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Journal Abstract Search

424 related items for PubMed ID: 8012702

  • 1. Potentiation by ACE inhibitors of the dilator response to bradykinin in the coronary microcirculation: interaction at the receptor level.
    Hecker M, Pörsti I, Bara AT, Busse R.
    Br J Pharmacol; 1994 Jan; 111(1):238-44. PubMed ID: 8012702
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  • 3. ACE inhibitor potentiation of bradykinin-induced venoconstriction.
    Hecker M, Blaukat A, Bara AT, Müller-Esterl W, Busse R.
    Br J Pharmacol; 1997 Aug; 121(7):1475-81. PubMed ID: 9257930
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  • 5. Endothelium-derived bradykinin: implications for angiotensin-converting enzyme-inhibitor therapy.
    Busse R, Fleming I, Hecker M.
    J Cardiovasc Pharmacol; 1993 Aug; 22 Suppl 5():S31-6. PubMed ID: 7508049
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  • 6. Significance of endothelial prostacyclin and nitric oxide in peripheral and pulmonary circulation.
    Gryglewski RJ, Chłopicki S, Uracz W, Marcinkiewicz E.
    Med Sci Monit; 2001 Aug; 7(1):1-16. PubMed ID: 11208485
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  • 7. Display of the characteristics of endothelium-derived hyperpolarizing factor by a cytochrome P450-derived arachidonic acid metabolite in the coronary microcirculation.
    Bauersachs J, Hecker M, Busse R.
    Br J Pharmacol; 1994 Dec; 113(4):1548-53. PubMed ID: 7889312
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  • 9. Relaxation of isolated coronary arteries by angiotensin-converting enzyme inhibitors: role of endothelium-derived kinins.
    Hecker M, Bara AT, Busse R.
    J Vasc Res; 1993 Dec; 30(5):257-62. PubMed ID: 8399986
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  • 10. Angiotensin-converting enzyme inhibitors unmask endogenous kinin production by bovine coronary artery endothelium.
    Hecker M, Bara AT, Busse R.
    Eur Heart J; 1993 Nov; 14 Suppl I():161-3. PubMed ID: 8293768
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  • 11. Potentiation of kinin analogues by ramiprilat is exclusively related to their degradation.
    Dendorfer A, Reibetamann S, Wolfrum S, Raasch W, Dominiak P.
    Hypertension; 2001 Jul; 38(1):142-6. PubMed ID: 11463775
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  • 12. Potentiation of the vascular response to kinins by inhibition of myocardial kininases.
    Dendorfer A, Wolfrum S, Schäfer U, Stewart JM, Inamura N, Dominiak P.
    Hypertension; 2000 Jan; 35(1 Pt 1):32-7. PubMed ID: 10642271
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  • 13. Characterization of endothelium-derived hyperpolarizing factor as a cytochrome P450-derived arachidonic acid metabolite in mammals.
    Hecker M, Bara AT, Bauersachs J, Busse R.
    J Physiol; 1994 Dec 01; 481 ( Pt 2)(Pt 2):407-14. PubMed ID: 7738833
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  • 15. Bradykinin potentiation by angiotensin-(1-7) and ACE inhibitors correlates with ACE C- and N-domain blockade.
    Tom B, de Vries R, Saxena PR, Danser AH.
    Hypertension; 2001 Jul 01; 38(1):95-9. PubMed ID: 11463767
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  • 16. Studies on the angiotensin-converting enzyme and the kinin B2 receptor in the rabbit jugular vein: modulation of contractile response to bradykinin.
    Gobeil F, Hallé S, Blais PA, Regoli D.
    Can J Physiol Pharmacol; 2002 Feb 01; 80(2):153-63. PubMed ID: 11934258
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  • 17. Prostacyclin and nitric oxide contribute to the vasodilator action of acetylcholine and bradykinin in the intact rabbit coronary bed.
    Lamontagne D, König A, Bassenge E, Busse R.
    J Cardiovasc Pharmacol; 1992 Oct 01; 20(4):652-7. PubMed ID: 1280723
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  • 18. Enhancement of cytosolic calcium, prostacyclin and nitric oxide by bradykinin and the ACE inhibitor ramiprilat in porcine brain capillary endothelial cells.
    Wiemer G, Popp R, Schölkens BA, Gögelein H.
    Brain Res; 1994 Feb 28; 638(1-2):261-6. PubMed ID: 7515323
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  • 19. ACE inhibitor and AT1 antagonist blockade of deformation-induced gene expression in the rabbit jugular vein through B2 receptor activation.
    Lauth M, Cattaruzza M, Hecker M.
    Arterioscler Thromb Vasc Biol; 2001 Jan 28; 21(1):61-6. PubMed ID: 11145934
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  • 20. Endothelium-derived hyperpolarizing factor, but not nitric oxide or prostacyclin release, is resistant to menadione-induced oxidative stress in the bovine coronary artery.
    Kaw S, Hecker M.
    Naunyn Schmiedebergs Arch Pharmacol; 1999 Feb 28; 359(2):133-9. PubMed ID: 10048598
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