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Journal Abstract Search

304 related items for PubMed ID: 7925557

  • 1. The immune response to Plasmodium chabaudi malaria in interleukin-4-deficient mice.
    von der Weid T, Kopf M, Köhler G, Langhorne J.
    Eur J Immunol; 1994 Oct; 24(10):2285-93. PubMed ID: 7925557
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  • 2. Possible modulation by male sex hormone of Th1/Th2 function in protection against Plasmodium chabaudi chabaudi AS infection in mice.
    Zhang Z, Chen L, Saito S, Kanagawa O, Sendo F.
    Exp Parasitol; 2000 Nov; 96(3):121-9. PubMed ID: 11162362
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  • 3. Plasmodium chabaudi chabaudi: differential susceptibility of gene-targeted mice deficient in IL-10 to an erythrocytic-stage infection.
    Linke A, Kühn R, Müller W, Honarvar N, Li C, Langhorne J.
    Exp Parasitol; 1996 Nov; 84(2):253-63. PubMed ID: 8932775
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  • 4. In vivo IL-12 production and IL-12 receptors beta1 and beta2 mRNA expression in the spleen are differentially up-regulated in resistant B6 and susceptible A/J mice during early blood-stage Plasmodium chabaudi AS malaria.
    Sam H, Stevenson MM.
    J Immunol; 1999 Feb 01; 162(3):1582-9. PubMed ID: 9973416
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  • 5. Parasite-specific IgM plays a significant role in the protective immune response to asexual erythrocytic stage Plasmodium chabaudi AS infection.
    Couper KN, Phillips RS, Brombacher F, Alexander J.
    Parasite Immunol; 2005 May 01; 27(5):171-80. PubMed ID: 15987340
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  • 6. Th1 and Th2 CD4+ T cell clones specific for Plasmodium chabaudi but not for an unrelated antigen protect against blood stage P. chabaudi infection.
    Taylor-Robinson AW, Phillips RS.
    Eur J Immunol; 1994 Jan 01; 24(1):158-64. PubMed ID: 8020550
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  • 13. Characterization of T cell-mediated responses in nonhealing and healing Leishmania major infections in the absence of endogenous IL-4.
    Kropf P, Etges R, Schopf L, Chung C, Sypek J, Müller I.
    J Immunol; 1997 Oct 01; 159(7):3434-43. PubMed ID: 9317142
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  • 16. Notch1 expression on T cells is not required for CD4+ T helper differentiation.
    Tacchini-Cottier F, Allenbach C, Otten LA, Radtke F.
    Eur J Immunol; 2004 Jun 01; 34(6):1588-96. PubMed ID: 15162428
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  • 17. Early treatment during a primary malaria infection modifies the development of cross immunity.
    Legorreta-Herrera M, Ventura-Ayala ML, Licona-Chávez RN, Soto-Cruz I, Hernández-Clemente FF.
    Parasite Immunol; 2004 Jan 01; 26(1):7-17. PubMed ID: 15198641
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  • 18. Recently activated naive CD4 T cells can help resting B cells, and can produce sufficient autocrine IL-4 to drive differentiation to secretion of T helper 2-type cytokines.
    Croft M, Swain SL.
    J Immunol; 1995 May 01; 154(9):4269-82. PubMed ID: 7536767
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  • 19. The time course of selected malarial infections in cytokine-deficient mice.
    van der Heyde HC, Pepper B, Batchelder J, Cigel F, Weidanz WP.
    Exp Parasitol; 1997 Feb 01; 85(2):206-13. PubMed ID: 9030670
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  • 20. IL-12 is not required for induction of type 1 cytokine responses in viral infections.
    Oxenius A, Karrer U, Zinkernagel RM, Hengartner H.
    J Immunol; 1999 Jan 15; 162(2):965-73. PubMed ID: 9916721
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