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67 related items for PubMed ID: 8051714

  • 1. Failure to observe a relationship between bis-(beta-chloroethyl)sulfide-induced NAD depletion and cytotoxicity in the rat keratinocyte culture.
    Lin P, Bernstein IA, Vaughan FL.
    J Toxicol Environ Health; 1994 Aug; 42(4):393-405. PubMed ID: 8051714
    [Abstract] [Full Text] [Related]

  • 2. Formation of interstrand DNA cross-links by bis-(2-chloroethyl)sulfide (BCES): a possible cytotoxic mechanism in rat keratinocytes.
    Lin P, Vaughan FL, Bernstein IA.
    Biochem Biophys Res Commun; 1996 Jan 17; 218(2):556-61. PubMed ID: 8561795
    [Abstract] [Full Text] [Related]

  • 3. Pretreatment of primary rat cutaneous epidermal keratinocyte culture with a low concentration of MNNG: effect on DNA cross-linking measured in situ after challenge with bis-2-chloroethyl sulfide.
    Sorscher DH, Conolly RB.
    J Toxicol Environ Health; 1989 Jan 17; 27(3):367-79. PubMed ID: 2474078
    [Abstract] [Full Text] [Related]

  • 4. Activation of poly [ADP-Ribose] polymerase in endothelial cells and keratinocytes: role in an in vitro model of sulfur mustard-mediated vesication.
    Hinshaw DB, Lodhi IJ, Hurley LL, Atkins KB, Dabrowska MI.
    Toxicol Appl Pharmacol; 1999 Apr 01; 156(1):17-29. PubMed ID: 10101095
    [Abstract] [Full Text] [Related]

  • 5. Inhibition of poly(ADP-ribose) polymerase (PARP) influences the mode of sulfur mustard (SM)-induced cell death in HaCaT cells.
    Kehe K, Raithel K, Kreppel H, Jochum M, Worek F, Thiermann H.
    Arch Toxicol; 2008 Jul 01; 82(7):461-70. PubMed ID: 18046540
    [Abstract] [Full Text] [Related]

  • 6. bis-(beta-chloroethyl)sulfide (BCES)-induced changes in epidermal cell homeostasis in vitro.
    Ku WW, Bernstein IA.
    Toxicol Appl Pharmacol; 1988 Sep 30; 95(3):397-411. PubMed ID: 3188008
    [Abstract] [Full Text] [Related]

  • 7. The role of NAD+ depletion in the mechanism of sulfur mustard-induced metabolic injury.
    Martens ME, Smith WJ.
    Cutan Ocul Toxicol; 2008 Sep 30; 27(1):41-53. PubMed ID: 18330833
    [Abstract] [Full Text] [Related]

  • 8. Bis(2-chloroethyl)sulfide (BCES) disturbs the progression of rat keratinocytes through the cell cycle.
    Lin PP, Bernstein IA, Vaughan FL.
    Toxicol Lett; 1996 Jan 30; 84(1):23-32. PubMed ID: 8597174
    [Abstract] [Full Text] [Related]

  • 9. Assessment of the role of DNA damage and repair in the survival of primary cultures of rat cutaneous keratinocytes exposed to bis(2-chloroethyl)sulfide.
    Ribeiro PL, Mitra RS, Bernstein IA.
    Toxicol Appl Pharmacol; 1991 Nov 30; 111(2):342-51. PubMed ID: 1957317
    [Abstract] [Full Text] [Related]

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  • 11. Macromolecular metabolism of a differentiated rat keratinocyte culture system following exposure to sulfur mustard.
    Vaughan FL, Zaman S, Scavarelli R, Bernstein IA.
    J Toxicol Environ Health; 1988 Nov 30; 23(4):507-18. PubMed ID: 2452256
    [Abstract] [Full Text] [Related]

  • 12. Appearance of interleukin 1 alpha relates DNA interstrand cross-links and cytotoxicity in cultured human keratinocytes exposed to bis-(2-chloroethyl) sulfide.
    Pu Y, Lin P, Vaughan FL, Bernstein IA.
    J Appl Toxicol; 1995 Nov 30; 15(6):477-82. PubMed ID: 8603935
    [Abstract] [Full Text] [Related]

  • 13. Nitric oxide toxicity in islet cells involves poly(ADP-ribose) polymerase activation and concomitant NAD+ depletion.
    Radons J, Heller B, Bürkle A, Hartmann B, Rodriguez ML, Kröncke KD, Burkart V, Kolb H.
    Biochem Biophys Res Commun; 1994 Mar 30; 199(3):1270-7. PubMed ID: 8147870
    [Abstract] [Full Text] [Related]

  • 14. Inhibition of tumor cell growth by interferon-gamma is mediated by two distinct mechanisms dependent upon oxygen tension: induction of tryptophan degradation and depletion of intracellular nicotinamide adenine dinucleotide.
    Aune TM, Pogue SL.
    J Clin Invest; 1989 Sep 30; 84(3):863-75. PubMed ID: 2503544
    [Abstract] [Full Text] [Related]

  • 15. DNA strand breaks and poly(ADP-ribose) synthetase activation in pancreatic islets--a new aspect to development of insulin-dependent diabetes and pancreatic B-cell tumors.
    Okamoto H, Yamamoto H.
    Princess Takamatsu Symp; 1983 Sep 30; 13():297-308. PubMed ID: 6317640
    [Abstract] [Full Text] [Related]

  • 16. Cytoprotective effect of gallotannin in oxidatively stressed HaCaT keratinocytes: the role of poly(ADP-ribose) metabolism.
    Bakondi E, Bai P, Erdélyi K, Szabó C, Gergely P, Virág L.
    Exp Dermatol; 2004 Mar 30; 13(3):170-8. PubMed ID: 14987257
    [Abstract] [Full Text] [Related]

  • 17. Poly(ADP-ribose) synthesis in blocked and damaged cells and its relation to carcinogenesis.
    Boulikas T.
    Anticancer Res; 1992 Mar 30; 12(3):885-98. PubMed ID: 1535771
    [Abstract] [Full Text] [Related]

  • 18. Different basal NAD levels determine opposite effects of poly(ADP-ribosyl)polymerase inhibitors on H2O2-induced apoptosis.
    Coppola S, Nosseri C, Maresca V, Ghibelli L.
    Exp Cell Res; 1995 Dec 30; 221(2):462-9. PubMed ID: 7493646
    [Abstract] [Full Text] [Related]

  • 19. Role of poly(ADP-ribose) polymerase in rapid intracellular acidification induced by alkylating DNA damage.
    Affar el B, Shah RG, Dallaire AK, Castonguay V, Shah GM.
    Proc Natl Acad Sci U S A; 2002 Jan 08; 99(1):245-50. PubMed ID: 11756665
    [Abstract] [Full Text] [Related]

  • 20. The use of human epidermal keratinocytes in culture as a model for studying the biochemical mechanisms of sulfur mustard toxicity.
    Smith WJ, Gross CL, Chan P, Meier HL.
    Cell Biol Toxicol; 1990 Jul 08; 6(3):285-91. PubMed ID: 2147571
    [Abstract] [Full Text] [Related]

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