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Journal Abstract Search

291 related items for PubMed ID: 7891692

  • 1. The t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation.
    Meyers S, Lenny N, Hiebert SW.
    Mol Cell Biol; 1995 Apr; 15(4):1974-82. PubMed ID: 7891692
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  • 2. The AML1/ETO fusion protein activates transcription of BCL-2.
    Klampfer L, Zhang J, Zelenetz AO, Uchida H, Nimer SD.
    Proc Natl Acad Sci U S A; 1996 Nov 26; 93(24):14059-64. PubMed ID: 8943060
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  • 3. Functional domains of the t(8;21) fusion protein, AML-1/ETO.
    Lenny N, Meyers S, Hiebert SW.
    Oncogene; 1995 Nov 02; 11(9):1761-9. PubMed ID: 7478604
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  • 4. AML-2 is a potential target for transcriptional regulation by the t(8;21) and t(12;21) fusion proteins in acute leukemia.
    Meyers S, Lenny N, Sun W, Hiebert SW.
    Oncogene; 1996 Jul 18; 13(2):303-12. PubMed ID: 8710369
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  • 5. AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocytic leukemia zinc finger protein.
    Melnick A, Carlile GW, McConnell MJ, Polinger A, Hiebert SW, Licht JD.
    Blood; 2000 Dec 01; 96(12):3939-47. PubMed ID: 11090081
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  • 6. Junctions of the AML1/MTG8(ETO) fusion are constant in t(8;21) acute myeloid leukemia detected by reverse transcription polymerase chain reaction.
    Kozu T, Miyoshi H, Shimizu K, Maseki N, Kaneko Y, Asou H, Kamada N, Ohki M.
    Blood; 1993 Aug 15; 82(4):1270-6. PubMed ID: 8353289
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  • 7. The AML1/ETO(MTG8) and AML1/Evi-1 leukemia-associated chimeric oncoproteins accumulate PEBP2beta(CBFbeta) in the nucleus more efficiently than wild-type AML1.
    Tanaka K, Tanaka T, Kurokawa M, Imai Y, Ogawa S, Mitani K, Yazaki Y, Hirai H.
    Blood; 1998 Mar 01; 91(5):1688-99. PubMed ID: 9473235
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  • 8. The t(8;21) fusion product, AML-1-ETO, associates with C/EBP-alpha, inhibits C/EBP-alpha-dependent transcription, and blocks granulocytic differentiation.
    Westendorf JJ, Yamamoto CM, Lenny N, Downing JR, Selsted ME, Hiebert SW.
    Mol Cell Biol; 1998 Jan 01; 18(1):322-33. PubMed ID: 9418879
    [Abstract] [Full Text] [Related]

  • 9. Identification of AML-1 and the (8;21) translocation protein (AML-1/ETO) as sequence-specific DNA-binding proteins: the runt homology domain is required for DNA binding and protein-protein interactions.
    Meyers S, Downing JR, Hiebert SW.
    Mol Cell Biol; 1993 Oct 01; 13(10):6336-45. PubMed ID: 8413232
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  • 11. Characterization of the ETO and AML1-ETO proteins involved in 8;21 translocation in acute myelogenous leukemia.
    Le XF, Claxton D, Kornblau S, Fan YH, Mu ZM, Chang KS.
    Eur J Haematol; 1998 Apr 01; 60(4):217-25. PubMed ID: 9579874
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  • 13. Persistence of multipotent progenitors expressing AML1/ETO transcripts in long-term remission patients with t(8;21) acute myelogenous leukemia.
    Miyamoto T, Nagafuji K, Akashi K, Harada M, Kyo T, Akashi T, Takenaka K, Mizuno S, Gondo H, Okamura T, Dohy H, Niho Y.
    Blood; 1996 Jun 01; 87(11):4789-96. PubMed ID: 8639850
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  • 15. Mechanisms of transcriptional repression by the t(8;21)-, t(12;21)-, and inv(16)-encoded fusion proteins.
    Heibert SW, Lutterbach B, Durst K, Wang L, Linggi B, Wu S, Wood L, Amann J, King D, Hou Y.
    Cancer Chemother Pharmacol; 2001 Aug 01; 48 Suppl 1():S31-4. PubMed ID: 11587363
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  • 17. Transcriptional regulation by the t(8;21) fusion protein, AML-1/ETO.
    Hiebert SW, Downing JR, Lenny N, Meyers S.
    Curr Top Microbiol Immunol; 1996 Aug 01; 211():253-8. PubMed ID: 8585956
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