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124 related items for PubMed ID: 8070815

  • 1. Molecular basis of the motheaten phenotype.
    Tsui FW, Tsui HW.
    Immunol Rev; 1994 Apr; 138():185-206. PubMed ID: 8070815
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  • 2.
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  • 3. Mutations at the murine motheaten locus are within the hematopoietic cell protein-tyrosine phosphatase (Hcph) gene.
    Shultz LD, Schweitzer PA, Rajan TV, Yi T, Ihle JN, Matthews RJ, Thomas ML, Beier DR.
    Cell; 1993 Jul 02; 73(7):1445-54. PubMed ID: 8324828
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  • 4. Identification of PTP1C mutation as the genetic defect in motheaten and viable motheaten mice: a step toward defining the roles of protein tyrosine phosphatases in the regulation of hemopoietic cell differentiation and function.
    Bignon JS, Siminovitch KA.
    Clin Immunol Immunopathol; 1994 Nov 02; 73(2):168-79. PubMed ID: 7923924
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  • 5. B and T cells are not required for the viable motheaten phenotype.
    Yu CC, Tsui HW, Ngan BY, Shulman MJ, Wu GE, Tsui FW.
    J Exp Med; 1996 Feb 01; 183(2):371-80. PubMed ID: 8627150
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  • 6. Increased expression of the stefin A cysteine proteinase inhibitor occurs in the myelomonocytic cell-infiltrated tissues of autoimmune motheaten mice.
    Mlinaric-Rascan I, Asa SL, Siminovitch KA.
    Am J Pathol; 1994 Oct 01; 145(4):902-12. PubMed ID: 7943179
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  • 7. Expression and catalytic activity of the tyrosine phosphatase PTP1C is severely impaired in motheaten and viable motheaten mice.
    Kozlowski M, Mlinaric-Rascan I, Feng GS, Shen R, Pawson T, Siminovitch KA.
    J Exp Med; 1993 Dec 01; 178(6):2157-63. PubMed ID: 8245788
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  • 8. Defective lymphopoiesis in the bone marrow of motheaten (me/me) and viable motheaten (mev/mev) mutant mice. II. Description of a microenvironmental defect for the generation of terminal deoxynucleotidyltransferase-positive bone marrow cells in vitro.
    Medlock ES, Goldschneider I, Greiner DL, Shultz L.
    J Immunol; 1987 Jun 01; 138(11):3590-7. PubMed ID: 3584969
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  • 9. Defective lymphopoiesis in bone marrow of motheaten (me/me) and viable motheaten (mev/mev) mutant mice. I. Analysis of development of prothymocytes, early B lineage cells, and terminal deoxynucleotidyl transferase-positive cells.
    Greiner DL, Goldschneider I, Komschlies KL, Medlock ES, Bollum FJ, Schultz L.
    J Exp Med; 1986 Oct 01; 164(4):1129-44. PubMed ID: 3531380
    [Abstract] [Full Text] [Related]

  • 10. Investigations into the regulation and function of the SH2 domain-containing protein-tyrosine phosphatase, SHP-1.
    Tsui FW, Martin A, Wang J, Tsui HW.
    Immunol Res; 2006 Oct 01; 35(1-2):127-36. PubMed ID: 17003515
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  • 11. Aberrant regulation of bone trace elements in motheaten and osteopetrosis mutant mice.
    Yamada G, Nakamura S, Haraguchi R, Sakai M, Suzuki K, Miyado K, Hasuwa H, Ogino Y, Minami T, Tohno Y, Blum M, Shultz LD.
    Cell Mol Biol (Noisy-le-grand); 1998 Mar 01; 44(2):315-9. PubMed ID: 9593582
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  • 12. DNA-binding antibodies from viable motheaten mutant mice: implications for B cell tolerance.
    Westhoff CM, Whittier A, Kathol S, McHugh J, Zajicek C, Shultz LD, Wylie DE.
    J Immunol; 1997 Sep 15; 159(6):3024-33. PubMed ID: 9300728
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  • 13. Mutation of the hematopoietic cell phosphatase (Hcph) gene is associated with resistance to gamma-irradiation-induced apoptosis in Src homology protein tyrosine phosphatase (SHP)-1-deficient "motheaten" mutant mice.
    Hsu HC, Shultz LD, Su X, Shi J, Yang PA, Relyea MJ, Zhang HG, Mountz JD.
    J Immunol; 2001 Jan 15; 166(2):772-80. PubMed ID: 11145649
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  • 14. Deficiency of SHP-1 protein-tyrosine phosphatase activity results in heightened osteoclast function and decreased bone density.
    Umeda S, Beamer WG, Takagi K, Naito M, Hayashi S, Yonemitsu H, Yi T, Shultz LD.
    Am J Pathol; 1999 Jul 15; 155(1):223-33. PubMed ID: 10393854
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  • 15. Deficiency of Src homology 2-containing phosphatase 1 results in abnormalities in murine neutrophil function: studies in motheaten mice.
    Kruger J, Butler JR, Cherapanov V, Dong Q, Ginzberg H, Govindarajan A, Grinstein S, Siminovitch KA, Downey GP.
    J Immunol; 2000 Nov 15; 165(10):5847-59. PubMed ID: 11067945
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  • 16. Macrophages from motheaten and viable motheaten mutant mice show increased proliferative responses to GM-CSF: detection of potential HCP substrates in GM-CSF signal transduction.
    Jiao H, Yang W, Berrada K, Tabrizi M, Shultz L, Yi T.
    Exp Hematol; 1997 Jul 15; 25(7):592-600. PubMed ID: 9216734
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  • 17. Establishment and characterization of pro-B cell lines from motheaten mutant mouse defective in SHP-1 protein tyrosine phosphatase.
    Miyamoto A, Kunisada T, Yamazaki H, Miyake K, Nishikawa SI, Sudo T, Shultz LD, Hayashi SI.
    Immunol Lett; 1998 Sep 15; 63(2):75-82. PubMed ID: 9761368
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  • 18. T cell developmental defects in 'viable motheaten' mice deficient in SHP-1 protein-tyrosine phosphatase. Developmental defects are corrected in vitro in the presence of normal hematopoietic-origin stromal cells and in vivo by exogenous IL-7.
    Christianson SW, Greiner DL, Deluca D, Leif J, Phillips NE, Hayes SM, Hayashi S, Joliat MJ, Lyons BL, Shultz LD.
    J Autoimmun; 2002 Mar 15; 18(2):119-30. PubMed ID: 11908944
    [Abstract] [Full Text] [Related]

  • 19. SHP-1 variant proteins are absent in motheaten mice despite presence of splice variant transcripts with open reading frames.
    Martin A, Tsui HW, Tsui FW.
    Mol Immunol; 1999 Mar 15; 36(15-16):1029-41. PubMed ID: 10698306
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  • 20. Absence of CD5 dramatically reduces progression of pulmonary inflammatory lesions in SHP-1 protein-tyrosine phosphatase-deficient 'viable motheaten' mice.
    Joliat MJ, Lang PA, Lyons BL, Burzenski L, Lynes MA, Yi T, Sundberg JP, Shultz LD.
    J Autoimmun; 2002 Mar 15; 18(2):105-17. PubMed ID: 11908943
    [Abstract] [Full Text] [Related]

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