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Journal Abstract Search

402 related items for PubMed ID: 7980421

  • 1.
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  • 2. Distinct role of 2-O-, N-, and 6-O-sulfate groups of heparin in the formation of the ternary complex with basic fibroblast growth factor and soluble FGF receptor-1.
    Rusnati M, Coltrini D, Caccia P, Dell'Era P, Zoppetti G, Oreste P, Valsasina B, Presta M.
    Biochem Biophys Res Commun; 1994 Aug 30; 203(1):450-8. PubMed ID: 8074689
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  • 4. Biologically active synthetic fragments of human basic fibroblast growth factor (bFGF): identification of two Asp-Gly-Arg-containing domains involved in the mitogenic activity of bFGF in endothelial cells.
    Presta M, Rusnati M, Urbinati C, Sommer A, Ragnotti G.
    J Cell Physiol; 1991 Dec 30; 149(3):512-24. PubMed ID: 1660485
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  • 6. Basic fibroblast growth factor (bFGF) dissociates rapidly from heparan sulfates but slowly from receptors. Implications for mechanisms of bFGF release from pericellular matrix.
    Moscatelli D.
    J Biol Chem; 1992 Dec 25; 267(36):25803-9. PubMed ID: 1464594
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  • 7. Differential structural requirements of heparin and heparan sulfate proteoglycans that promote binding of basic fibroblast growth factor to its receptor.
    Aviezer D, Levy E, Safran M, Svahn C, Buddecke E, Schmidt A, David G, Vlodavsky I, Yayon A.
    J Biol Chem; 1994 Jan 07; 269(1):114-21. PubMed ID: 8276782
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  • 8. Possible mechanism for lead inhibition of vascular endothelial cell proliferation: a lower response to basic fibroblast growth factor through inhibition of heparan sulfate synthesis.
    Fujiwara Y, Kaji T.
    Toxicology; 1999 Apr 15; 133(2-3):147-57. PubMed ID: 10378481
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  • 10. Sulfate moieties in the subendothelial extracellular matrix are involved in basic fibroblast growth factor sequestration, dimerization, and stimulation of cell proliferation.
    Miao HQ, Ishai-Michaeli R, Atzmon R, Peretz T, Vlodavsky I.
    J Biol Chem; 1996 Mar 01; 271(9):4879-86. PubMed ID: 8617759
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  • 11. Basic fibroblast growth factor is internalized through both receptor-mediated and heparan sulfate-mediated mechanisms.
    Roghani M, Moscatelli D.
    J Biol Chem; 1992 Nov 05; 267(31):22156-62. PubMed ID: 1429568
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  • 12. Heparin oligosaccharides: inhibitors of the biological activity of bFGF on Caco-2 cells.
    Jayson GC, Gallagher JT.
    Br J Cancer; 1997 Nov 05; 75(1):9-16. PubMed ID: 9000592
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  • 13. Preparation of affinity-fractionated, heparin-derived oligosaccharides and their effects on selected biological activities mediated by basic fibroblast growth factor.
    Ishihara M, Tyrrell DJ, Stauber GB, Brown S, Cousens LS, Stack RJ.
    J Biol Chem; 1993 Mar 05; 268(7):4675-83. PubMed ID: 8444841
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  • 15. Basic fibroblast growth factor binding and processing by human glioma cells.
    Murphy PR, Knee RS.
    Mol Cell Endocrinol; 1995 Oct 30; 114(1-2):193-203. PubMed ID: 8674845
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  • 16. Basic fibroblast growth factor (FGF-2) internalization through the heparan sulfate proteoglycans-mediated pathway: an ultrastructural approach.
    Gleizes PE, Noaillac-Depeyre J, Amalric F, Gas N.
    Eur J Cell Biol; 1995 Jan 30; 66(1):47-59. PubMed ID: 7750519
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  • 17. Sulfated malto-oligosaccharides bind to basic FGF, inhibit endothelial cell proliferation, and disrupt endothelial cell tube formation.
    Foxall C, Wei Z, Schaefer ME, Casabonne M, Fugedi P, Peto C, Castellot JJ, Brandley BK.
    J Cell Physiol; 1996 Sep 30; 168(3):657-67. PubMed ID: 8816920
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  • 18. Importance of size and sulfation of heparin in release of basic fibroblast growth factor from the vascular endothelium and extracellular matrix.
    Ishai-Michaeli R, Svahn CM, Weber M, Chajek-Shaul T, Korner G, Ekre HP, Vlodavsky I.
    Biochemistry; 1992 Feb 25; 31(7):2080-8. PubMed ID: 1536850
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  • 19. Degradation of cell surface heparan sulfates decreases the high affinity binding of basic FGF to endothelial cells, but not to FRTL-5 rat thyroid cells.
    Emoto N, Isozaki O, Ohmura E, Shizume K, Tsushima T, Demura H.
    Thyroid; 1995 Dec 25; 5(6):455-60. PubMed ID: 8808095
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