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Journal Abstract Search


236 related items for PubMed ID: 7784077

  • 1. Critical role of Rho in cell transformation by oncogenic Ras.
    Prendergast GC, Khosravi-Far R, Solski PA, Kurzawa H, Lebowitz PF, Der CJ.
    Oncogene; 1995 Jun 15; 10(12):2289-96. PubMed ID: 7784077
    [Abstract] [Full Text] [Related]

  • 2. An essential role for Rac in Ras transformation.
    Qiu RG, Chen J, Kirn D, McCormick F, Symons M.
    Nature; 1995 Mar 30; 374(6521):457-9. PubMed ID: 7700355
    [Abstract] [Full Text] [Related]

  • 3. Rit, a non-lipid-modified Ras-related protein, transforms NIH3T3 cells without activating the ERK, JNK, p38 MAPK or PI3K/Akt pathways.
    Rusyn EV, Reynolds ER, Shao H, Grana TM, Chan TO, Andres DA, Cox AD.
    Oncogene; 2000 Sep 28; 19(41):4685-94. PubMed ID: 11032018
    [Abstract] [Full Text] [Related]

  • 4. Ras-interacting domain of Ral GDP dissociation stimulator like (RGL) reverses v-Ras-induced transformation and Raf-1 activation in NIH3T3 cells.
    Okazaki M, Kishida S, Murai H, Hinoi T, Kikuchi A.
    Cancer Res; 1996 May 15; 56(10):2387-92. PubMed ID: 8625316
    [Abstract] [Full Text] [Related]

  • 5. Signal transduction elements of TC21, an oncogenic member of the R-Ras subfamily of GTP-binding proteins.
    Movilla N, Crespo P, Bustelo XR.
    Oncogene; 1999 Oct 21; 18(43):5860-9. PubMed ID: 10557073
    [Abstract] [Full Text] [Related]

  • 6. Ornithine decarboxylase induction in transformation by H-Ras and RhoA.
    Shantz LM, Pegg AE.
    Cancer Res; 1998 Jul 01; 58(13):2748-53. PubMed ID: 9661886
    [Abstract] [Full Text] [Related]

  • 7. Rac1 and extracellularly regulated kinase activation are sufficient for E1A-dependent cooperative transformation of primary epithelial cells, but progression can only be modulated by E1A or Rac1.
    Fischer RS, Zheng Y, Quinlan MP.
    Cell Growth Differ; 1998 Mar 01; 9(3):209-21. PubMed ID: 9543387
    [Abstract] [Full Text] [Related]

  • 8. Possible involvement of the inactivation of the Rho-Rho-kinase pathway in oncogenic Ras-induced transformation.
    Izawa I, Amano M, Chihara K, Yamamoto T, Kaibuchi K.
    Oncogene; 1998 Dec 03; 17(22):2863-71. PubMed ID: 9879992
    [Abstract] [Full Text] [Related]

  • 9. Effector domain mutants of Rho dissociate cytoskeletal changes from nuclear signaling and cellular transformation.
    Zohar M, Teramoto H, Katz BZ, Yamada KM, Gutkind JS.
    Oncogene; 1998 Aug 27; 17(8):991-8. PubMed ID: 9747878
    [Abstract] [Full Text] [Related]

  • 10. Cellular functions of TC10, a Rho family GTPase: regulation of morphology, signal transduction and cell growth.
    Murphy GA, Solski PA, Jillian SA, Pérez de la Ossa P, D'Eustachio P, Der CJ, Rush MG.
    Oncogene; 1999 Jul 01; 18(26):3831-45. PubMed ID: 10445846
    [Abstract] [Full Text] [Related]

  • 11. Retroviral gene transfer of dominant negative raf-1 mutants suppresses ha-ras-induced transformation and delays tumor formation.
    Heinicke T, Radziwill G, Nawrath M, Rommel C, Pavlovic J, Moelling K.
    Cancer Gene Ther; 2000 May 01; 7(5):697-706. PubMed ID: 10830717
    [Abstract] [Full Text] [Related]

  • 12. Rho family proteins and Ras transformation: the RHOad less traveled gets congested.
    Zohn IM, Campbell SL, Khosravi-Far R, Rossman KL, Der CJ.
    Oncogene; 1998 Sep 17; 17(11 Reviews):1415-38. PubMed ID: 9779988
    [Abstract] [Full Text] [Related]

  • 13. Transformation suppressor activity of C3G is independent of its CDC25-homology domain.
    Guerrero C, Fernandez-Medarde A, Rojas JM, Font de Mora J, Esteban LM, Santos E.
    Oncogene; 1998 Feb 05; 16(5):613-24. PubMed ID: 9482107
    [Abstract] [Full Text] [Related]

  • 14. trans-Dominant suppressor mutations of the H-ras oncogene.
    Ogiso Y, Gutierrez L, Wrathall LS, Lu YY, Blair DG, Clanton DJ, Hwang YW, Shih TY.
    Cell Growth Differ; 1990 May 05; 1(5):217-24. PubMed ID: 2150753
    [Abstract] [Full Text] [Related]

  • 15. Protein kinase Cepsilon is oncogenic in colon epithelial cells by interaction with the ras signal transduction pathway.
    Perletti GP, Concari P, Brusaferri S, Marras E, Piccinini F, Tashjian AH.
    Oncogene; 1998 Jun 25; 16(25):3345-8. PubMed ID: 9681835
    [Abstract] [Full Text] [Related]

  • 16. Essential role of Raf in Ras transformation and deregulation of matrix metalloproteinase expression in ovarian epithelial cells.
    Ulkü AS, Schäfer R, Der CJ.
    Mol Cancer Res; 2003 Dec 25; 1(14):1077-88. PubMed ID: 14707291
    [Abstract] [Full Text] [Related]

  • 17. Inhibition of oncogene-mediated transformation by ectopic expression of p21Waf1 in NIH3T3 cells.
    Michieli P, Li W, Lorenzi MV, Miki T, Zakut R, Givol D, Pierce JH.
    Oncogene; 1996 Feb 15; 12(4):775-84. PubMed ID: 8632899
    [Abstract] [Full Text] [Related]

  • 18. R-Ras induces malignant, but not morphologic, transformation of NIH3T3 cells.
    Cox AD, Brtva TR, Lowe DG, Der CJ.
    Oncogene; 1994 Nov 15; 9(11):3281-8. PubMed ID: 7936652
    [Abstract] [Full Text] [Related]

  • 19. Cytoskeletal organization in tropomyosin-mediated reversion of ras-transformation: Evidence for Rho kinase pathway.
    Shah V, Bharadwaj S, Kaibuchi K, Prasad GL.
    Oncogene; 2001 Apr 19; 20(17):2112-21. PubMed ID: 11360195
    [Abstract] [Full Text] [Related]

  • 20. Oncogenic activation of human R-ras by point mutations analogous to those of prototype H-ras oncogenes.
    Saez R, Chan AM, Miki T, Aaronson SA.
    Oncogene; 1994 Oct 19; 9(10):2977-82. PubMed ID: 8084601
    [Abstract] [Full Text] [Related]


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