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Journal Abstract Search

193 related items for PubMed ID: 7803857

  • 1. Mitochondrial morphological and functional defects in yeast caused by yme1 are suppressed by mutation of a 26S protease subunit homologue.
    Campbell CL, Tanaka N, White KH, Thorsness PE.
    Mol Biol Cell; 1994 Aug; 5(8):899-905. PubMed ID: 7803857
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  • 2. Escape of mitochondrial DNA to the nucleus in yme1 yeast is mediated by vacuolar-dependent turnover of abnormal mitochondrial compartments.
    Campbell CL, Thorsness PE.
    J Cell Sci; 1998 Aug; 111 ( Pt 16)():2455-64. PubMed ID: 9683639
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  • 3. Biochemical and functional analysis of the YME1 gene product, an ATP and zinc-dependent mitochondrial protease from S. cerevisiae.
    Weber ER, Hanekamp T, Thorsness PE.
    Mol Biol Cell; 1996 Feb; 7(2):307-17. PubMed ID: 8688560
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  • 4. Genetic and biochemical basis for viability of yeast lacking mitochondrial genomes.
    Kominsky DJ, Brownson MP, Updike DL, Thorsness PE.
    Genetics; 2002 Dec; 162(4):1595-604. PubMed ID: 12524335
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  • 5. Inactivation of YME1, a member of the ftsH-SEC18-PAS1-CDC48 family of putative ATPase-encoding genes, causes increased escape of DNA from mitochondria in Saccharomyces cerevisiae.
    Thorsness PE, White KH, Fox TD.
    Mol Cell Biol; 1993 Sep; 13(9):5418-26. PubMed ID: 8355690
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  • 11. Requirement for the yeast gene LON in intramitochondrial proteolysis and maintenance of respiration.
    Suzuki CK, Suda K, Wang N, Schatz G.
    Science; 1994 Apr 08; 264(5156):273-6. PubMed ID: 8146662
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  • 17. Yeast counterparts of subunits S5a and p58 (S3) of the human 26S proteasome are encoded by two multicopy suppressors of nin1-1.
    Kominami K, Okura N, Kawamura M, DeMartino GN, Slaughter CA, Shimbara N, Chung CH, Fujimuro M, Yokosawa H, Shimizu Y, Tanahashi N, Tanaka K, Toh-e A.
    Mol Biol Cell; 1997 Jan 08; 8(1):171-87. PubMed ID: 9017604
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  • 20. Hereditary spastic paraplegia: mitochondrial metalloproteases of yeast.
    Pearce DA.
    Hum Genet; 1999 Jun 08; 104(6):443-8. PubMed ID: 10453730
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