubMed ID: 7559791
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  • 3. Stress-induced insolubilization of certain proteins in ascites tumor cells.
    Kabakov AE, Gabai VL.
    Arch Biochem Biophys; 1994 Mar; 309(2):247-53. PubMed ID: 8135534
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  • 4. Heat shock-induced accumulation of 70-kDa stress protein (HSP70) can protect ATP-depleted tumor cells from necrosis.
    Kabakov AE, Gabai VL.
    Exp Cell Res; 1995 Mar; 217(1):15-21. PubMed ID: 7867713
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  • 5. [Damage to and interphase death of Ehrlich ascites carcinoma tumor cells at different growth stages during energy starvation and heat shock].
    Mosin AF, Gabaĭ VL, Makarova IuM, Mosina VA.
    Tsitologiia; 1994 Mar; 36(4):384-92. PubMed ID: 7809976
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  • 6. Induction of heat-shock protein synthesis and thermotolerance in EL-4 ascites tumor cells by transient ATP depletion after ischemic stress.
    Gabai VL, Kabakov AE.
    Exp Mol Pathol; 1994 Apr; 60(2):88-99. PubMed ID: 8070544
    [Abstract] [Full Text] [Related]

  • 7. The expression of the growth-related 25kDa protein (p25) of Ehrlich ascites tumor cells is increased by hyperthermic treatment (heat shock).
    Oesterreich S, Benndorf R, Bielka H.
    Biomed Biochim Acta; 1990 Apr; 49(4):219-26. PubMed ID: 2403339
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  • 8. Inhibition of colony formation of NIH 3T3 cells by the expression of the small molecular weight heat shock protein HSP27: involvement of its phosphorylation and aggregation at the C-terminal region.
    Arata S, Hamaguchi S, Nose K.
    J Cell Physiol; 1997 Jan; 170(1):19-26. PubMed ID: 9012781
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  • 9. Expression of HSP27 results in increased sensitivity to tumor necrosis factor, etoposide, and H2O2 in an oxidative stress-resistant cell line.
    Mairesse N, Bernaert D, Del Bino G, Horman S, Mosselmans R, Robaye B, Galand P.
    J Cell Physiol; 1998 Dec; 177(4):606-17. PubMed ID: 10092213
    [Abstract] [Full Text] [Related]

  • 10. Heat-induced morphological and biochemical changes in the nuclear lamina from Ehrlich ascites tumor cells in vivo.
    Krachmarov CP, Traub P.
    J Cell Biochem; 1993 Jul; 52(3):308-19. PubMed ID: 8366142
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  • 12. MAPKAP kinase 2 is activated by heat shock and TNF-alpha: in vivo phosphorylation of small heat shock protein results from stimulation of the MAP kinase cascade.
    Engel K, Ahlers A, Brach MA, Herrmann F, Gaestel M.
    J Cell Biochem; 1995 Feb; 57(2):321-30. PubMed ID: 7759569
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  • 14. Effects of the overexpression of the small heat shock protein, HSP27, on the sensitivity of human fibroblast cells exposed to oxidative stress.
    Arata S, Hamaguchi S, Nose K.
    J Cell Physiol; 1995 Jun; 163(3):458-65. PubMed ID: 7775589
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  • 15. Heat-shock gene expression and cell cycle changes during mammalian embryonic development.
    Walsh D, Li K, Wass J, Dolnikov A, Zeng F, Zhe L, Edwards M.
    Dev Genet; 1993 Jun; 14(2):127-36. PubMed ID: 8482017
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  • 17. Photodynamic therapy-mediated oxidative stress as a molecular switch for the temporal expression of genes ligated to the human heat shock promoter.
    Luna MC, Ferrario A, Wong S, Fisher AM, Gomer CJ.
    Cancer Res; 2000 Mar 15; 60(6):1637-44. PubMed ID: 10749134
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