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Journal Abstract Search

221 related items for PubMed ID: 7780150

  • 1. The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion.
    Romana SP, Mauchauffé M, Le Coniat M, Chumakov I, Le Paslier D, Berger R, Bernard OA.
    Blood; 1995 Jun 15; 85(12):3662-70. PubMed ID: 7780150
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  • 2. TEL-AML1 translocations with TEL and CDKN2 inactivation in acute lymphoblastic leukemia cell lines.
    Kim DH, Moldwin RL, Vignon C, Bohlander SK, Suto Y, Giordano L, Gupta R, Fears S, Nucifora G, Rowley JD.
    Blood; 1996 Aug 01; 88(3):785-94. PubMed ID: 8704231
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  • 7. The AML1 gene: a transcription factor involved in the pathogenesis of myeloid and lymphoid leukemias.
    Lo Coco F, Pisegna S, Diverio D.
    Haematologica; 1997 Aug 01; 82(3):364-70. PubMed ID: 9234595
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  • 8. The 12;21 translocation involving TEL and deletion of the other TEL allele: two frequently associated alterations found in childhood acute lymphoblastic leukemia.
    Raynaud S, Cave H, Baens M, Bastard C, Cacheux V, Grosgeorge J, Guidal-Giroux C, Guo C, Vilmer E, Marynen P, Grandchamp B.
    Blood; 1996 Apr 01; 87(7):2891-9. PubMed ID: 8639909
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  • 9. TEL/AML-1 dimerizes and is associated with a favorable outcome in childhood acute lymphoblastic leukemia.
    McLean TW, Ringold S, Neuberg D, Stegmaier K, Tantravahi R, Ritz J, Koeffler HP, Takeuchi S, Janssen JW, Seriu T, Bartram CR, Sallan SE, Gilliland DG, Golub TR.
    Blood; 1996 Dec 01; 88(11):4252-8. PubMed ID: 8943861
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  • 11. Lack of TEL-AML1 fusion transcript resulting from a cryptic t(12;21) in adult B lineage acute lymphoblastic leukemia in Taiwan.
    Shih LY, Chou TB, Liang DC, Tzeng YS, Rubnitz JE, Downing JR, Pui CH.
    Leukemia; 1996 Sep 01; 10(9):1456-8. PubMed ID: 8751462
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  • 12. Analysis of TEL proteins in human leukemias.
    Poirel H, Lacronique V, Mauchauffé M, Le Coniat M, Raffoux E, Daniel MT, Erickson P, Drabkin H, MacLeod RA, Drexler HG, Ghysdael J, Berger R, Bernard OA.
    Oncogene; 1998 Jun 04; 16(22):2895-903. PubMed ID: 9671410
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  • 15. TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis.
    Shurtleff SA, Buijs A, Behm FG, Rubnitz JE, Raimondi SC, Hancock ML, Chan GC, Pui CH, Grosveld G, Downing JR.
    Leukemia; 1995 Dec 04; 9(12):1985-9. PubMed ID: 8609706
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  • 16. High frequency of t(12;21) in childhood B-lineage acute lymphoblastic leukemia.
    Romana SP, Poirel H, Leconiat M, Flexor MA, Mauchauffé M, Jonveaux P, Macintyre EA, Berger R, Bernard OA.
    Blood; 1995 Dec 01; 86(11):4263-9. PubMed ID: 7492786
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  • 17. Expression levels of TEL, AML1, and the fusion products TEL-AML1 and AML1-TEL versus drug sensitivity and clinical outcome in t(12;21)-positive pediatric acute lymphoblastic leukemia.
    Stams WA, den Boer ML, Beverloo HB, Meijerink JP, van Wering ER, Janka-Schaub GE, Pieters R.
    Clin Cancer Res; 2005 Apr 15; 11(8):2974-80. PubMed ID: 15837750
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  • 18. Low frequency of the TEL/AML1 fusion gene in acute lymphoblastic leukaemia in Spain.
    García-Sanz R, Alaejos I, Orfão A, Rasillo A, Chillón MC, Tabernero MD, Mateos MV, López-Pérez R, González D, Balanzategui A, González M, San Miguel JF, Bortolucci A.
    Br J Haematol; 1999 Dec 15; 107(3):667-9. PubMed ID: 10583273
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  • 19. Molecular genetic studies on 167 pediatric ALL patients from different areas of Pakistan confirm a low frequency of the favorable prognosis fusion oncogene TEL-AML1 (t 12; 21) in underdeveloped countries of the region.
    Iqbal Z.
    Asian Pac J Cancer Prev; 2014 Dec 15; 15(8):3541-6. PubMed ID: 24870754
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