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286 related items for PubMed ID: 7935449

  • 1. Anisomycin-activated protein kinases p45 and p55 but not mitogen-activated protein kinases ERK-1 and -2 are implicated in the induction of c-fos and c-jun.
    Cano E, Hazzalin CA, Mahadevan LC.
    Mol Cell Biol; 1994 Nov; 14(11):7352-62. PubMed ID: 7935449
    [Abstract] [Full Text] [Related]

  • 2. Identification of anisomycin-activated kinases p45 and p55 in murine cells as MAPKAP kinase-2.
    Cano E, Doza YN, Ben-Levy R, Cohen P, Mahadevan LC.
    Oncogene; 1996 Feb 15; 12(4):805-12. PubMed ID: 8632902
    [Abstract] [Full Text] [Related]

  • 3. Neither ERK nor JNK/SAPK MAP kinase subtypes are essential for histone H3/HMG-14 phosphorylation or c-fos and c-jun induction.
    Cano E, Hazzalin CA, Kardalinou E, Buckle RS, Mahadevan LC.
    J Cell Sci; 1995 Nov 15; 108 ( Pt 11)():3599-609. PubMed ID: 8586671
    [Abstract] [Full Text] [Related]

  • 4. Anisomycin selectively desensitizes signalling components involved in stress kinase activation and fos and jun induction.
    Hazzalin CA, Le Panse R, Cano E, Mahadevan LC.
    Mol Cell Biol; 1998 Apr 15; 18(4):1844-54. PubMed ID: 9528756
    [Abstract] [Full Text] [Related]

  • 5. Anisomycin and rapamycin define an area upstream of p70/85S6k containing a bifurcation to histone H3-HMG-like protein phosphorylation and c-fos-c-jun induction.
    Kardalinou E, Zhelev N, Hazzalin CA, Mahadevan LC.
    Mol Cell Biol; 1994 Feb 15; 14(2):1066-74. PubMed ID: 8289787
    [Abstract] [Full Text] [Related]

  • 6. p38/RK is essential for stress-induced nuclear responses: JNK/SAPKs and c-Jun/ATF-2 phosphorylation are insufficient.
    Hazzalin CA, Cano E, Cuenda A, Barratt MJ, Cohen P, Mahadevan LC.
    Curr Biol; 1996 Aug 01; 6(8):1028-31. PubMed ID: 8805335
    [Abstract] [Full Text] [Related]

  • 7. Jun N-terminal kinase mediates activation of skeletal muscle glycogen synthase by insulin in vivo.
    Moxham CM, Tabrizchi A, Davis RJ, Malbon CC.
    J Biol Chem; 1996 Nov 29; 271(48):30765-73. PubMed ID: 8940056
    [Abstract] [Full Text] [Related]

  • 8. The stress-activated protein kinase subfamily of c-Jun kinases.
    Kyriakis JM, Banerjee P, Nikolakaki E, Dai T, Rubie EA, Ahmad MF, Avruch J, Woodgett JR.
    Nature; 1994 May 12; 369(6476):156-60. PubMed ID: 8177321
    [Abstract] [Full Text] [Related]

  • 9. Epidermal growth factor induces c-fos and c-jun mRNA via Raf-1/MEK1/ERK-dependent and -independent pathways in bovine luteal cells.
    Chen DB, Davis JS.
    Mol Cell Endocrinol; 2003 Feb 28; 200(1-2):141-54. PubMed ID: 12644307
    [Abstract] [Full Text] [Related]

  • 10. Anisomycin uses multiple mechanisms to stimulate mitogen-activated protein kinases and gene expression and to inhibit neuronal differentiation in PC12 phaeochromocytoma cells.
    Törocsik B, Szeberényi J.
    Eur J Neurosci; 2000 Feb 28; 12(2):527-32. PubMed ID: 10712794
    [Abstract] [Full Text] [Related]

  • 11. c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK.
    Deng T, Karin M.
    Nature; 1994 Sep 08; 371(6493):171-5. PubMed ID: 8072547
    [Abstract] [Full Text] [Related]

  • 12. Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for lipopolysaccharide stimulation of tumor necrosis factor alpha (TNF-alpha) translation: glucocorticoids inhibit TNF-alpha translation by blocking JNK/SAPK.
    Swantek JL, Cobb MH, Geppert TD.
    Mol Cell Biol; 1997 Nov 08; 17(11):6274-82. PubMed ID: 9343388
    [Abstract] [Full Text] [Related]

  • 13. Transforming growth factor-alpha and epidermal growth factor activate mitogen-activated protein kinase and its substrates in intestinal epithelial cells.
    Oliver BL, Sha'afi RI, Hajjar JJ.
    Proc Soc Exp Biol Med; 1995 Nov 08; 210(2):162-70. PubMed ID: 7568287
    [Abstract] [Full Text] [Related]

  • 14. Activated Ki-Ras suppresses 12-O-tetradecanoylphorbol-13-acetate-induced activation of the c-Jun NH2-terminal kinase pathway in human colon cancer cells.
    Okumura K, Shirasawa S, Nishioka M, Sasazuki T.
    Cancer Res; 1999 May 15; 59(10):2445-50. PubMed ID: 10344756
    [Abstract] [Full Text] [Related]

  • 15. Signalling to chromatin and the superinduction of proto-oncogenes.
    Zhelev N, Kardalinou E, Hazzalin CA, Cano E, Barratt MJ, Mahadevan LC.
    Biochem Soc Trans; 1993 Nov 15; 21(4):907-11. PubMed ID: 8132091
    [No Abstract] [Full Text] [Related]

  • 16. Protein kinase C and protein kinase A inhibit calcium-dependent but not stress-dependent c-Jun N-terminal kinase activation in rat liver epithelial cells.
    Li X, Yu H, Graves LM, Earp HS.
    J Biol Chem; 1997 Jun 06; 272(23):14996-5002. PubMed ID: 9169474
    [Abstract] [Full Text] [Related]

  • 17. Protein synthesis inhibitors reveal differential regulation of mitogen-activated protein kinase and stress-activated protein kinase pathways that converge on Elk-1.
    Zinck R, Cahill MA, Kracht M, Sachsenmaier C, Hipskind RA, Nordheim A.
    Mol Cell Biol; 1995 Sep 06; 15(9):4930-8. PubMed ID: 7651411
    [Abstract] [Full Text] [Related]

  • 18. Effects of the inhibition of p38/RK MAP kinase on induction of five fos and jun genes by diverse stimuli.
    Hazzalin CA, Cuenda A, Cano E, Cohen P, Mahadevan LC.
    Oncogene; 1997 Nov 06; 15(19):2321-31. PubMed ID: 9393876
    [Abstract] [Full Text] [Related]

  • 19. c-Jun N-terminal phosphorylation correlates with activation of the JNK subgroup but not the ERK subgroup of mitogen-activated protein kinases.
    Minden A, Lin A, Smeal T, Dérijard B, Cobb M, Davis R, Karin M.
    Mol Cell Biol; 1994 Oct 06; 14(10):6683-8. PubMed ID: 7935387
    [Abstract] [Full Text] [Related]

  • 20. Angiotensin II stimulates calcium-dependent activation of c-Jun N-terminal kinase.
    Zohn IE, Yu H, Li X, Cox AD, Earp HS.
    Mol Cell Biol; 1995 Nov 06; 15(11):6160-8. PubMed ID: 7565768
    [Abstract] [Full Text] [Related]

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