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114 related items for PubMed ID: 7805890

  • 1.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 2. Dephosphorylation studies of SKNSH-SY 5Y cell Tau proteins by endogenous phosphatase activity.
    Soulié C, Lépagnol J, Delacourte A, Caillet-Boudin ML.
    Neurosci Lett; 1996 Mar 15; 206(2-3):189-92. PubMed ID: 8710183
    [Abstract] [Full Text] [Related]

  • 3. [Detection of Alzheimer type pathological epitopes on Tau proteins of neuroblastoma cells after treatment with okadaic acid].
    Sautière PE, Caillet-Boudin ML, Wattez A, Buée-Scherrer V, Delacourte A.
    C R Acad Sci III; 1993 Mar 15; 316(5):533-5. PubMed ID: 7693312
    [Abstract] [Full Text] [Related]

  • 4. Okadaic acid enhances abnormal phosphorylation on tau proteins.
    Furiya Y, Sahara N, Mori H.
    Neurosci Lett; 1993 Jun 25; 156(1-2):67-9. PubMed ID: 8414191
    [Abstract] [Full Text] [Related]

  • 5. The extent of phosphorylation of fetal tau is comparable to that of PHF-tau from Alzheimer paired helical filaments.
    Kenessey A, Yen SH.
    Brain Res; 1993 Nov 26; 629(1):40-6. PubMed ID: 8287279
    [Abstract] [Full Text] [Related]

  • 6. Reversible heat stress-related loss of phosphorylated Alzheimer-type epitopes in Tau proteins of human neuroblastoma cells.
    Chiang MF, Liu WK, Yen SH.
    J Neurosci; 1993 Nov 26; 13(11):4854-60. PubMed ID: 7693894
    [Abstract] [Full Text] [Related]

  • 7. Differential sensitivity to proteolysis by brain calpain of adult human tau, fetal human tau and PHF-tau.
    Mercken M, Grynspan F, Nixon RA.
    FEBS Lett; 1995 Jul 10; 368(1):10-4. PubMed ID: 7615058
    [Abstract] [Full Text] [Related]

  • 8. Induction of a specific tau Alzheimer epitope in SY-5Y neuroblastoma cells.
    Caillet-Boudin ML, Delacourte A.
    Neuroreport; 1996 Dec 20; 8(1):307-10. PubMed ID: 9051801
    [Abstract] [Full Text] [Related]

  • 9. Posttranslational modifications of tau in paired helical filaments.
    Morishima M, Ihara Y.
    Dementia; 1994 Dec 20; 5(5):282-8. PubMed ID: 7951685
    [Abstract] [Full Text] [Related]

  • 10. The phosphorylation state of tau in the developing rat brain is regulated by phosphoprotein phosphatases.
    Mawal-Dewan M, Henley J, Van de Voorde A, Trojanowski JQ, Lee VM.
    J Biol Chem; 1994 Dec 09; 269(49):30981-7. PubMed ID: 7983034
    [Abstract] [Full Text] [Related]

  • 11. Developmental changes in tau phosphorylation: fetal tau is transiently phosphorylated in a manner similar to paired helical filament-tau characteristic of Alzheimer's disease.
    Brion JP, Smith C, Couck AM, Gallo JM, Anderton BH.
    J Neurochem; 1993 Dec 09; 61(6):2071-80. PubMed ID: 8245963
    [Abstract] [Full Text] [Related]

  • 12. Site-specific regulation of Alzheimer-like tau phosphorylation in living neurons.
    Burack MA, Halpain S.
    Neuroscience; 1996 May 09; 72(1):167-84. PubMed ID: 8730715
    [Abstract] [Full Text] [Related]

  • 13. The phosphatase inhibitor okadaic acid induces a phosphorylated paired helical filament tau epitope in human LA-N-5 neuroblastoma cells.
    Vandermeeren M, Lübke U, Six J, Cras P.
    Neurosci Lett; 1993 Apr 16; 153(1):57-60. PubMed ID: 7685510
    [Abstract] [Full Text] [Related]

  • 14. Unique Alzheimer's disease paired helical filament specific epitopes involve double phosphorylation at specific sites.
    Hoffmann R, Lee VM, Leight S, Varga I, Otvos L.
    Biochemistry; 1997 Jul 01; 36(26):8114-24. PubMed ID: 9201960
    [Abstract] [Full Text] [Related]

  • 15. Protein kinases involved in the phosphorylation of human tau protein in transfected COS-1 cells.
    Medina M, García-Rocha M, Padilla R, Pérez M, Montejo de Garcini E, Avila J.
    Biochim Biophys Acta; 1996 May 24; 1316(1):43-50. PubMed ID: 8634342
    [Abstract] [Full Text] [Related]

  • 16. Okadaic acid induces hyperphosphorylated forms of tau protein in human brain slices.
    Harris KA, Oyler GA, Doolittle GM, Vincent I, Lehman RA, Kincaid RL, Billingsley ML.
    Ann Neurol; 1993 Jan 24; 33(1):77-87. PubMed ID: 8494335
    [Abstract] [Full Text] [Related]

  • 17. Epitope mapping of monoclonal antibodies to the paired helical filaments of Alzheimer's disease: identification of phosphorylation sites in tau protein.
    Goedert M, Jakes R, Crowther RA, Cohen P, Vanmechelen E, Vandermeeren M, Cras P.
    Biochem J; 1994 Aug 01; 301 ( Pt 3)(Pt 3):871-7. PubMed ID: 7519852
    [Abstract] [Full Text] [Related]

  • 18. Okadaic acid mediates tau phosphorylation via sustained activation of the L-voltage-sensitive calcium channel.
    Ekinci FJ, Ortiz D, Shea TB.
    Brain Res Mol Brain Res; 2003 Oct 07; 117(2):145-51. PubMed ID: 14559148
    [Abstract] [Full Text] [Related]

  • 19. Tau released from paired helical filaments with formic acid or guanidine is susceptible to calpain-mediated proteolysis.
    Yang LS, Gordon-Krajcer W, Ksiezak-Reding H.
    J Neurochem; 1997 Oct 07; 69(4):1548-58. PubMed ID: 9326284
    [Abstract] [Full Text] [Related]

  • 20. Plaque biogenesis in brain aging and Alzheimer's disease. I. Progressive changes in phosphorylation states of paired helical filaments and neurofilaments.
    Su JH, Cummings BJ, Cotman CW.
    Brain Res; 1996 Nov 11; 739(1-2):79-87. PubMed ID: 8955927
    [Abstract] [Full Text] [Related]

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